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Hye-Jin Lee, Haifei Shi, Hella S Brönneke, Bo-Yeong Jin, Sang-Hyun Choi, Randy J Seeley, and Dong-Hoon Kim

associated with pericytes, angiogenesis, macrophages, inflammation, and ECM were analyzed using Student’s t -test. Differences with P < 0.05 were considered statistically significant. Results Vascular reactivity is suppressed only in the VAT of

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Antonios Chatzigeorgiou, Eleni Kandaraki, Christina Piperi, Sarantis Livadas, Athanasios G Papavassiliou, Michael Koutsilieris, Apostolos Papalois, and Evanthia Diamanti-Kandarakis

and SR-BI ( Miyazaki et al . 2002 ). Macrophage SR-A, the product of the MSR1 gene, is a multifunctional, multiligand pattern recognition receptor with roles in innate immunity, apoptotic cell clearance and age-related degenerative pathologies, such

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Alena Nareika, Yeong-Bin Im, Bryan A Game, Elizabeth H Slate, John J Sanders, Steven D London, Maria F Lopes-Virella, and Yan Huang

, studies have shown that macrophages isolated from CD14 knockout mice are unable to produce TNFα, IL-1β, and interferon-inducible protein-10 in response to LPS and these mice have an early death in pneumococcal infection ( Vogel et al . 1998 ). Clearly

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Aran Son, Namju Kang, Sue Young Oh, Ki Woo Kim, Shmuel Muallem, Yu-Mi Yang, and Dong Min Shin

. Examples are autoimmune arthritis, osteoporosis, periodontitis, osteopetrosis and bone tumors ( Takayanagi 2007 , Zaidi 2007 ). Multinucleated mature osteoclasts originate from bone marrow-derived monocytes/macrophages (BMMs) through two essential factors

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Yun Wang, Yue Zheng, Patsy M Nishina, and Jürgen K Naggert

inflammation, and obesity-associated cardiomyopathy. Obesity-induced inflammation is characterized in part by the infiltration of macrophages and other immune cells into white adipose tissue, and has been observed in both rodents and humans ( Wellen

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M L Batista Jr, R X Neves, S B Peres, A S Yamashita, C S Shida, S R Farmer, and M Seelaender

ATG GTG AGA AAC C-3′ and antisense, 5′-CCC ATG GAT GTA CAG TAG CAG A-3′) and macrophage migration inhibitory factor (MIF; NM 031051.1: sense, 5′-TCC CGG ACC ACG TCA TGA CTT T-3′ and antisense, 5′-CGT TGG CTG CGT TCA TGT CGT AAT-3′) were designed using

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Tristan S Allemann, Gursimran K Dhamrait, Naomi J Fleury, Tamara N Abel, Prue H Hart, Robyn M Lucas, Vance B Matthews, and Shelley Gorman

cells from iBAT using fluorochrome-conjugated antibodies, after blocking Fc receptors. Cells were stained with antibodies to identify: (i) macrophages (CD86-BIO (with streptavidin-PE-Cy5), CD11b-APC-Cy7, CD11c-APC, MHC Class II-PE, CD301-PE-Cy7 and F4

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Gabriele Wolf, Nicole Aumann, Marta Michalska, Antje Bast, Jürgen Sonnemann, James F Beck, Uwe Lendeckel, Philip Newsholme, and Reinhard Walther

, an infiltration of pancreatic islets with lymphocytes, macrophages and neutrophil granulocytes releasing various pro-inflammatory cytokines ( Eizirik & Mandrup-Poulsen 2001 ). Chronic exposure to cytokines such as tumour necrosis factor-α (TNF

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Linda Vignozzi, Annamaria Morelli, Erica Sarchielli, Paolo Comeglio, Sandra Filippi, Ilaria Cellai, Elena Maneschi, Sergio Serni, Mauro Gacci, Marco Carini, Marie-Pierre Piccinni, Farid Saad, Luciano Adorini, Gabriella B Vannelli, and Mario Maggi

et al . 2009 ). Briefly, prostate sections were incubated overnight at 4 °C or with the monoclonal mouse anti-CD45 (1:100 vol/vol, DakoCytomation, Copenhagen, Denmark), the anti-macrophage antibody RAM11 (Dako, Carpenteria, CA, USA; 1:80 vol/vol), or

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Amanda E Garza, Elijah Trefts, Isis A Katayama Rangel, Danielle Brooks, Rene Baudrand, Burhanuddin Moize, Jose R Romero, Sanjay Ranjit, Thitinan Treesaranuwattana, Tham M Yao, Gail K Adler, Luminita H Pojoga, and Gordon H Williams

weight/body weight ratio in any of the four treatment groups or two genotypes ( Table 1 ). As a measure of cardiac injury, as described in the methods section. Macrophage infiltrates and focal adhesions were minimal to not observed in the aldosterone