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Chengyuan Lin School of Biological Sciences, University of Hong Kong, Hong Kong
Clinical Division, School of Chinese Medicine, Hong Kong Baptist University, Hong Kong
YMU-HKBU Joint Laboratory of Traditional Natural Medicine, Yunnan Minzu University, Kunming, China

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Xue Jiang School of Biological Sciences, University of Hong Kong, Hong Kong

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Mulan He School of Biological Sciences, University of Hong Kong, Hong Kong

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Ling Zhao Clinical Division, School of Chinese Medicine, Hong Kong Baptist University, Hong Kong

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Tao Huang Clinical Division, School of Chinese Medicine, Hong Kong Baptist University, Hong Kong

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Zhaoxiang Bian Clinical Division, School of Chinese Medicine, Hong Kong Baptist University, Hong Kong
YMU-HKBU Joint Laboratory of Traditional Natural Medicine, Yunnan Minzu University, Kunming, China

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Anderson O L Wong School of Biological Sciences, University of Hong Kong, Hong Kong

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E 2012 Cloning and differential expression pattern of pituitary adenylyl cyclase-activating polypeptide and PACAP-specific receptor in darkbarbel catfish Pelteobagrus vachelli. Comparative Biochemistry and Physiology Part B: Biochemistry and

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Research Article
DOI:
https://doi.org/10.1530/JOE-16-0433
Volume/Issue:
Volume 233: Issue 1
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C García-Luna Department of Neurosciences Research, Molecular Neurophysiology Laboratory, National Institute of Psychiatry Ramón de la Fuente Muñiz, Mexico City, Mexico

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P Soberanes-Chávez Department of Neurosciences Research, Molecular Neurophysiology Laboratory, National Institute of Psychiatry Ramón de la Fuente Muñiz, Mexico City, Mexico

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P de Gortari Department of Neurosciences Research, Molecular Neurophysiology Laboratory, National Institute of Psychiatry Ramón de la Fuente Muñiz, Mexico City, Mexico

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concentrations in rats . Physiology and Behavior 40 323 – 328 . ( doi:10.1016/0031-9384(87)90054-0 ) de Gortari P Mengod G 2010 Dopamine D1, D2 and mu-opioid receptors are co-expressed with adenylyl cyclase 5 and phosphodiesterase 7B mRNAs in

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DOI:
https://doi.org/10.1530/JOE-16-0402
Volume/Issue:
Volume 232: Issue 1
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Francesca Spiga Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology, College of Engineering, Wellcome Trust Centre for Biomedical Modelling and Analysis, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK

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Jamie J Walker Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology, College of Engineering, Wellcome Trust Centre for Biomedical Modelling and Analysis, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK
Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology, College of Engineering, Wellcome Trust Centre for Biomedical Modelling and Analysis, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK
Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology, College of Engineering, Wellcome Trust Centre for Biomedical Modelling and Analysis, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK

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Rita Gupta Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology, College of Engineering, Wellcome Trust Centre for Biomedical Modelling and Analysis, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK

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John R Terry Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology, College of Engineering, Wellcome Trust Centre for Biomedical Modelling and Analysis, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK
Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology, College of Engineering, Wellcome Trust Centre for Biomedical Modelling and Analysis, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK

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Stafford L Lightman Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology, College of Engineering, Wellcome Trust Centre for Biomedical Modelling and Analysis, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK

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specific cell surface G-protein-coupled receptor, the melanocortin type-2 receptor (MC2R; Mountjoy et al . 1994 ). ACTH binding to MC2R leads to activation of adenylyl cyclase, followed by an increase in intracellular levels of cAMP, which in turn

Article Type:
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DOI:
https://doi.org/10.1530/JOE-15-0132
Volume/Issue:
Volume 226: Issue 2
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Sushil K Mahata VA San Diego Healthcare System Metabolic Physiology & Ultrastructural Biology Lab., Department of Medicine, University of California at San Diego, La Jolla, CA, USA

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Hong Zheng Department of Cellular and Integrative Physiology, University of Nebraska Medical Center, Omaha, NE, USA

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Sumana Mahata Caltech Division of Biology, California Institute of Technology, Pasadena, CA, USA

