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Richard A Ehrhardt Departments of Animal Science and Large Animal Clinical Sciences, Michigan State University, East Lansing, Michigan, USA

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Andreas Foskolos Institute of Biological, Environmental and Rural Sciences, Aberystwyth University, Aberystwyth, UK

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Sarah L Giesy Department of Animal Science, Cornell University, Ithaca, New York, USA

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Stephanie R Wesolowski University of Colorado School of Medicine, Aurora, Colorado, USA

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Christopher S Krumm Department of Animal Science, Cornell University, Ithaca, New York, USA

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W Ronald Butler Department of Animal Science, Cornell University, Ithaca, New York, USA

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Susan M Quirk Department of Animal Science, Cornell University, Ithaca, New York, USA

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Matthew R Waldron Department of Animal Science, Cornell University, Ithaca, New York, USA

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Yves R Boisclair Department of Animal Science, Cornell University, Ithaca, New York, USA

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sum of reciprocal effects on production and removal. On the production side, leptin activates hypophysiotropic thyrotrophin-releasing hormone (TRH) neurons directly via its receptor, as well as indirectly by modulating the production of the

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0031
Volume/Issue:
Volume 229: Issue 2
Free access
Xianghui Meng Chinese Medicine Hospital in Linyi City, Linyi, China

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Shuhang Xu Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Jiangsu Province Academy of Traditional Chinese Medicine, Nanjing, China

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Guofang Chen Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Jiangsu Province Academy of Traditional Chinese Medicine, Nanjing, China

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Michael Derwahl Division of Endocrinology, Department of Medicine, St. Hedwig Hospital, Berlin, Germany

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Chao Liu Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Jiangsu Province Academy of Traditional Chinese Medicine, Nanjing, China

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insulin action ( Giannarelli et al. 2003 ). At the level of the liver, metformin increases insulin-mediated suppression of hepatic glucose production, mainly by reducing gluconeogenesis. In skeletal muscle it promotes insulin receptor phosphorylation

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-16-0450
Volume/Issue:
Volume 233: Issue 1
Free access
Erica Gentilin Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy
Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy

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Federico Tagliati Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy

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Massimo Terzolo Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy

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Matteo Zoli Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy

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Marcello Lapparelli Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy

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Mariella Minoia Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy

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Maria Rosaria Ambrosio Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy

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Ettore C degli Uberti Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy
Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy

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Maria Chiara Zatelli Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy
Section of Endocrinology, Laboratorio in Rete del Tecnopolo Tecnologie delle Terapie Avanzate (LTTA), Division of Internal Medicine I, Department of Neurosurgery, Neurosurgery Unit, Department of Medical Sciences, University of Ferrara, Via Savonarola 9, 44100 Ferrara, Italy

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effectively employed to treat CD but needs permanent glucocorticoid supplementation ( Porpiglia et al . 2004 ). Several medical therapies, including steroidogenic inhibitors, centrally acting agents, and glucocorticoid receptor inhibitors, are currently being

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0210
Volume/Issue:
Volume 218: Issue 3
Free access
Francisco J Arjona Department of Animal Physiology, Departamento de Biología, Instituto de Zoología, Institute for Water and Wetland Research, Faculty of Science, Radboud University Nijmegen, Heyendaalseweg 135, 6525 AJ Nijmegen, The Netherlands
Department of Animal Physiology, Departamento de Biología, Instituto de Zoología, Institute for Water and Wetland Research, Faculty of Science, Radboud University Nijmegen, Heyendaalseweg 135, 6525 AJ Nijmegen, The Netherlands

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Luis Vargas-Chacoff Department of Animal Physiology, Departamento de Biología, Instituto de Zoología, Institute for Water and Wetland Research, Faculty of Science, Radboud University Nijmegen, Heyendaalseweg 135, 6525 AJ Nijmegen, The Netherlands
Department of Animal Physiology, Departamento de Biología, Instituto de Zoología, Institute for Water and Wetland Research, Faculty of Science, Radboud University Nijmegen, Heyendaalseweg 135, 6525 AJ Nijmegen, The Netherlands

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María P Martín del Río Department of Animal Physiology, Departamento de Biología, Instituto de Zoología, Institute for Water and Wetland Research, Faculty of Science, Radboud University Nijmegen, Heyendaalseweg 135, 6525 AJ Nijmegen, The Netherlands

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Gert Flik Department of Animal Physiology, Departamento de Biología, Instituto de Zoología, Institute for Water and Wetland Research, Faculty of Science, Radboud University Nijmegen, Heyendaalseweg 135, 6525 AJ Nijmegen, The Netherlands

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Juan M Mancera Department of Animal Physiology, Departamento de Biología, Instituto de Zoología, Institute for Water and Wetland Research, Faculty of Science, Radboud University Nijmegen, Heyendaalseweg 135, 6525 AJ Nijmegen, The Netherlands

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Peter H M Klaren Department of Animal Physiology, Departamento de Biología, Instituto de Zoología, Institute for Water and Wetland Research, Faculty of Science, Radboud University Nijmegen, Heyendaalseweg 135, 6525 AJ Nijmegen, The Netherlands

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-to-T 3 conversion. However, we cannot exclude a feedback mechanism in which cortisol reduces the activity of hypothalamic corticotropin-releasing hormone (CRH), urotensin-I (UI), and thyrotropin-releasing hormone (TRH), factors with both

