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-producing cells in vitro . To achieve this, nuclear receptor subfamily 5, group A, member 1 ( NF5A1 that was previously known as SF-1 or AD4BP ) is a key molecule. NF5A1 is the steroidogenic tissue-specific transcription factor that controls the expression
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Department of Physiology, College of Medicine, National Cheng Kung University, Tainan, Taiwan
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-inducible factor (HIF)-dependent and HIF-independent pathways. The HIF-independent pathway is mainly mediated by changing protein phosphorylation status (acute) and global transcription/translation efficiency (chronic), whereas the HIF-dependent pathway is
EMI0363 Necker University, Paris, France
Pathology Department, Robert Debré Hospital, 3EA3102 Paris VII University, Paris, France
Endocrinology Department, Robert Debré Hospital, Paris, France
Paediatric Endocrinology Department, Necker Enfants Malades Hospital, Paris, France
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EMI0363 Necker University, Paris, France
Pathology Department, Robert Debré Hospital, 3EA3102 Paris VII University, Paris, France
Endocrinology Department, Robert Debré Hospital, Paris, France
Paediatric Endocrinology Department, Necker Enfants Malades Hospital, Paris, France
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EMI0363 Necker University, Paris, France
Pathology Department, Robert Debré Hospital, 3EA3102 Paris VII University, Paris, France
Endocrinology Department, Robert Debré Hospital, Paris, France
Paediatric Endocrinology Department, Necker Enfants Malades Hospital, Paris, France
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EMI0363 Necker University, Paris, France
Pathology Department, Robert Debré Hospital, 3EA3102 Paris VII University, Paris, France
Endocrinology Department, Robert Debré Hospital, Paris, France
Paediatric Endocrinology Department, Necker Enfants Malades Hospital, Paris, France
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and dendritic cells, were studied by reverse transcription and polymerase chain reaction (RT–PCR). CSF1 (colony-stimulating factor 1) and GM–CSF (granulocyte/macrophage colony-stimulating factor), which are known to be the most important growth factors
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
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Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
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Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
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Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
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Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
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Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
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Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
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Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
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defined in the human POMC promoter, domain IV (−376 to −417) had the distinctive property of being active in DMS-79 cells but not in AtT-20 cells ( Picon et al. 1995 ). It was reported that E2 transcription factor binding was required for the activity of
Department of Endocrinology, Medical Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Laboratory of Functional Genomics, Department of Medical Genome Science, Graduate School of Frontier Sciences, The University of Tokyo, 4-6-1 Shirokanedai, Minatoku, Tokyo, Japan
Division for Sex Differentiation, National Institute for Basic Biology, National Institutes of Natural Sciences, Higashiyama 5-1, Myondaiji-cho, Okazaki, Japan
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Department of Endocrinology, Medical Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Laboratory of Functional Genomics, Department of Medical Genome Science, Graduate School of Frontier Sciences, The University of Tokyo, 4-6-1 Shirokanedai, Minatoku, Tokyo, Japan
Division for Sex Differentiation, National Institute for Basic Biology, National Institutes of Natural Sciences, Higashiyama 5-1, Myondaiji-cho, Okazaki, Japan
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Department of Endocrinology, Medical Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Laboratory of Functional Genomics, Department of Medical Genome Science, Graduate School of Frontier Sciences, The University of Tokyo, 4-6-1 Shirokanedai, Minatoku, Tokyo, Japan
Division for Sex Differentiation, National Institute for Basic Biology, National Institutes of Natural Sciences, Higashiyama 5-1, Myondaiji-cho, Okazaki, Japan
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Department of Endocrinology, Medical Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Laboratory of Functional Genomics, Department of Medical Genome Science, Graduate School of Frontier Sciences, The University of Tokyo, 4-6-1 Shirokanedai, Minatoku, Tokyo, Japan
