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Se Hee Min Division of Endocrinology, Department of Internal Medicine, Seoul National University Hospital, Seoul, Republic of Korea

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Jung Hee Kim Division of Endocrinology, Department of Internal Medicine, Seoul National University Hospital, Seoul, Republic of Korea

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Yu Mi Kang Innovative Research Institute for Cell Therapy, Seoul, Republic of Korea

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Seung Hak Lee Department of Rehabilitation Medicine, Seoul National University Hospital, Seoul, Republic of Korea

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Byung-Mo Oh Department of Rehabilitation Medicine, Seoul National University Hospital, Seoul, Republic of Korea

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Kyou-Sup Han Department of Laboratory Medicine, Seoul National University Hospital, Seoul, Republic of Korea

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Meihua Zhang Department of Biomedical Science, Seoul National University College of Medicine, Seoul, Republic of Korea

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Hoe Suk Kim Department of Radiology, Seoul National University Hospital, Seoul, Republic of Korea

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Woo Kyung Moon Department of Biomedical Science, Seoul National University College of Medicine, Seoul, Republic of Korea
Department of Radiology, Seoul National University Hospital, Seoul, Republic of Korea

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Hakmo Lee Innovative Research Institute for Cell Therapy, Seoul, Republic of Korea

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Kyong Soo Park Division of Endocrinology, Department of Internal Medicine, Seoul National University Hospital, Seoul, Republic of Korea
Innovative Research Institute for Cell Therapy, Seoul, Republic of Korea

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Hye Seung Jung Division of Endocrinology, Department of Internal Medicine, Seoul National University Hospital, Seoul, Republic of Korea
Innovative Research Institute for Cell Therapy, Seoul, Republic of Korea

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-derived mesenchymal stem cells (MSC), however, MSC were not incorporated into tissues but remained in the gaps between muscles. Instead, secretion of cytokines, including fibroblast growth factor 2 (FGF2) and vascular endothelial growth factor (VEGF), was increased in

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0516
Volume/Issue:
Volume 239: Issue 3
Restricted access
Aarti D Rohira Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA

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David M Lonard Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA

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Bert W O’Malley Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA

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, both TAMs and MDSCs promote resistance to anti-VEGF therapy. Continued investigations into the interactions between the tumor stromal and epithelial cells are therefore important for the identification of new therapeutic targets. Tissue stroma

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-20-0511
Volume/Issue:
Volume 248: Issue 2
Free access
P Froment INSERM U.418, UMR Communications Cellulaire et Différenciation, Hôpital Debrousse, 29 rue Soeur Bouvier, 69322 Lyon, France
INSERM U.545, Institut Pasteur de Lille et Faculté de Pharmacie Université de Lille 2, 1 rue du Pr Calmette, 59019 Lille, France
LMCB, Department of Molecular Biomedical Research, V.I.B., Technologiepark 927, B-9052 Ghent (Zwijnaarde), Belgium
Physiologie de la reproduction et des comportements, UMR 6175 INRA-CNRS-Université F. Rabelais de Tours-Haras Nationaux, 37380 Nouzilly, France

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F Gizard INSERM U.418, UMR Communications Cellulaire et Différenciation, Hôpital Debrousse, 29 rue Soeur Bouvier, 69322 Lyon, France
INSERM U.545, Institut Pasteur de Lille et Faculté de Pharmacie Université de Lille 2, 1 rue du Pr Calmette, 59019 Lille, France
LMCB, Department of Molecular Biomedical Research, V.I.B., Technologiepark 927, B-9052 Ghent (Zwijnaarde), Belgium
Physiologie de la reproduction et des comportements, UMR 6175 INRA-CNRS-Université F. Rabelais de Tours-Haras Nationaux, 37380 Nouzilly, France

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D Defever INSERM U.418, UMR Communications Cellulaire et Différenciation, Hôpital Debrousse, 29 rue Soeur Bouvier, 69322 Lyon, France
INSERM U.545, Institut Pasteur de Lille et Faculté de Pharmacie Université de Lille 2, 1 rue du Pr Calmette, 59019 Lille, France
LMCB, Department of Molecular Biomedical Research, V.I.B., Technologiepark 927, B-9052 Ghent (Zwijnaarde), Belgium
Physiologie de la reproduction et des comportements, UMR 6175 INRA-CNRS-Université F. Rabelais de Tours-Haras Nationaux, 37380 Nouzilly, France

