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cytokines such as interleukin-1β (IL-1β), tumor necrosis factor-α (TNF-α) and interferon-γ (INF-γ; Mandrup-Poulsen 2001 ). Specifically, IL-1β induces the expression of inducible nitric oxide synthase (iNOS) in islet β-cells leading to the overproduction of
Laboratoire de Biologie de la Differentation et du Development, Universite Victor Segalen Bordeaux 2, 146 rue Leo Saignat 33076 Bordeaux cedex, France
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Laboratoire de Biologie de la Differentation et du Development, Universite Victor Segalen Bordeaux 2, 146 rue Leo Saignat 33076 Bordeaux cedex, France
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Laboratoire de Biologie de la Differentation et du Development, Universite Victor Segalen Bordeaux 2, 146 rue Leo Saignat 33076 Bordeaux cedex, France
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Laboratoire de Biologie de la Differentation et du Development, Universite Victor Segalen Bordeaux 2, 146 rue Leo Saignat 33076 Bordeaux cedex, France
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Laboratoire de Biologie de la Differentation et du Development, Universite Victor Segalen Bordeaux 2, 146 rue Leo Saignat 33076 Bordeaux cedex, France
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Laboratoire de Biologie de la Differentation et du Development, Universite Victor Segalen Bordeaux 2, 146 rue Leo Saignat 33076 Bordeaux cedex, France
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Laboratoire de Biologie de la Differentation et du Development, Universite Victor Segalen Bordeaux 2, 146 rue Leo Saignat 33076 Bordeaux cedex, France
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Laboratoire de Biologie de la Differentation et du Development, Universite Victor Segalen Bordeaux 2, 146 rue Leo Saignat 33076 Bordeaux cedex, France
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Laboratoire de Biologie de la Differentation et du Development, Universite Victor Segalen Bordeaux 2, 146 rue Leo Saignat 33076 Bordeaux cedex, France
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Experimental animals The transgenic mice expressing interferon (IFN)γ in the pancreatic beta cells have been previously described ( Sarvetnick et al. 1988 , 1990 , Gu & Sarvetnick 1993 , 1994 ). The IFNγ transgenic mice used in the present study
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Salmonella typhimurium ( Edwards et al. 1991 ), T-cell proliferation ( Nagy et al. 1983 ), the secretion of interferon-γ ( Cesario et al. 1994 ) and the tumoricidal activation of macrophages ( Bernton et al. 1988 ). The restoration of PRL can reverse
Institute of Medical Biochemistry and Laboratory Diagnostics, First Faculty of Medicine and General University Hospital, Charles University, Prague, Czech Republic
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Diabetes Centre, Institute for Clinical and Experimental Medicine, Prague, Czech Republic
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Diabetes Centre, Institute for Clinical and Experimental Medicine, Prague, Czech Republic
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, facilitates cardiac regeneration after myocardial infarction IFN, interferon; IL, interleukin; MMe macrophages, metabolically activated macrophages; NK cells, natural killer cells; NKT cells, natural killer T cells; Tc cells, T cytotoxic cells; Th
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mediating these changes are incompletely understood. In vitro studies have shown that interleukin (IL)-1β, interferon-γ and IL-6 decrease D1 promoter activity in the hepatocellular carcinoma cell line (HepG2), IL-1β being the most potent ( Jakobs
Department of Zoology, University of Hong Kong, Hong Kong, China
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Department of Zoology, University of Hong Kong, Hong Kong, China
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Department of Zoology, University of Hong Kong, Hong Kong, China
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adhesion molecules NM_133298 Gpnmb 1.6 13.6 Glycoprotein (transmembrane) nmb Signaling molecules NM_133624 Gbp2 1.9 5.2 Guanylate-binding protein 2, interferon-inducible AW917760 1.3 7.9 Similar to myoferlin (Fer-1-like protein 3) H31511 Trim2 1.5 6
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Department of Endocrinology, Diabetology and Clinical Nutrition, University Hospital Zurich (USZ) and University of Zurich (UZH), Zürich, Switzerland
School of Cardiovascular and Metabolic Medicine and Sciences, Faculty of Life Sciences & Medicine, King’s College London, London, UK
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states, M1 and M2 ( Mantovani et al. 2004 , Gordon & Taylor 2005 ). M1 macrophages are induced by pro-inflammatory mediators, such as lipopolysaccharide (LPS) and interferon-γ (IFN-γ), and produce pro-inflammatory cytokines (TNF-α, IL-6, IL-12
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induced) 2 −1.311 0.012 LOC623121 PREDICTED: similar to Interferon-activatable protein 203 (Ifi-203) (interferon-inducible protein p203) −1.278 0.015 Ppm1f protein phosphatase 1F (PP2C domain containing) −1.271 0.031 Nme5 NME/NM23 family member 5 −1.257 0
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group. The total number of fluorescein-positive nuclei in the three sections was reported to the total number of ethidium positive nuclei and expressed as a percentage. Nitric oxide (NO . ), monocyte chemoattractant protein-1 (MCP-1) and interferon
Departments of Medicine and of Biochemistry and Molecular Genetics, University of Colorado Health Sciences Center, Denver, Colorado 80262, USA
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Departments of Medicine and of Biochemistry and Molecular Genetics, University of Colorado Health Sciences Center, Denver, Colorado 80262, USA
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Departments of Medicine and of Biochemistry and Molecular Genetics, University of Colorado Health Sciences Center, Denver, Colorado 80262, USA
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Departments of Medicine and of Biochemistry and Molecular Genetics, University of Colorado Health Sciences Center, Denver, Colorado 80262, USA
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Departments of Medicine and of Biochemistry and Molecular Genetics, University of Colorado Health Sciences Center, Denver, Colorado 80262, USA
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Departments of Medicine and of Biochemistry and Molecular Genetics, University of Colorado Health Sciences Center, Denver, Colorado 80262, USA
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Departments of Medicine and of Biochemistry and Molecular Genetics, University of Colorado Health Sciences Center, Denver, Colorado 80262, USA
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Departments of Medicine and of Biochemistry and Molecular Genetics, University of Colorado Health Sciences Center, Denver, Colorado 80262, USA
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Departments of Medicine and of Biochemistry and Molecular Genetics, University of Colorado Health Sciences Center, Denver, Colorado 80262, USA
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activation in the pituitary, and for maximal repression by Pit-1β, may reflect a requirement for multiple interactions to recruit CBP. Such a requirement has been shown for other genes such as the P31 element of the interferon-beta gene ( Yang et al. 2002