Division for Sex Differentiation, National Institute for Basic Biology, National Institutes of Natural Sciences, Higashiyama 5-1, Myondaiji-cho, Okazaki, Japan
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Department of Endocrinology, Medical Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Laboratory of Functional Genomics, Department of Medical Genome Science, Graduate School of Frontier Sciences, The University of Tokyo, 4-6-1 Shirokanedai, Minatoku, Tokyo, Japan
Division for Sex Differentiation, National Institute for Basic Biology, National Institutes of Natural Sciences, Higashiyama 5-1, Myondaiji-cho, Okazaki, Japan
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Department of Endocrinology, Medical Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Laboratory of Functional Genomics, Department of Medical Genome Science, Graduate School of Frontier Sciences, The University of Tokyo, 4-6-1 Shirokanedai, Minatoku, Tokyo, Japan
Division for Sex Differentiation, National Institute for Basic Biology, National Institutes of Natural Sciences, Higashiyama 5-1, Myondaiji-cho, Okazaki, Japan
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Department of Endocrinology, Medical Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Laboratory of Functional Genomics, Department of Medical Genome Science, Graduate School of Frontier Sciences, The University of Tokyo, 4-6-1 Shirokanedai, Minatoku, Tokyo, Japan
Division for Sex Differentiation, National Institute for Basic Biology, National Institutes of Natural Sciences, Higashiyama 5-1, Myondaiji-cho, Okazaki, Japan
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Department of Endocrinology, Medical Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Laboratory of Functional Genomics, Department of Medical Genome Science, Graduate School of Frontier Sciences, The University of Tokyo, 4-6-1 Shirokanedai, Minatoku, Tokyo, Japan
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Department of Endocrinology, Medical Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Laboratory of Functional Genomics, Department of Medical Genome Science, Graduate School of Frontier Sciences, The University of Tokyo, 4-6-1 Shirokanedai, Minatoku, Tokyo, Japan
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Department of Endocrinology, Medical Research Institute, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima Bunkyouku, Tokyo, Japan
Laboratory of Functional Genomics, Department of Medical Genome Science, Graduate School of Frontier Sciences, The University of Tokyo, 4-6-1 Shirokanedai, Minatoku, Tokyo, Japan
Division for Sex Differentiation, National Institute for Basic Biology, National Institutes of Natural Sciences, Higashiyama 5-1, Myondaiji-cho, Okazaki, Japan
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properties that were lost in the GC lines appears to be extremely difficult. One member of the orphan nuclear receptor subfamily, the adrenal-4 binding protein (Ad4BP), also known as steroidogenic factor-1 or NR5A1, regulates the transcription of
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that GPR30 signaling triggers lead to the induced expression of c-fos in macrophages ( Kanda & Watanabe 2003 a ) and activation of the transcription factor CREB in keratinocytes ( Kanda & Watanabe 2003 b , 2004 ). These in turn activate the
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transcription factor E2F1 is involved in the regulation of cell cycle regulation ( Yoshikawa 2000 ). Additionally, the activity of cyclin–CDK complex can be regulated by the inhibitory interaction with CDK inhibitors (CKIs), such as p21 CIP1 , p27 KIP1 and p57
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Departamento de Fisiología, Urología, Center for Integrative Medicine and Innovative Sciences, Department of Urology, Pontificia Universidad Católica de Chile, Santiago de Chile, Chile
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( Wen et al . 2013 ) and that the induction of VEGF was mediated by binding of the transcription factor AR and SP1 to the core promoter region of VEGF ( Eisermann et al . 2013 ). Androgen deprivation therapy (ADT), the standard treatment for advanced
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Laboratory of Molecular and Cellular Biochemistry, Faculty of Dental Science, Kyushu University, Fukuoka, Japan
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.01 (Student’s t test). We previously showed that GluOC activates the transcription factor CREB (cAMP response element–binding protein) and increases the expression of the transcription factor FoxO1 (forkhead box protein O1) in 3T3-L1 adipocytes
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s at 58 °C, 45 s at 72 °C; 5 min at 72 °C). PCR cycle number was 35 for all primer pairs with the exception of Isl1 (29 cycles) and forkhead box transcription factor a2 (Foxa2; 40 cycles). PCR products were separated and visualised on 1% agarose gels