Search Results
Search for other papers by I. D. Gilham in
Google Scholar
PubMed
Search for other papers by B. I. Baker in
Google Scholar
PubMed
ABSTRACT
This work examines the difference in responsiveness to stress which characterizes fish adapted to white and black backgrounds. Trout were maintained in black or white tanks for 2 weeks and then subjected to intermittent intense or moderate noise stress for periods between 1 h and 5 days, or to the stress of being injected daily with a large volume of liquid for 3 days. Plasma cortisol concentrations increased more readily and to a greater extent in fish from black tanks in response to moderate stress or brief intense stress. Dexamethasone suppressed the stress-induced rise of cortisol in white-adapted fish but was only partially effective in trout from black backgrounds. These differences in plasma cortisol between black- and white-adapted fish can be related to the different titres of plasma ACTH, apparently derived from the pars distalis.
Removal of the neurointermediate lobe (NIL) from black-adapted eels markedly depressed the normal rise in plasma cortisol elicited by noise stress. It is suggested that products from the NIL may modulate the stress response of the hypothalamo-pituitary axis although other routes through which background colour could affect the pituitary responsiveness to stress are also considered. In several cases, stress also enhanced the secretion of MSH from the NIL.
J. Endocr. (1985) 105, 99–105
Central Research Laboratory, Sanwa Kagaku Kenkyusho Co. Ltd, 363 Shiosaki, Hokusei, Inabe, Mie 511-0406, Japan
Department of Pharmacology, Oita University Faculty of Medicine, 1-1 Idaigaoka, Hasama, Yufu, Oita 879-5593, Japan
Search for other papers by Shin Tsunekawa in
Google Scholar
PubMed
Central Research Laboratory, Sanwa Kagaku Kenkyusho Co. Ltd, 363 Shiosaki, Hokusei, Inabe, Mie 511-0406, Japan
Department of Pharmacology, Oita University Faculty of Medicine, 1-1 Idaigaoka, Hasama, Yufu, Oita 879-5593, Japan
Search for other papers by Naoki Yamamoto in
Google Scholar
PubMed
Central Research Laboratory, Sanwa Kagaku Kenkyusho Co. Ltd, 363 Shiosaki, Hokusei, Inabe, Mie 511-0406, Japan
Department of Pharmacology, Oita University Faculty of Medicine, 1-1 Idaigaoka, Hasama, Yufu, Oita 879-5593, Japan
Search for other papers by Katsura Tsukamoto in
Google Scholar
PubMed
Central Research Laboratory, Sanwa Kagaku Kenkyusho Co. Ltd, 363 Shiosaki, Hokusei, Inabe, Mie 511-0406, Japan
Department of Pharmacology, Oita University Faculty of Medicine, 1-1 Idaigaoka, Hasama, Yufu, Oita 879-5593, Japan
Search for other papers by Yuji Itoh in
Google Scholar
PubMed
Central Research Laboratory, Sanwa Kagaku Kenkyusho Co. Ltd, 363 Shiosaki, Hokusei, Inabe, Mie 511-0406, Japan
Department of Pharmacology, Oita University Faculty of Medicine, 1-1 Idaigaoka, Hasama, Yufu, Oita 879-5593, Japan
Search for other papers by Yukiko Kaneko in
Google Scholar
PubMed
Central Research Laboratory, Sanwa Kagaku Kenkyusho Co. Ltd, 363 Shiosaki, Hokusei, Inabe, Mie 511-0406, Japan
Department of Pharmacology, Oita University Faculty of Medicine, 1-1 Idaigaoka, Hasama, Yufu, Oita 879-5593, Japan
Search for other papers by Toshihide Kimura in
Google Scholar
PubMed
Central Research Laboratory, Sanwa Kagaku Kenkyusho Co. Ltd, 363 Shiosaki, Hokusei, Inabe, Mie 511-0406, Japan
Department of Pharmacology, Oita University Faculty of Medicine, 1-1 Idaigaoka, Hasama, Yufu, Oita 879-5593, Japan
Search for other papers by Yoh Ariyoshi in
Google Scholar
PubMed
