It is now well known that many androgenic substances possess certain gynaecogenic properties and it is therefore not surprising that administration of androgens has been found to promote mammary development in the rat [Selye, McEuen, and Collip, 1936; Nelson and Gallagher, 1936; Astwood, Geschickter, and Rausch, 1937; Reece and Mixner, 1939], mouse [van Heuverswyn, Folley, and Gardner, 1939], and guinea-pig [Bottomley and Folley, 1938a]. These observations assume added significance in view of recent reports on the clinical use of testosterone propionate for the treatment of certain mastopathic conditions in women [see Turpault, 1937; Desmarest and Capitain, 1937; Loeser, 1938]. The rationale of such treatment must presumably lie in an attempt to reduce the oestrogenic stimulation of the breast by suppression of the gonadotrophic potency of the pituitary. In view of the fact that the agent used for this purpose is known to exert direct growth, and in some cases
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