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Malin Hedengran Faulds
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Chunyan Zhao
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Karin Dahlman-Wright
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Jan-Åke Gustafsson Department of Biosciences and Nutrition, Center for Nuclear Receptors and Cell Signaling, Novum, Karolinska Institutet, S-141 83 Huddinge, Sweden

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-Ludwig A Clemenz M Hohmann S Hartge M Sprang C Frost N Krikov M Bhanot S Barros R Morani A 2008 Metabolic actions of estrogen receptor beta (ERbeta) are mediated by a negative cross-talk with PPARgamma . PLoS Genetics 4 e1000108

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-11-0044
Volume/Issue:
Volume 212: Issue 1
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Aarti D Rohira Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA

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David M Lonard Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA

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Bert W O’Malley Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA

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-epithelium paracrine signaling Paracrine signaling between stromal and epithelial cells by cytokines, growth factors and hormones have been shown to regulate the crosstalk between tissue stroma and parenchyma. Previous studies have shown that steroid hormones such as

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-20-0511
Volume/Issue:
Volume 248: Issue 2
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Elizabeth I Tang The Mary M. Wohlford Laboratory for Male Contraceptive Research, Center for Biomedical Research, Population Council, 1230 York Avenue, New York, New York 10065, USA

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Dolores D Mruk The Mary M. Wohlford Laboratory for Male Contraceptive Research, Center for Biomedical Research, Population Council, 1230 York Avenue, New York, New York 10065, USA

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C Yan Cheng The Mary M. Wohlford Laboratory for Male Contraceptive Research, Center for Biomedical Research, Population Council, 1230 York Avenue, New York, New York 10065, USA

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Stehbens SJ Yap AS 2009 Touch, grasp, deliver and control: functional cross-talk between microtubules and cell adhesions . Traffic 10 268 – 274 . ( doi:10.1111/j.1600-0854.2008.00869.x ) Al-Bassam J Chang F 2011 Regulation of

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https://doi.org/10.1530/JOE-12-0586
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Volume 217: Issue 2
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Salvatore Valiante
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Marina Prisco
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Rosaria Sciarrillo Dipartimento delle Scienze Biologiche, Dipartimento di Scienze Biologiche ed Ambientali, Sezione di Biologia Evolutiva e Comparata, Università Federico II di Napoli, Via Mezzocannone, 8, 80134 Napoli, Italy

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Maria De Falco
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Anna Capaldo
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Flaminia Gay
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Piero Andreuccetti
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Vincenza Laforgia
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Lorenzo Varano
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antagonists, on in vitro cell co-cultures in order to clarify the role of these peptides in the crosstalk between chromaffin and steroidogenic cells. Materials and Methods Animals The animals (number=50; 14–16 g body weight each) were housed in

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0127
Volume/Issue:
Volume 196: Issue 2
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Liyuan Tian Molecular Biology, Endocrinology, Departments of

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Zhiqiang Wu Molecular Biology, Endocrinology, Departments of

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Yali Zhao Molecular Biology, Endocrinology, Departments of

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Yuanguang Meng Molecular Biology, Endocrinology, Departments of

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Yiling Si Molecular Biology, Endocrinology, Departments of

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Xiaobing Fu Molecular Biology, Endocrinology, Departments of

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Yiming Mu Molecular Biology, Endocrinology, Departments of

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Weidong Han Molecular Biology, Endocrinology, Departments of

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involvement of LRP16 in the classic estrogen-activated signaling suggests crosstalk between estrogen-repressed and estrogen-activated pathways in estrogen-insensitive ovarian cancer cells. The crosstalk between different ERα-mediated signaling pathways was

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0054
Volume/Issue:
Volume 202: Issue 1
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Mina Takahashi Medicine and Clinical Science, Departments of

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Fumio Otsuka Medicine and Clinical Science, Departments of

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Tomoko Miyoshi Medicine and Clinical Science, Departments of

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Hiroyuki Otani Medicine and Clinical Science, Departments of

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Junko Goto Medicine and Clinical Science, Departments of

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Misuzu Yamashita Medicine and Clinical Science, Departments of

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Toshio Ogura Medicine and Clinical Science, Departments of

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Hirofumi Makino Medicine and Clinical Science, Departments of

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Hiroyoshi Doihara Medicine and Clinical Science, Departments of

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et al . 2002 ), a functional crosstalk may exist between the breast cancer BMP system, ER signal, and MAPK pathway. In this regard, Yamamoto et al . (2002) reported that estrogen inhibition on BMP2-induced SMAD signaling was due to the direct

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0226
Volume/Issue:
Volume 199: Issue 3
Free access
S Giannini Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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S Benvenuti Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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P Luciani Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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C Manuelli Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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I Cellai Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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C Deledda Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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A Pezzatini Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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G B Vannelli Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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E Maneschi Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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C M Rotella Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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M Serio Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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A Peri Diabetes and Metabolic Diseases Unit, Endocrine Unit, Institute of Dermatology and Venereology, Department of Anatomy, Department of Clinical Physiopathology, Center for Research, Transfer and High Education on Chronic, Inflammatory, Degenerative and Neoplastic Disorders for the Development of Novel Therapies (DENOThe)

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( Fig. 4 ). According to previous evidence that a cross-talk between the IGF system and estrogen in the brain occurs (Mendez et al . 2003, 2006 ), the effect of 17β-estradiol on IGF1 production was determined. The exposure of FNC to 17β-estradiol (10