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Xuefei Liu Department of Cellular and Integrative Physiology, University of Nebraska Medical Center, Omaha, NE, USA

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Kaushik P Patel Department of Cellular and Integrative Physiology, University of Nebraska Medical Center, Omaha, NE, USA

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secretion in chromaffin cells. Cis and trans signaling determinants of pituitary adenylyl cyclase-activating polypeptide (PACAP) . Journal of Clinical Investigation 101 863 – 876 . ( doi:10.1172/JCI1129 ) Tomaszek A Kiczak L Bania J

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https://doi.org/10.1530/JOE-16-0146
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Volume 230: Issue 3
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William F Powell Jr
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Kevin J Barry
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Irena Tulum
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Tatsuya Kobayashi
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Stephen E Harris Endocrine Unit, Department of Periodontics, Massachusetts General Hospital and Harvard Medical School, Thier 1101, 50 Blossom Street, Boston, Massachusetts 02114, USA

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F Richard Bringhurst
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Paola Divieti Pajevic
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=36.7±23.9 picomol/mg bone) indicating an intact adenylyl cyclase machinery in these osteocyte-enriched bone fragments. To assess that PPR ablation was confined to osteocytes and not to osteoblasts, we performed cAMP accumulation in response to PTH in

Article Type:
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DOI:
https://doi.org/10.1530/JOE-10-0308
Volume/Issue:
Volume 209: Issue 1
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Toshihiro Sakurai Department of Endocrine Pharmacology, Tokyo University of Pharmacy and Life Sciences, Horinouchi 1432-1, Hachioji, Tokyo 192-0392, Japan

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Kenta Suzuki Department of Endocrine Pharmacology, Tokyo University of Pharmacy and Life Sciences, Horinouchi 1432-1, Hachioji, Tokyo 192-0392, Japan

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Mikihiro Yoshie Department of Endocrine Pharmacology, Tokyo University of Pharmacy and Life Sciences, Horinouchi 1432-1, Hachioji, Tokyo 192-0392, Japan

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Keisuke Hashimoto Department of Endocrine Pharmacology, Tokyo University of Pharmacy and Life Sciences, Horinouchi 1432-1, Hachioji, Tokyo 192-0392, Japan

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Eiichi Tachikawa Department of Endocrine Pharmacology, Tokyo University of Pharmacy and Life Sciences, Horinouchi 1432-1, Hachioji, Tokyo 192-0392, Japan

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Kazuhiro Tamura Department of Endocrine Pharmacology, Tokyo University of Pharmacy and Life Sciences, Horinouchi 1432-1, Hachioji, Tokyo 192-0392, Japan

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tube formation in each treatment. Additional experiment has shown that H89, a PKA inhibitor, clearly blocked PGE 2 -induced tube formation as well as forskolin (an adenylyl cyclase activator)-induced changes. Figure 3 Effects of the prostanoid EP

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DOI:
https://doi.org/10.1530/JOE-10-0357
Volume/Issue:
Volume 209: Issue 1
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Lu Fu Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China


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Hongyuan Zhang Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China
Institute of Cardiovascular Disease Research, Xuzhou Medical University, Xuzhou, Jiangsu, China


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Jeremiah Ong’achwa Machuki Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China

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Tingting Zhang Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China
Institute of Cardiovascular Disease Research, Xuzhou Medical University, Xuzhou, Jiangsu, China

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Lin Han Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China

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Lili Sang Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China

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Lijuan Wu Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China
Institute of Cardiovascular Disease Research, Xuzhou Medical University, Xuzhou, Jiangsu, China

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Zhiwei Zhao Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China
Institute of Cardiovascular Disease Research, Xuzhou Medical University, Xuzhou, Jiangsu, China

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Matthew James Turley National Heart and Lung Institute, Imperial College London, UK

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Xide Hu Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China

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Hongjian Hou Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China

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Dongye Li Institute of Cardiovascular Disease Research, Xuzhou Medical University, Xuzhou, Jiangsu, China

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Sian E Harding National Heart and Lung Institute, Imperial College London, UK

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Hong Sun Physiology Department, Xuzhou Medical University, Xuzhou, Jiangsu, China

