Search Results
Search for other papers by Kristien Vandenborne in
Google Scholar
PubMed
Search for other papers by Bert De Groef in
Google Scholar
PubMed
Search for other papers by Sofie M E Geelissen in
Google Scholar
PubMed
Search for other papers by Eduard R Kühn in
Google Scholar
PubMed
Search for other papers by Veerle M Darras in
Google Scholar
PubMed
Search for other papers by Serge Van der Geyten in
Google Scholar
PubMed
expression via the glucocorticoid receptor (GR) ( Kretz et al. 1999 , Feldman & Weidenfeld 2002 ). Glucocorticoids have been found to regulate CRF mRNA at two sites, by inhibiting gene transcription and by decreasing the mRNA stability ( Ma et al. 2001
IRGS, Biogem, Ariano Irpino, Avellino, Italy
Search for other papers by Marco Colella in
Google Scholar
PubMed
Search for other papers by Valeria Nittoli in
Google Scholar
PubMed
Search for other papers by Alfonsina Porciello in
Google Scholar
PubMed
Search for other papers by Immacolata Porreca in
Google Scholar
PubMed
Search for other papers by Carla Reale in
Google Scholar
PubMed
Search for other papers by Filomena Russo in
Google Scholar
PubMed
Search for other papers by Nicola Antonino Russo in
Google Scholar
PubMed
Search for other papers by Luca Roberto in
Google Scholar
PubMed
IEOS-CNR, Naples, Italy
Search for other papers by Francesco Albano in
Google Scholar
PubMed
Molecular Medicine and Medical Biotechnologies, University of Naples ‘Federico II’, Naples, Italy
Search for other papers by Mario De Felice in
Google Scholar
PubMed
Search for other papers by Massimo Mallardo in
Google Scholar
PubMed
IRGS, Biogem, Ariano Irpino, Avellino, Italy
IEOS-CNR, Naples, Italy
Search for other papers by Concetta Ambrosino in
Google Scholar
PubMed
tissue/cell-specific levels of T 3 and, finally, on TH receptors (THRs). Local T 3 levels depend on an ensemble of tissue/cell-specific factors, such as deiodinases and TH transporters, which is relatively independent of serum TH levels ( Colella et al
Search for other papers by Cun Li in
Google Scholar
PubMed
Search for other papers by Thomas J McDonald in
Google Scholar
PubMed
Search for other papers by Guoyao Wu in
Google Scholar
PubMed
Search for other papers by Mark J Nijland in
Google Scholar
PubMed
Search for other papers by Peter W Nathanielsz in
Google Scholar
PubMed
ARH orexigenic:anorexigenic peptide balance; iii) in the setting of the increased fetal cortisol levels, we have previously described ( Nijland et al . 2010 ) that IUGR increases fetal ARH glucocorticoid receptor (GR) expression; and iv) as STAT3
Max Planck Institute for Experimental Endocrinology, Department of Cell and Molecular Biology, Fritz-Lipmann-Institut, Institute of Biomedicine, Institute for Biochemistry, University of East Anglia, Feodor-Lynen-Str. 7, D-30625 Hannover, Germany
Search for other papers by Jens Mittag in
Google Scholar
PubMed
Search for other papers by Wiebke Oehr in
Google Scholar
PubMed
Search for other papers by Heike Heuer in
Google Scholar
PubMed
Search for other papers by Tuula Hämäläinen in
Google Scholar
PubMed
Search for other papers by Bent Brachvogel in
Google Scholar
PubMed
Search for other papers by Ernst Pöschl in
Google Scholar
PubMed
Max Planck Institute for Experimental Endocrinology, Department of Cell and Molecular Biology, Fritz-Lipmann-Institut, Institute of Biomedicine, Institute for Biochemistry, University of East Anglia, Feodor-Lynen-Str. 7, D-30625 Hannover, Germany
Search for other papers by Karl Bauer in
Google Scholar
PubMed
indicated that A5 not only enhances gonadotropin secretion following stimulation by gonadotrophin-releasing hormone (GnRH; Kawaminami et al . 2002 ), but also stimulates basal PRL secretion while counteracting thyrotrophin-releasing hormone (TRH) induced
Search for other papers by Asghar Ali in
Google Scholar
PubMed
Search for other papers by Callie M Swanepoel in
Google Scholar
PubMed
Search for other papers by Quinton A Winger in
Google Scholar
PubMed
Search for other papers by Paul J Rozance in
Google Scholar
PubMed
Search for other papers by Russell V Anthony in
Google Scholar
