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Hitesh Soni Department of Physiology, University of Tennessee Health Science Center, Memphis, Tennessee 38163, USA

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Adebowale Adebiyi Department of Physiology, University of Tennessee Health Science Center, Memphis, Tennessee 38163, USA

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cause vasodilation. For example, UII-induced endothelium-dependent vasodilation in rat-isolated coronary and mesenteric arteries precontracted with 5-HT and methoxamine respectively ( Bottrill et al . 2000 ). UII also dilated ET-1-precontracted human

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0556
Volume/Issue:
Volume 217: Issue 3
Free access
Stephen Mandang Department of Obstetrics and Gynaecology, Monash Institute for Medical Research, Monash Medical Centre, Monash University, 246 Clayton Road, Clayton, Victoria 3168, Australia

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Ursula Manuelpillai Department of Obstetrics and Gynaecology, Monash Institute for Medical Research, Monash Medical Centre, Monash University, 246 Clayton Road, Clayton, Victoria 3168, Australia

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Euan M Wallace Department of Obstetrics and Gynaecology, Monash Institute for Medical Research, Monash Medical Centre, Monash University, 246 Clayton Road, Clayton, Victoria 3168, Australia

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produce activin A ( Manuelpillai et al. 2001 , Tannetta et al. 2003 ), suggesting that the endothelium may be another source of circulating activin A in preeclampsia. In contrast, while we were also able to confirm that monocytes produce activin A in

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-06-0061
Volume/Issue:
Volume 192: Issue 3
Free access
Elena Grossini Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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Flavia Prodam Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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Gillian Elisabeth Walker Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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Lorenzo Sigaudo Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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Serena Farruggio Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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Kevin Bellofatto Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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Patrizia Marotta Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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Claudio Molinari Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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David Mary Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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Gianni Bona Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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Giovanni Vacca Laboratory of Physiology and Experimental Surgery, Pediatric Unit, Department of Translational Medicine, University Eastern Piedmont ‘A. Avogadro’, Via Solaroli 17, Azienda Ospedaliera Universitaria Maggiore della Carita, Corso Mazzini 36, I-28100 Novara, Italy

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been associated with an impaired endothelium-dependent vasodilation ( Tan et al . 2004 ), which is a predictor of coronary events. However, little is known about the physiological role of monomeric adiponectin in the in vivo acute modulation of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0170
Volume/Issue:
Volume 222: Issue 1
Free access
Katrine Dahl Bjørnholm Section of Experimental Animal Models, Department of Veterinary and Animal Sciences, University of Copenhagen, Frederiksberg, Denmark
Department of Cardiovascular Disease Research, Novo Nordisk, Måløv, Denmark

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Gro Klitgaard Povlsen Department of Diabetes Pharmacology 1, Novo Nordisk, Måløv, Denmark

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Maria Elm Ougaard Department of Pathology and Imaging, Novo Nordisk, Måløv, Denmark

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Charles Pyke Department of Pathology and Imaging, Novo Nordisk, Måløv, Denmark

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Günaj Rakipovski Department of Cardiovascular Disease Research, Novo Nordisk, Måløv, Denmark

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Pernille Tveden-Nyborg Section of Experimental Animal Models, Department of Veterinary and Animal Sciences, University of Copenhagen, Frederiksberg, Denmark

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Jens Lykkesfeldt Section of Experimental Animal Models, Department of Veterinary and Animal Sciences, University of Copenhagen, Frederiksberg, Denmark

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Gry Freja Skovsted Section of Experimental Animal Models, Department of Veterinary and Animal Sciences, University of Copenhagen, Frederiksberg, Denmark

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assess endothelium dependent vasodilation by acetylcholine (Ach, 10 -10 –10 -4 M, Sigma, stock solution 0.1 M in Milli-Q) and endothelium independent NO mediated vasodilation by sodium nitroprusside (SNP, 10 -10 –10 -5 M, Sigma, stock solution 0.1 M in

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-20-0608
Volume/Issue:
Volume 248: Issue 3
Restricted access
Seiji Tsutsumi
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Xi Zhang
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Keiko Takata
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Kazuhiro Takahashi
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Richard H Karas Department of Obstetrics and Gynecology, Molecular Cardiology Research Institute, Yamagata University School of Medicine, 2-2-2 Iida-Nishi, Yamagata 990-9585, Japan

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Hirohisa Kurachi
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Michael E Mendelsohn Department of Obstetrics and Gynecology, Molecular Cardiology Research Institute, Yamagata University School of Medicine, 2-2-2 Iida-Nishi, Yamagata 990-9585, Japan

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vasoconstriction in vessels from which the endothelium has been removed from both humans and wild-type animals ( Mügge et al . 1997 , Binko & Majewski 1998 ), an effect that is blocked by pharmacological inhibition of NOS2A ( Binko & Majewski 1998 ). Estrogen has

