associated with pericytes, angiogenesis, macrophages, inflammation, and ECM were analyzed using Student’s t -test. Differences with P < 0.05 were considered statistically significant. Results Vascular reactivity is suppressed only in the VAT of
Search Results
Hye-Jin Lee, Haifei Shi, Hella S Brönneke, Bo-Yeong Jin, Sang-Hyun Choi, Randy J Seeley, and Dong-Hoon Kim
Antonios Chatzigeorgiou, Eleni Kandaraki, Christina Piperi, Sarantis Livadas, Athanasios G Papavassiliou, Michael Koutsilieris, Apostolos Papalois, and Evanthia Diamanti-Kandarakis
and SR-BI ( Miyazaki et al . 2002 ). Macrophage SR-A, the product of the MSR1 gene, is a multifunctional, multiligand pattern recognition receptor with roles in innate immunity, apoptotic cell clearance and age-related degenerative pathologies, such
Alena Nareika, Yeong-Bin Im, Bryan A Game, Elizabeth H Slate, John J Sanders, Steven D London, Maria F Lopes-Virella, and Yan Huang
, studies have shown that macrophages isolated from CD14 knockout mice are unable to produce TNFα, IL-1β, and interferon-inducible protein-10 in response to LPS and these mice have an early death in pneumococcal infection ( Vogel et al . 1998 ). Clearly
Aran Son, Namju Kang, Sue Young Oh, Ki Woo Kim, Shmuel Muallem, Yu-Mi Yang, and Dong Min Shin
. Examples are autoimmune arthritis, osteoporosis, periodontitis, osteopetrosis and bone tumors ( Takayanagi 2007 , Zaidi 2007 ). Multinucleated mature osteoclasts originate from bone marrow-derived monocytes/macrophages (BMMs) through two essential factors
Yun Wang, Yue Zheng, Patsy M Nishina, and Jürgen K Naggert
inflammation, and obesity-associated cardiomyopathy. Obesity-induced inflammation is characterized in part by the infiltration of macrophages and other immune cells into white adipose tissue, and has been observed in both rodents and humans ( Wellen
M L Batista Jr, R X Neves, S B Peres, A S Yamashita, C S Shida, S R Farmer, and M Seelaender
ATG GTG AGA AAC C-3′ and antisense, 5′-CCC ATG GAT GTA CAG TAG CAG A-3′) and macrophage migration inhibitory factor (MIF; NM 031051.1: sense, 5′-TCC CGG ACC ACG TCA TGA CTT T-3′ and antisense, 5′-CGT TGG CTG CGT TCA TGT CGT AAT-3′) were designed using
Tristan S Allemann, Gursimran K Dhamrait, Naomi J Fleury, Tamara N Abel, Prue H Hart, Robyn M Lucas, Vance B Matthews, and Shelley Gorman
cells from iBAT using fluorochrome-conjugated antibodies, after blocking Fc receptors. Cells were stained with antibodies to identify: (i) macrophages (CD86-BIO (with streptavidin-PE-Cy5), CD11b-APC-Cy7, CD11c-APC, MHC Class II-PE, CD301-PE-Cy7 and F4
Gabriele Wolf, Nicole Aumann, Marta Michalska, Antje Bast, Jürgen Sonnemann, James F Beck, Uwe Lendeckel, Philip Newsholme, and Reinhard Walther
, an infiltration of pancreatic islets with lymphocytes, macrophages and neutrophil granulocytes releasing various pro-inflammatory cytokines ( Eizirik & Mandrup-Poulsen 2001 ). Chronic exposure to cytokines such as tumour necrosis factor-α (TNF
Linda Vignozzi, Annamaria Morelli, Erica Sarchielli, Paolo Comeglio, Sandra Filippi, Ilaria Cellai, Elena Maneschi, Sergio Serni, Mauro Gacci, Marco Carini, Marie-Pierre Piccinni, Farid Saad, Luciano Adorini, Gabriella B Vannelli, and Mario Maggi
et al . 2009 ). Briefly, prostate sections were incubated overnight at 4 °C or with the monoclonal mouse anti-CD45 (1:100 vol/vol, DakoCytomation, Copenhagen, Denmark), the anti-macrophage antibody RAM11 (Dako, Carpenteria, CA, USA; 1:80 vol/vol), or
Amanda E Garza, Elijah Trefts, Isis A Katayama Rangel, Danielle Brooks, Rene Baudrand, Burhanuddin Moize, Jose R Romero, Sanjay Ranjit, Thitinan Treesaranuwattana, Tham M Yao, Gail K Adler, Luminita H Pojoga, and Gordon H Williams
weight/body weight ratio in any of the four treatment groups or two genotypes ( Table 1 ). As a measure of cardiac injury, as described in the methods section. Macrophage infiltrates and focal adhesions were minimal to not observed in the aldosterone