PubMed
). CSH has high affinity for prolactin (PRL) receptors as well as growth hormone (GH) receptors ( Handwerger & Freemark 2000 ), although a distinct CSH receptor may exist in fetal liver ( Freemark et al. 1987 , Hill et al. 1988 , Pratt et al. 1995
Search for other papers by I M McGonnell in
Google Scholar
PubMed
Search for other papers by R C Fowkes in
Google Scholar
PubMed
2005 ). In fish, as in other non-mammalian vertebrates, corticotrophin-releasing factor (CRF) stimulates not only corticotrophin release from the pituitary but also thyrotrophin release ( Seasholtz et al. 2002 ). Thyrotrophin-releasing hormone (TRH
Search for other papers by E Oliveira in
Google Scholar
PubMed
Search for other papers by E G Moura in
Google Scholar
PubMed
Search for other papers by A P Santos-Silva in
Google Scholar
PubMed
Search for other papers by A T S Fagundes in
Google Scholar
PubMed
Search for other papers by A S Rios in
Google Scholar
PubMed
Search for other papers by Y Abreu-Villaça in
Google Scholar
PubMed
Search for other papers by J F Nogueira Neto in
Google Scholar
PubMed
Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute
Search for other papers by M C F Passos in
Google Scholar
PubMed
Search for other papers by P C Lisboa in
Google Scholar
PubMed
out by our group concerning other programing models ( Passos et al . 2004 , Bonomo et al . 2007 ), in which BW changes were not accompanied by food intake alterations. Leptin and leptin receptors are both found in skeletal muscle ( Wang et al
Department of Microbiology, Medical and Health Science Centre, University of Debrecen, Hungary
Search for other papers by E Mezosi in
Google Scholar
PubMed
Department of Microbiology, Medical and Health Science Centre, University of Debrecen, Hungary
Search for other papers by J Szabo in
Google Scholar
PubMed
Department of Microbiology, Medical and Health Science Centre, University of Debrecen, Hungary
Search for other papers by E V Nagy in
Google Scholar
PubMed
Department of Microbiology, Medical and Health Science Centre, University of Debrecen, Hungary
Search for other papers by A Borbely in
Google Scholar
PubMed
Department of Microbiology, Medical and Health Science Centre, University of Debrecen, Hungary
Search for other papers by E Varga in
Google Scholar
PubMed
Department of Microbiology, Medical and Health Science Centre, University of Debrecen, Hungary
Search for other papers by G Paragh in
Google Scholar
PubMed
Department of Microbiology, Medical and Health Science Centre, University of Debrecen, Hungary
Search for other papers by Z Varga in
Google Scholar
PubMed
Introduction Most effects of thyroid hormone are mediated by a direct modulation of gene activity via interaction of the thyroid hormone–nuclear receptor complex with specific DNA sequences ( Ojamaa et al. 1996 ). Several results
Search for other papers by Renata Lopes Araujo in
Google Scholar
PubMed
Search for other papers by Bruno Moulin de Andrade in
Google Scholar
PubMed
Search for other papers by Álvaro Souto Padron de Figueiredo in
Google Scholar
PubMed
Search for other papers by Monique Leandro da Silva in
Google Scholar
PubMed
Search for other papers by Michelle Porto Marassi in
Google Scholar
PubMed
Search for other papers by Valmara dos Santos Pereira in
Google Scholar
PubMed
Search for other papers by Eliete Bouskela in
Google Scholar
PubMed
Search for other papers by Denise P Carvalho in
Google Scholar
PubMed
controlling body mass ( Oh & Kaplan 1994 , Moreno et al . 2003 , Lanni et al . 2005 , Villicev et al . 2007 ). Decreased thyroid function seems to be related to impaired thyrotropin-releasing hormone (TRH) and thyroid-stimulating hormone (TSH) secretion
Telethon Kids Institute, Perth, Australia
Healthy Environments and Lives (HEAL) Network
Search for other papers by Caitlin S Wyrwoll in
Google Scholar
PubMed
expression of PlGF, VEGF and their receptors in a placental insufficiency model of fetal growth restriction . Journal of Physiology 550 641 – 656 . ( https://doi.org/10.1113/jphysiol.2003.039511 ) Regnault TRH Orbus RJ de Vrijer B Davidsen ML Galan HL