Central Research Laboratory, Sanwa Kagaku Kenkyusho Co. Ltd, 363 Shiosaki, Hokusei, Inabe, Mie 511-0406, Japan
Department of Pharmacology, Oita University Faculty of Medicine, 1-1 Idaigaoka, Hasama, Yufu, Oita 879-5593, Japan
Search for other papers by Yoshitaka Miura in
Google Scholar
PubMed
Central Research Laboratory, Sanwa Kagaku Kenkyusho Co. Ltd, 363 Shiosaki, Hokusei, Inabe, Mie 511-0406, Japan
Department of Pharmacology, Oita University Faculty of Medicine, 1-1 Idaigaoka, Hasama, Yufu, Oita 879-5593, Japan
Search for other papers by Yutaka Oiso in
Google Scholar
PubMed
Central Research Laboratory, Sanwa Kagaku Kenkyusho Co. Ltd, 363 Shiosaki, Hokusei, Inabe, Mie 511-0406, Japan
Department of Pharmacology, Oita University Faculty of Medicine, 1-1 Idaigaoka, Hasama, Yufu, Oita 879-5593, Japan
Search for other papers by Ichiro Niki in
Google Scholar
PubMed
participate in the pathogenesis of diabetes mellitus ( Donath et al. 1999 , Mandrup-Poulsen 2003 , Scheuner et al. 2005 ). Among them, more attention has recently been directed to endoplasmic reticulum (ER) stress. ER stress is a series of cellular
Search for other papers by F. E. BADRICK in
Google Scholar
PubMed
Search for other papers by R. W. BRIMBLECOMBE in
Google Scholar
PubMed
Search for other papers by M. REISS in
Google Scholar
PubMed
SUMMARY
Rats stressed by swimming in cold water (15° C) show a considerably decreased uptake of 32P by the thyroid gland, which is comparable with the decrease in the uptake of 131I. The 32P uptake of the adrenals is increased.
These changes are independent of the anterior lobe of the pituitary, since they also occur after hypophysectomy. It is assumed that they are caused by the increased production of vaso-constrictor substances, since treatment with adrenaline produced identical changes in the function of thyroid and adrenals in both intact and hypophysectomized rats.
The significance of these changes in formulating a concept of the physiological reaction of the organism to conditions of stress is discussed.
Search for other papers by David A Baltzegar in
Google Scholar
PubMed
Search for other papers by Benjamin J Reading in
Google Scholar
PubMed
Search for other papers by Jonathon D Douros in
Google Scholar
PubMed
Search for other papers by Russell J Borski in
Google Scholar
PubMed
discrete roles of mobilizing energy during periods of stress or catabolism among vertebrate groups. Two leptin paralogs ( lepa and lepb ) exist in many teleosts, suggesting genome duplication as a potential mechanism of functional divergence ( Gorissen
Search for other papers by Parveen Abidi in
Google Scholar
PubMed
Search for other papers by Haiyan Zhang in
Google Scholar
PubMed
Search for other papers by Syed M Zaidi in
Google Scholar
PubMed
Search for other papers by Wen-Jun Shen in
Google Scholar
PubMed
Search for other papers by Susan Leers-Sucheta in
Google Scholar
PubMed
Search for other papers by Yuan Cortez in
Google Scholar
PubMed
Search for other papers by Jiahuai Han in
Google Scholar
PubMed
Department of Veterans Affairs Palo Alto Health Care System, Division of Gastroenterology and Hepatology, Department of Immunology, Geriatric Research, Education and Clinical Center (GRECC), 3801 Miranda Avenue, Palo Alto, California 94304, USA
Search for other papers by Salman Azhar in
Google Scholar
PubMed
steroidogenic function is accompanied by a significant alteration in oxidative status of rat adrenal and testicular tissues including enhanced oxidative stress, loss of enzymatic and non-enzymatic antioxidants and increased membrane lipid peroxidation ( Azhar