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B Staels INSERM U.418, UMR Communications Cellulaire et Différenciation, Hôpital Debrousse, 29 rue Soeur Bouvier, 69322 Lyon, France
INSERM U.545, Institut Pasteur de Lille et Faculté de Pharmacie Université de Lille 2, 1 rue du Pr Calmette, 59019 Lille, France
LMCB, Department of Molecular Biomedical Research, V.I.B., Technologiepark 927, B-9052 Ghent (Zwijnaarde), Belgium
Physiologie de la reproduction et des comportements, UMR 6175 INRA-CNRS-Université F. Rabelais de Tours-Haras Nationaux, 37380 Nouzilly, France

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J Dupont INSERM U.418, UMR Communications Cellulaire et Différenciation, Hôpital Debrousse, 29 rue Soeur Bouvier, 69322 Lyon, France
INSERM U.545, Institut Pasteur de Lille et Faculté de Pharmacie Université de Lille 2, 1 rue du Pr Calmette, 59019 Lille, France
LMCB, Department of Molecular Biomedical Research, V.I.B., Technologiepark 927, B-9052 Ghent (Zwijnaarde), Belgium
Physiologie de la reproduction et des comportements, UMR 6175 INRA-CNRS-Université F. Rabelais de Tours-Haras Nationaux, 37380 Nouzilly, France

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P Monget INSERM U.418, UMR Communications Cellulaire et Différenciation, Hôpital Debrousse, 29 rue Soeur Bouvier, 69322 Lyon, France
INSERM U.545, Institut Pasteur de Lille et Faculté de Pharmacie Université de Lille 2, 1 rue du Pr Calmette, 59019 Lille, France
LMCB, Department of Molecular Biomedical Research, V.I.B., Technologiepark 927, B-9052 Ghent (Zwijnaarde), Belgium
Physiologie de la reproduction et des comportements, UMR 6175 INRA-CNRS-Université F. Rabelais de Tours-Haras Nationaux, 37380 Nouzilly, France

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prostaglandin production. Other studies suggest that PPARγ plays a role in angiogenesis, as by regulating vascular endothelial growth factor (VEGF), secreted by lutein cells. VEGF maintains the corpus luteum function during pregnancy ( Kaczmarek et al. 2005

Article Type:
Review Article
DOI:
https://doi.org/10.1677/joe.1.06667
Volume/Issue:
Volume 189: Issue 2
Free access
Chalida Nakalekha Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School
Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School
Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School

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Chieko Yokoyama Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School
Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School

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Hiroyuki Miura Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School

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Neil Alles Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School
Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School

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Kazuhiro Aoki Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School
Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School

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Keiichi Ohya Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School

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Ikuo Morita Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School
Departments of, Cellular Physiological Chemistry, Fixed Prosthodontics, International Research Center for Molecular Science in Tooth and Bone Diseases (Global COE program), Institute of Cellular and System Medicine, Department of Hard Tissue Engineering (Pharmacology), Graduate School

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human mesenchymal stem cells . Journal of Cellular Physiology 200 400 – 406 . Clarkin CE Emery RJ Pitsillides AA Wheeler-Jones CP 2008 Evaluation of VEGF-mediated signaling in primary human cells reveals a paracrine action for VEGF in

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0376
Volume/Issue:
Volume 204: Issue 2
Free access
Wei Zhang Department of Physiology and Pathophysiology, Institutes of Biomedical Sciences, Department of Endocrinology, Shanghai Medical College

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Xin-Hong Wang Department of Physiology and Pathophysiology, Institutes of Biomedical Sciences, Department of Endocrinology, Shanghai Medical College

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Si-Feng Chen Department of Physiology and Pathophysiology, Institutes of Biomedical Sciences, Department of Endocrinology, Shanghai Medical College

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Guo-Ping Zhang Department of Physiology and Pathophysiology, Institutes of Biomedical Sciences, Department of Endocrinology, Shanghai Medical College

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Ning Lu Department of Physiology and Pathophysiology, Institutes of Biomedical Sciences, Department of Endocrinology, Shanghai Medical College

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Ren-Ming Hu Department of Physiology and Pathophysiology, Institutes of Biomedical Sciences, Department of Endocrinology, Shanghai Medical College