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0292
Volume/Issue:
Volume 199: Issue 2
Free access
Colin P Sibley Maternal and Fetal Health Research Centre, Division of Developmental Biology and Medicine, School of Medical Sciences, Faculty of Biology, Medicine and Health, University of Manchester, Manchester, UK
St Mary’s Hospital, Central Manchester University Hospitals NHS Foundation Trust, Manchester Academic Health Science Centre, Manchester, UK

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cytotrophoblast (EVT) cells invade the spiral arteries so that both the endothelium and smooth muscle of the vessels are eroded ( Burton et al . 2017 ). This invasion by the EVT is extensive and initially plugs the terminal end of the vessels, preventing any

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-17-0185
Volume/Issue:
Volume 234: Issue 2
Open access
Bo Chen Key Laboratory of Transplant Engineering and Immunology, Department of Human Anatomy, Ministry of Health, Regenerative Medicine Research Center, West China Hospital, Sichuan University, No.1, Keyuan Road 4th, Wuhou District, Chengdu, Sichuan Province 610041, People's Republic of China
Key Laboratory of Transplant Engineering and Immunology, Department of Human Anatomy, Ministry of Health, Regenerative Medicine Research Center, West China Hospital, Sichuan University, No.1, Keyuan Road 4th, Wuhou District, Chengdu, Sichuan Province 610041, People's Republic of China

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Yanrong Lu Key Laboratory of Transplant Engineering and Immunology, Department of Human Anatomy, Ministry of Health, Regenerative Medicine Research Center, West China Hospital, Sichuan University, No.1, Keyuan Road 4th, Wuhou District, Chengdu, Sichuan Province 610041, People's Republic of China

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Younan Chen Key Laboratory of Transplant Engineering and Immunology, Department of Human Anatomy, Ministry of Health, Regenerative Medicine Research Center, West China Hospital, Sichuan University, No.1, Keyuan Road 4th, Wuhou District, Chengdu, Sichuan Province 610041, People's Republic of China

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Jingqiu Cheng Key Laboratory of Transplant Engineering and Immunology, Department of Human Anatomy, Ministry of Health, Regenerative Medicine Research Center, West China Hospital, Sichuan University, No.1, Keyuan Road 4th, Wuhou District, Chengdu, Sichuan Province 610041, People's Republic of China

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Introduction The vascular endothelium synthesizes and releases multiple biologically active molecules that modulate vascular structure, vasodilation, vasoconstriction, and thrombolysis and form a natural barrier that maintains internal homeostasis

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-14-0662
Volume/Issue:
Volume 225: Issue 3
Free access
Robson A S Santos Departments of Physiology and Biophysics, Morphology, Department of Biochemistry and Molecular Biology, Max‐Delbrück Center for Molecular Medicine (MDC), Biological Sciences Institute, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil

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Anderson J Ferreira Departments of Physiology and Biophysics, Morphology, Department of Biochemistry and Molecular Biology, Max‐Delbrück Center for Molecular Medicine (MDC), Biological Sciences Institute, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil

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Thiago Verano-Braga Departments of Physiology and Biophysics, Morphology, Department of Biochemistry and Molecular Biology, Max‐Delbrück Center for Molecular Medicine (MDC), Biological Sciences Institute, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil
Departments of Physiology and Biophysics, Morphology, Department of Biochemistry and Molecular Biology, Max‐Delbrück Center for Molecular Medicine (MDC), Biological Sciences Institute, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil

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Michael Bader Departments of Physiology and Biophysics, Morphology, Department of Biochemistry and Molecular Biology, Max‐Delbrück Center for Molecular Medicine (MDC), Biological Sciences Institute, Federal University of Minas Gerais, Belo Horizonte, MG, Brazil

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). The ACE2/Ang-(1–7)/Mas axis induces the release of vasodilators, including prostanoids, NO and endothelium-derived hyperpolarizing factor ( Porsti et al . 1994 , Brosnihan et al . 1996 , Li et al . 1997 , Muthalif et al . 1998 , Iyer et al

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0341
Volume/Issue:
Volume 216: Issue 2
Free access
Georgios A Christou Laboratory of Physiology, Medical School, University of Ioannina, 45110 Ioannina, Greece

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Dimitrios N Kiortsis Laboratory of Physiology, Medical School, University of Ioannina, 45110 Ioannina, Greece

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subjects ( Shimotomai et al . 2005 ). Adiponectin is normally present on the endothelium and in the smooth muscle cells of intrarenal arteries/arterioles and on the endothelium of glomerular and peritubular capillaries, while a weaker signal for

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-13-0578
Volume/Issue:
Volume 221: Issue 2
Free access
Charles A Ducsay
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Dean A Myers Center for Perinatal Biology, Department of Obstetrics and Gynecology, Loma Linda University School of Medicine, Loma Linda, California 92350, USA

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nitric oxide synthase activity in an agonist-dependent manner . Biochemical Journal 398 279 – 288 doi:10.1042/BJ20060371 . Chen JX Meyrick B 2004 Hypoxia increases Hsp90 binding to eNOS via PI3K-Akt in porcine coronary artery endothelium

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-11-0034
Volume/Issue:
Volume 210: Issue 3
Free access