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response to oxygen deficiency in this organ. However, the precise mechanism regulating GLUT1 expression during placentation has not been fully clarified. Heterodimeric hypoxia-inducible factor-1 (HIF-1) is a transcriptional activator that mediates
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Lawson Health Research Institute, Medicine, Paediatrics, St Joseph's Health Care, 268 Grosvenor Street, Room H404, London, Ontario, Canada N6A 4V2 Departments of
Lawson Health Research Institute, Medicine, Paediatrics, St Joseph's Health Care, 268 Grosvenor Street, Room H404, London, Ontario, Canada N6A 4V2 Departments of
Lawson Health Research Institute, Medicine, Paediatrics, St Joseph's Health Care, 268 Grosvenor Street, Room H404, London, Ontario, Canada N6A 4V2 Departments of
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depends on the expression of key transcription factors such as PDX-1 and PTF1α within the ductal cells ( Murtaugh 2007 ). PDX-1 is also required in the mature β-cell where it trans-activates the insulin and GLUT2 gene promoters. Other transcription factors
Medicine, Faculty of Life Sciences, Centre for Molecular
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translocating to the nucleus, and interacting with other transcription factors and transcriptional modulators ( Chen et al . 2001 , Stevens et al . 2003 , Garside et al . 2004 , O'Malley 2005 , McMaster & Ray 2007 ). In vivo target cells are subject to
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-releasing hormone (CRH), brain-derived neurotropic factor (BDNF), and cytokines ( Goujon et al . 1997 , Schulkin et al . 1998 , Morsink et al . 2006 , Schulte-Herbruggen et al . 2006 ). The GR transcriptional activity is dependent on the cell type, the
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. Although high concentrations of ROS are catatonic, they are now known to function as signal transducing molecules at lower concentrations, modulating indirectly the activity of many enzymes and transcription factors. Classical regulation of the activity of
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renal cortical tissue in HF rats was attenuated by dapagliflozin and metformin administration ( P < 0.05) ( Fig. 1I and J ). The effect of dapagliflozin on renal gluconeogenic enzyme expression might be mediated through transcription factors p
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Hebei Key Laboratory for Chronic Diseases, North China University of Science and Technology, Tangshan, Hebei, China
Tangshan Key Laboratory of Basic Research in Medicine Development, North China University of Science and Technology, Tangshan, Hebei, China
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Hebei Key Laboratory for Chronic Diseases, North China University of Science and Technology, Tangshan, Hebei, China
Tangshan Key Laboratory of Basic Research in Medicine Development, North China University of Science and Technology, Tangshan, Hebei, China
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potentially reverse hepatic fibrosis ( Mannaerts et al. 2015 ). As a coactivator of transcription factors, YAP promotes cell proliferation and inhibits apoptosis through the interaction between its transcriptional coactivator activity and TEAD
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-dependent transcription factors. By modulating the transcription of target genes in response to ligand, TRs play key physiological roles in the regulation of many aspects of development, growth and metabolism, including the regulation of mitochondrial activity ( Flamant
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Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan
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Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan
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Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan
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Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan
Division of Molecular and Metabolic Medicine, Department of Physiology and Cell Biology, Kobe University Graduate School of Medicine, Kobe, Japan
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glucose levels were similar to those of WT mice (113.16 ± 14.03 (WT) vs 125.33 ± 13.19 ( Chrebp −/− ) , respectively). mRNA expression of Srebf2, a key transcription factor controlling synthesis and uptake of cholesterol and their target genes, was
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number of insulin-regulated transcriptional factors are engaged in lipid metabolism and adipose tissue development. The examples are sterol regulatory element-binding protein 1c (SREBP1c), responsible for triglyceride biosynthesis, as well as PPARG