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X Ma X Sun H 2019 Pathological cardiac hypertrophy: the synergy of adenylyl cyclases inhibition in cardiac and immune cells during chronic catecholamine stress . Journal of Molecular Medicine 97 897 – 907 . ( https://doi.org/10.1007/s00109

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DOI:
https://doi.org/10.1530/JOE-20-0451
Volume/Issue:
Volume 249: Issue 3
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Claire V Hutchinson
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James A Walker College of Medicine, Harvard Medical School, Division of Biomedical Sciences, Biological Sciences and Psychology, University of Leicester, Leicester LE1 9HN, UK

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Colin Davidson College of Medicine, Harvard Medical School, Division of Biomedical Sciences, Biological Sciences and Psychology, University of Leicester, Leicester LE1 9HN, UK

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: oestrogen has a higher affinity for ERα than ERβ. AC, adenylyl cyclase; PKA, protein kinase A, MMP, metalloproteinase, PI3K, phosphoinositide 3-kinase, GSK3β, glycogen synthase kinase 3β. G15 is a GPER antagonist. A full colour version of this figure

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https://doi.org/10.1530/JOE-14-0349
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Volume 223: Issue 2
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Jing Lu Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA
Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA

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Joshua Reese Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA

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Ying Zhou Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA

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Emmet Hirsch Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA
Department of OB/GYN, Department of OB/GYN, NorthShore University HealthSystem, 2650 Ridge Avenue, Evanston, Illinois 60201, USA

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receptor ( Blackmore et al . 1991 ). In a cell model system of human parafollicular cells, P4BSA increases the release of calcitonin through activation of adenylyl cyclase and PKA ( Lu & Tsai 2007 ). Flock et al . (2013) reported that activation of mPRs

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https://doi.org/10.1530/JOE-14-0470
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Volume 224: Issue 2
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F Aréchiga-Ceballos Neurofisiología Molecular, Escuela de Dietética y Nutrición, Dirección de Investigaciones en Neurociencias, Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz (INPRFM), Calzada México-Xochimilco 101, Col. San Lorenzo Huipulco, C.P. 14370, México, Distrito Federal, México
Neurofisiología Molecular, Escuela de Dietética y Nutrición, Dirección de Investigaciones en Neurociencias, Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz (INPRFM), Calzada México-Xochimilco 101, Col. San Lorenzo Huipulco, C.P. 14370, México, Distrito Federal, México

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E Alvarez-Salas Neurofisiología Molecular, Escuela de Dietética y Nutrición, Dirección de Investigaciones en Neurociencias, Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz (INPRFM), Calzada México-Xochimilco 101, Col. San Lorenzo Huipulco, C.P. 14370, México, Distrito Federal, México

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G Matamoros-Trejo Neurofisiología Molecular, Escuela de Dietética y Nutrición, Dirección de Investigaciones en Neurociencias, Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz (INPRFM), Calzada México-Xochimilco 101, Col. San Lorenzo Huipulco, C.P. 14370, México, Distrito Federal, México

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M I Amaya Neurofisiología Molecular, Escuela de Dietética y Nutrición, Dirección de Investigaciones en Neurociencias, Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz (INPRFM), Calzada México-Xochimilco 101, Col. San Lorenzo Huipulco, C.P. 14370, México, Distrito Federal, México

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C García-Luna Neurofisiología Molecular, Escuela de Dietética y Nutrición, Dirección de Investigaciones en Neurociencias, Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz (INPRFM), Calzada México-Xochimilco 101, Col. San Lorenzo Huipulco, C.P. 14370, México, Distrito Federal, México

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P de Gortari Neurofisiología Molecular, Escuela de Dietética y Nutrición, Dirección de Investigaciones en Neurociencias, Instituto Nacional de Psiquiatría Ramón de la Fuente Muñiz (INPRFM), Calzada México-Xochimilco 101, Col. San Lorenzo Huipulco, C.P. 14370, México, Distrito Federal, México

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.brainres.2009.07.094 ) de Gortari P Mengod G 2010 Dopamine D1, D2 and mu-opioid receptors are co-expressed with adenylyl cyclase 5 and phosphodiesterase 7B mRNAs in striatal rat cells . Brain Research 1310 37 – 45 . ( doi:10.1016/j.brainres.2009

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DOI:
https://doi.org/10.1530/JOE-13-0458
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Volume 221: Issue 1
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