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Hui-Ming Jin Department of Physiology and Pathophysiology, Institutes of Biomedical Sciences, Department of Endocrinology, Shanghai Medical College
Department of Physiology and Pathophysiology, Institutes of Biomedical Sciences, Department of Endocrinology, Shanghai Medical College

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-Paque was from Amersham Biosciences. Vascular endothelial growth factor (VEGF), basic fibroblast growth factor (bFGF), and insulin-like growth factor (IGF) were obtained from Cytolab Ltd (Rehovot, Israel). CD117-PE, CD34-FITC, Flk-1-APC, and Cyclin E

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0036
Volume/Issue:
Volume 197: Issue 3
Free access
Zhen-Yue Tong Center for Cell and Vascular Biology, Children’s Research Institute, 700 Children’s Drive, Columbus, Ohio 43205, USA
Department of Surgery and
Department of Molecular and Cellular Biochemistry, The Ohio State University, Columbus, Ohio 43212, USA

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David R Brigstock Center for Cell and Vascular Biology, Children’s Research Institute, 700 Children’s Drive, Columbus, Ohio 43205, USA
Department of Surgery and
Department of Molecular and Cellular Biochemistry, The Ohio State University, Columbus, Ohio 43212, USA

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vascular endothelial growth factor (VEGF) and inhibits VEGF-induced angiogenesis. FASEB Journal 16 219 –221. Ivkovic S , Yoon BS, Popoff SN, Safadi FF, Libuda DE, Stephenson RC, Daluiski A & Lyons KM 2003 Connective tissue

Article Type:
Other
DOI:
https://doi.org/10.1677/joe.1.06719
Volume/Issue:
Volume 188: Issue 3
Free access
Alice S Green
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Paul J Rozance Department of Animal Sciences, Department of Pediatrics, University of Arizona, 1650 East Limberlost Drive, Tucson, Arizona 85719, USA

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Sean W Limesand
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↔ Corticosterone ↔ Corticosterone ↔ Corticosterone ↑ Corticosterone ↓ IGF2 ↓ Taurine ↑ Taurine ↑ Taurine ↓ VEGF ↑ Catecholamines ↔ Catecholamines Fetal pancreatic characteristics  β-Cell mass NR ↔ ↓ ↓ ↓ 76% ↔ ↔  β-Cell fraction ↓/↔ NR ↓ ↓ ↓ 42% ↔ ↔  Islet

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0399
Volume/Issue:
Volume 205: Issue 3
Free access
Brit H Boehmer Department of Pediatrics, Perinatal Research Center, University of Colorado School of Medicine, Aurora, Colorado, USA

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Sean W Limesand School of Animal and Comparative Biomedical Sciences, University of Arizona, Tucson, Arizona, USA

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Paul J Rozance Department of Pediatrics, Perinatal Research Center, University of Colorado School of Medicine, Aurora, Colorado, USA

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. Carr DJ Wallace JM Aitken RP Milne JS Martin JF Zachary IC Peebles DM David AL 2016 Peri- and postnatal effects of prenatal adenoviral VEGF gene therapy in growth-restricted sheep . Biology of Reproduction 94 142

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-17-0076
Volume/Issue:
Volume 235: Issue 2
Free access
Jacqueline M Wallace Rowett Institute, University of Aberdeen, Aberdeen, UK

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was based on the premise that local uterine artery adenovirus (Ad.)-mediated overexpression of vascular endothelial growth factor (VEGF) in overnourished dams would increase uterine blood flow as demonstrated in normally developing adult pregnancies

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-18-0670
Volume/Issue:
Volume 242: Issue 1
Free access
Gordon J Allan SIPBS, University of Strathclyde, Glasgow G1 1XW, UK

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James Beattie SIPBS, University of Strathclyde, Glasgow G1 1XW, UK

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David J Flint SIPBS, University of Strathclyde, Glasgow G1 1XW, UK

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, these molecules are considered to exert their effects extracellularly by interaction with growth factors. For example, CTGF binds to VEGF ( Inoki et al . 2002 ) and inhibits its actions and binds to, and enhances the actions of, TGFB1 ( Abreu et al

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0269
Volume/Issue:
Volume 199: Issue 2
Free access