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) axis activity at every level. At the hypothalamus, stress-induced release of corticotropin-releasing hormone (CRH) and activation of γ-aminobutyric acid (GABA)-ergic signalling mediate the suppression of gonadotropin-releasing hormone (GnRH) pulsatility
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Department of Endocrinology, Imperial College Healthcare NHS Trust, Hammersmith Hospital, London, UK
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.1016/j.yhbeh.2014.01.001 Clarkson J Herbison AE 2006 Postnatal development of kisspeptin neurons in mouse hypothalamus; sexual dimorphism and projections to gonadotropin-releasing hormone neurons . Endocrinology 147 5817 – 5825 . ( https
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corticosterone from the adrenal cortex ( Herman et al . 2016 ). The gonadotropin-releasing hormone (GnRH) neurons of the preoptic area (POA) respond to kisspeptin ( Irwig et al . 2004 , Han et al . 2005 , Messager et al . 2005 ) and other neuropeptides
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estrogens regulate the reproductive hormones LH and FSH by primarily altering the release of gonadotropin-releasing hormone (GnRH) ( Shupnik 1996 ). In the same way, estrogens could affect rQSOX expression in pituitary indirectly through the hypothalamic
State Key Laboratory of Reproductive Medicine, Department of Physiology, MOE-Shanghai Key Laboratory of Children's Environmental Health, Nanjing Medical University, Hanzhong Road 140, Nanjing 210029, China
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State Key Laboratory of Reproductive Medicine, Department of Physiology, MOE-Shanghai Key Laboratory of Children's Environmental Health, Nanjing Medical University, Hanzhong Road 140, Nanjing 210029, China
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gonadotropin-releasing hormone neuron firing activity . PNAS 102 15682 – 15687 . ( doi:10.1073/pnas.0504270102 ) Clarkson J d'Anglemont de Tassigny X Moreno AS Colledge WH Herbison AE 2008 Kisspeptin–GPR54 signaling is essential for
Department of Cell Biology, Physiology and Immunology, University of Cordoba, Cordoba, Spain
Hospital Universitario Reina Sofia, Cordoba, Spain
CIBER Fisiopatología de la Obesidad y Nutrición, Instituto de Salud Carlos III, Cordoba, Spain
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Department of Cell Biology, Physiology and Immunology, University of Cordoba, Cordoba, Spain
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FiDiPro Program, Department of Physiology, University of Turku, Turku, Finland
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the so-called hypothalamic–pituitary–gonadal (HPG) axis. The major hierarchical element of this neurohormonal axis is the decapeptide, gonadotropin-releasing hormone (GnRH), which is produced by a discrete population of neurons in the basal forebrain
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brain via the hypothalamic–pituitary–gonadal (HPG) axis. In the hypothalamus, neurons release gonadotropin-releasing hormone 1 (GnRH1), a signaling peptide that stimulates the release of gonadotropins from the pituitary, which in turn causes the gonads
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other secretagogs, e.g. thyroptropin-releasing hormone (TRH), GHRH, and gonadotropin-releasing hormone (GnRH), evoked no adenohypophyseal hormone secretion ( Meij et al. 1997 d ). Eight weeks after surgery, the CRH stimulation test was not able to
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secretion of gonadotrophic hormone … [and yet] (b) the anterior lobe of the pituitary has at most a very scanty nerve supply’. Despite his focus on reproduction and the unsolved mystery about the route by which hormone-releasing signals access the pituitary
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2003 Tamoxifen induces gonadotropin-releasing hormone self-priming through an estrogen-dependent progesterone receptor expression in the gonadotrope of the rat. Neuroendocrinology 77 424 –434. Bellido C , Aguilar R