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0613
Volume/Issue:
Volume 198: Issue 3
Free access
José E Sánchez-Criado Department of Cell Biology, Physiology and Immunology, University of Córdoba, Avda. Menendez Pidal s/n, 14004 Córdoba, Spain
Department of Physiology, University of La Laguna, La Laguna, Spain
Department of Comparative Pathology, University of Córdoba, Córdoba, Spain

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José C Garrido-Gracia Department of Cell Biology, Physiology and Immunology, University of Córdoba, Avda. Menendez Pidal s/n, 14004 Córdoba, Spain
Department of Physiology, University of La Laguna, La Laguna, Spain
Department of Comparative Pathology, University of Córdoba, Córdoba, Spain

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Carmina Bellido Department of Cell Biology, Physiology and Immunology, University of Córdoba, Avda. Menendez Pidal s/n, 14004 Córdoba, Spain
Department of Physiology, University of La Laguna, La Laguna, Spain
Department of Comparative Pathology, University of Córdoba, Córdoba, Spain

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Rafaela Aguilar Department of Cell Biology, Physiology and Immunology, University of Córdoba, Avda. Menendez Pidal s/n, 14004 Córdoba, Spain
Department of Physiology, University of La Laguna, La Laguna, Spain
Department of Comparative Pathology, University of Córdoba, Córdoba, Spain

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Pedro Guelmes Department of Cell Biology, Physiology and Immunology, University of Córdoba, Avda. Menendez Pidal s/n, 14004 Córdoba, Spain
Department of Physiology, University of La Laguna, La Laguna, Spain
Department of Comparative Pathology, University of Córdoba, Córdoba, Spain

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Pedro Abreu Department of Cell Biology, Physiology and Immunology, University of Córdoba, Avda. Menendez Pidal s/n, 14004 Córdoba, Spain
Department of Physiology, University of La Laguna, La Laguna, Spain
Department of Comparative Pathology, University of Córdoba, Córdoba, Spain

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Rafael Alonso Department of Cell Biology, Physiology and Immunology, University of Córdoba, Avda. Menendez Pidal s/n, 14004 Córdoba, Spain
Department of Physiology, University of La Laguna, La Laguna, Spain
Department of Comparative Pathology, University of Córdoba, Córdoba, Spain

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Inmaculada Barranco Department of Cell Biology, Physiology and Immunology, University of Córdoba, Avda. Menendez Pidal s/n, 14004 Córdoba, Spain
Department of Physiology, University of La Laguna, La Laguna, Spain
Department of Comparative Pathology, University of Córdoba, Córdoba, Spain

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Yolanda Millán Department of Cell Biology, Physiology and Immunology, University of Córdoba, Avda. Menendez Pidal s/n, 14004 Córdoba, Spain
Department of Physiology, University of La Laguna, La Laguna, Spain
Department of Comparative Pathology, University of Córdoba, Córdoba, Spain

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Juana Martín de las Mulas Department of Cell Biology, Physiology and Immunology, University of Córdoba, Avda. Menendez Pidal s/n, 14004 Córdoba, Spain
Department of Physiology, University of La Laguna, La Laguna, Spain
Department of Comparative Pathology, University of Córdoba, Córdoba, Spain

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4 , elicits LHRH self-priming. While the oestrogen-induced LHRH self-priming involves intracellular cAMP/PKA crosstalk with PR ( Turgeon & Waring 1991 , Waring & Turgeon 1992 ), the mechanism of TX-induced LHRH self-priming is not completely known

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06654
Volume/Issue:
Volume 190: Issue 1
Free access
Ralf Baumeister Bio 3, Bioinformatics and Molecular Genetics, University of Freiburg, Germany
ZBSA – Freiburg Center for Systems Biology, University of Freiburg, Germany
Renal Division, University Hospital Freiburg, Germany

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Elke Schaffitzel Bio 3, Bioinformatics and Molecular Genetics, University of Freiburg, Germany
ZBSA – Freiburg Center for Systems Biology, University of Freiburg, Germany
Renal Division, University Hospital Freiburg, Germany

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Maren Hertweck Bio 3, Bioinformatics and Molecular Genetics, University of Freiburg, Germany
ZBSA – Freiburg Center for Systems Biology, University of Freiburg, Germany
Renal Division, University Hospital Freiburg, Germany

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crosstalks with the insulin pathway and acts as a downstream effector of DAF-2 to determine longevity ( Hsu et al. 2003 ), while a regulatory link between SKN-1 and the DAF-2/DAF-16 pathway still needs to be established. Such an interaction, however, is

Article Type:
Review Article
DOI:
https://doi.org/10.1677/joe.1.06856
Volume/Issue:
Volume 190: Issue 2
Free access
Giampaolo Trivellin Department of Endocrinology, Endocrinology Division, Bart's and the London School of Medicine, Queen Mary University of London, London EC1M 6BQ, UK
Department of Endocrinology, Endocrinology Division, Bart's and the London School of Medicine, Queen Mary University of London, London EC1M 6BQ, UK

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Márta Korbonits Department of Endocrinology, Endocrinology Division, Bart's and the London School of Medicine, Queen Mary University of London, London EC1M 6BQ, UK

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receptor (ERα and ERβ) and the androgen receptor (AR). The crosstalk of AhR with ER and AR explicates via a direct association in the nucleus, which modulates oestrogen/androgen signalling both positively and negatively depending on the cellular context

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-11-0054
Volume/Issue:
Volume 210: Issue 2
Free access