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-10-0416
Volume/Issue:
Volume 208: Issue 3
Free access
Brit H Boehmer Department of Pediatrics, Perinatal Research Center, University of Colorado School of Medicine, Aurora, Colorado, USA

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Sean W Limesand School of Animal and Comparative Biomedical Sciences, University of Arizona, Tucson, Arizona, USA

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Paul J Rozance Department of Pediatrics, Perinatal Research Center, University of Colorado School of Medicine, Aurora, Colorado, USA

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increase to inhibit fetal GSIS. Experimental interventions to disrupt catecholamine signaling are depicted. Pharmacological adrenergic receptor blockade enhan­ced GSIS in PI-IUGR fetuses at 70% and 90% gestation, providing even more evidence that

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-17-0076
Volume/Issue:
Volume 235: Issue 2
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Alan G Watts Department of Biological Sciences, USC Dornsife College of Letters, Arts, and Sciences, University of Southern California, Hedco Neuroscience Building, MC 2520, Los Angeles, California 90089‐2520, USA

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thyroid-stimulating hormone (TRF/TRH). Those for prolactin (dopamine, a release-inhibiting factor), ACTH (CRF/CRH), and growth hormone (GRF/GHRH and somatostatin) were identified at various times during the following decade. The structures of oxytocin and

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-15-0157
Volume/Issue:
Volume 226: Issue 2
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K Boelaert Division of Medical Sciences, IBR Building, 2nd floor, The Medical School, University of Birmingham, Birmingham B15 2TT, UK

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J A Franklyn Division of Medical Sciences, IBR Building, 2nd floor, The Medical School, University of Birmingham, Birmingham B15 2TT, UK

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-releasing hormone (TRH), which is secreted by the hypothalamus. Unbound or free T3 and T4 (fT3 and fT4 respectively) exert a negative feedback on the synthesis and release of TSH and TRH in order to maintain circulating thyroid hormone levels within the required

Article Type:
Other
DOI:
https://doi.org/10.1677/joe.1.06131
Volume/Issue:
Volume 187: Issue 1
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Cristina Capatina Department of Endocrinology, CI Parhon National Institute of Endocrinology, Department of Endocrinology, Carol Davila University of Medicine and Pharmacy, Bucharest, Romania
Department of Endocrinology, CI Parhon National Institute of Endocrinology, Department of Endocrinology, Carol Davila University of Medicine and Pharmacy, Bucharest, Romania

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John A H Wass Department of Endocrinology, CI Parhon National Institute of Endocrinology, Department of Endocrinology, Carol Davila University of Medicine and Pharmacy, Bucharest, Romania

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isolated disorder. This last condition is called familial isolated PA (FIPA); 15% of these cases harbor germline mutations of the AIP gene (encoding the aryl-hydrocarbon receptor interacting protein), but the responsible gene mutation is unknown in the

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-15-0109
Volume/Issue:
Volume 226: Issue 2
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Elaine de Oliveira Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute

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Egberto G Moura Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute

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Ana Paula Santos-Silva Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute

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Cíntia R Pinheiro Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute

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Natalia S Lima Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute

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José Firmino Nogueira-Neto Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute

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Andre L Nunes-Freitas Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute

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Yael Abreu-Villaça Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute

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Magna C F Passos Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute

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Patrícia C Lisboa Department of Physiological Sciences, Laboratory of Lipids, Department of Applied Nutrition, Roberto Alcantara Gomes Biology Institute

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-protein diet changes deiodinase activities and pituitary TSH response to TRH in adult rats . Experimental Biology and Medicine 233 57 – 63 . Liu RH Mizuta M Matsukura S 2004 The expression and functional role of nicotinic acetylcholine receptors

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-10-0104
Volume/Issue:
Volume 206: Issue 1
Free access
Michelle P Marassi Laboratório de Fisiologia Endócrina, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, CCS-Bloco G-Cidade Universitária, Ilha do Fundão, Rio de Janeiro 21949-900, Brazil

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Rodrigo S Fortunato Laboratório de Fisiologia Endócrina, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, CCS-Bloco G-Cidade Universitária, Ilha do Fundão, Rio de Janeiro 21949-900, Brazil

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Alba C Matos da Silva Laboratório de Fisiologia Endócrina, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, CCS-Bloco G-Cidade Universitária, Ilha do Fundão, Rio de Janeiro 21949-900, Brazil

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Valmara S Pereira Laboratório de Fisiologia Endócrina, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, CCS-Bloco G-Cidade Universitária, Ilha do Fundão, Rio de Janeiro 21949-900, Brazil

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Denise P Carvalho Laboratório de Fisiologia Endócrina, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, CCS-Bloco G-Cidade Universitária, Ilha do Fundão, Rio de Janeiro 21949-900, Brazil

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Doris Rosenthal Laboratório de Fisiologia Endócrina, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, CCS-Bloco G-Cidade Universitária, Ilha do Fundão, Rio de Janeiro 21949-900, Brazil

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Vânia M Corrêa da Costa Laboratório de Fisiologia Endócrina, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro, CCS-Bloco G-Cidade Universitária, Ilha do Fundão, Rio de Janeiro 21949-900, Brazil

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(in females) regulate TSH receptor levels in the immature rat thyroid, suggesting that postnatal elevation of sex steroids might have a physiological relevance to the TSH-induced mitogenic activity in thyrocytes. The postnatal surge in sex steroids

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06901
Volume/Issue:
Volume 192: Issue 1
Free access