Mercy Perinatal Research Centre, Translational Proteomics, Department of Obstetrics and Gynaecology
Mercy Perinatal Research Centre, Translational Proteomics, Department of Obstetrics and Gynaecology
Search for other papers by Martha Lappas in
Google Scholar
PubMed
Mercy Perinatal Research Centre, Translational Proteomics, Department of Obstetrics and Gynaecology
Search for other papers by Amberlee Mittion in
Google Scholar
PubMed
Mercy Perinatal Research Centre, Translational Proteomics, Department of Obstetrics and Gynaecology
Search for other papers by Michael Permezel in
Google Scholar
PubMed
a reduced capacity to respond to oxidative stress in terms of 8-isoprostane (marker of lipid peroxidation) and tumour necrosis factor α (TNFα) release ( Coughlan et al . 2004 b ). We concluded that GDM placenta may be pre-conditioned by transient
Search for other papers by Kotaro Horiguchi in
Google Scholar
PubMed
Search for other papers by Tom Kouki in
Google Scholar
PubMed
Search for other papers by Ken Fujiwara in
Google Scholar
PubMed
Search for other papers by Takehiro Tsukada in
Google Scholar
PubMed
Search for other papers by Floren Ly in
Google Scholar
PubMed
Search for other papers by Motoshi Kikuchi in
Google Scholar
PubMed
Search for other papers by Takashi Yashiro in
Google Scholar
PubMed
identify the mechanism responsible for the extension of FS cell cytoplasmic processes. First, we observed stress fiber formation in FS cells in the presence of laminin, an ECM component of the basement membrane. Stress fibers are composed of bundles of 10
Search for other papers by Chad Osterlund in
Google Scholar
PubMed
Search for other papers by Robert L Spencer in
Google Scholar
PubMed
negative feedback function associated with a wide range of clinical disorders (e.g. depression, post-traumatic stress disorder, type II diabetes, chronic fatigue syndrome, fibromyalgia, and chronic facial pain) and associated precursor conditions (e
Search for other papers by Qingling Huang in
Google Scholar
PubMed
Search for other papers by Elena Timofeeva in
Google Scholar
PubMed
Search for other papers by Denis Richard in
Google Scholar
PubMed
). After a session of treadmill running, about all PVN CRFergic neurons express the immediate early gene c-fos , a marker of persisting neuronal activity ( Timofeeva et al. 2003 ). Besides its stress-related hypophysiotropic effects, CRF blunts energy
Division of Endocrinology, Department of Endocrinology and Diabetology, Department of Pediatrics, Laboratory of Clinical Investigations, Department of Medicine, Washington Hospital Center, 110 Irving Street Northwest, Washington, District of Columbia 20010-2910, USA
Search for other papers by Joanna Klubo-Gwiezdzinska in
Google Scholar
PubMed
Search for other papers by Kirk Jensen in
Google Scholar
PubMed
Search for other papers by Andrew Bauer in
Google Scholar
PubMed
Search for other papers by Aneeta Patel in
Google Scholar
PubMed
Search for other papers by John Costello Jr in
Google Scholar
PubMed
Search for other papers by Kenneth D Burman in
Google Scholar
PubMed
Search for other papers by Leonard Wartofsky in
Google Scholar
PubMed
Search for other papers by Matthew J Hardwick in
Google Scholar
PubMed
Search for other papers by Vasyl V Vasko in
Google Scholar
PubMed
Coulter (Fullerton, CA, USA). For induction of oxidative stress, thyroid cancer cells were treated with H 2 O 2 . H 2 O 2 stock solution (100 mM) was added into cell culture medium to achieve the desired working concentration. For TSPO ligand studies, PK