, osteoclasts, and osteocytes to maintain equilibrium between bone resorption and bone formation ( Khosla et al. 2012 ). However, increasing research has focused on the inflammation caused by estrogen deficiency ( Weitzmann & Pacifici 2006
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Wenjing Wu, Jiayao Fu, Yijing Gu, Yu Wei, Pengfei Ma, and Junhua Wu
Y Okuma, A E O’Connor, J A Muir, P G Stanton, D M de Kretser, and M P Hedger
Introduction Systemic and local inflammation inhibit male reproductive function by disrupting the hypothalamic–pituitary–Leydig cell axis and production of sperm ( O’Bryan et al. 2000 , Hales 2002 ). Studies have shown that
S P Sivarajasingam, N Imami, and M R Johnson
understanding of the nature and role of cytokines, chemokines, and hormones and their involvement in signalling within the myometrium particularly during labour. Myometrial inflammation Inflammation typically involves white cell infiltration and the
Hao Wu, Junduo Wu, Shengzhu Zhou, Wenlin Huang, Ying Li, Huan Zhang, Junnan Wang, and Ye Jia
) activation in diabetic vasculopathy ( Csiszar et al . 2009 , Orimo et al . 2009 , Yang et al . 2011 , Liu et al . 2016 ). Activation of SIRT1 by resveratrol improved palmitate-induced inflammation and insulin resistance ( Liu et al . 2016
Linda Vignozzi, Ilaria Cellai, Raffaella Santi, Letizia Lombardelli, Annamaria Morelli, Paolo Comeglio, Sandra Filippi, Federica Logiodice, Marco Carini, Gabriella Nesi, Mauro Gacci, Marie-Pierre Piccinni, Luciano Adorini, and Mario Maggi
wealth of recent epidemiological and histopatological studies have clearly evidenced that chronic inflammation is a common finding in BPH (see Kramer et al . (2007) , Fibbi et al . (2010 b ) and Schauer & Rowley (2011) for reviews). Data from two
J Kwakkel, H C van Beeren, M T Ackermans, M C Platvoet-ter Schiphorst, E Fliers, W M Wiersinga, and A Boelen
, timing, and severity of illness. 1) Turpentine-induced abscess in the hindlimb, a model of local chronic inflammation. 2) Streptococcus pneumoniae infection, a lethal model that results in severe pneumonia and sepsis. Muscle D2 expression and D2
Ziping Jiang, Junduo Wu, Fuzhe Ma, Jun Jiang, Linlin Xu, Lei Du, Wenlin Huang, Zhaohui Wang, Ye Jia, Laijin Lu, and Hao Wu
a/KEAP1/NRF2 signaling, and enhanced OS and inflammation in a time-dependent manner. Moreover, the role of miR-200a in controlling the expression and function of the KEAP1/NRF2 signaling in ECs was confirmed by using specific mimic and inhibitor of
Maayan Vatarescu, Sapir Bechor, Yulia Haim, Tal Pecht, Tanya Tarnovscki, Noa Slutsky, Ori Nov, Hagit Shapiro, Avishai Shemesh, Angel Porgador, Nava Bashan, and Assaf Rudich
Introduction Adipose tissue inflammation is frequently viewed as a central mechanism in the pathogenesis of obesity-associated glucose intolerance and metabolic dysregulation. In humans, association studies demonstrated that adipose tissue
Ken Kubota, Toshiro Kubota, Daisuke Kamei, Makoto Murakami, Ichiro Kudo, Takeshi Aso, and Ikuo Morita
increased appreciably during tissue inflammation or damage ( Murakami et al. 2003 ). As shown in this paper, however, mPGES-2 expression increased following LPS treatment under conditions where mPGES-1 was reduced. The present data demonstrated that mPGES
