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phosphorylates signaling molecules such as GSK3β and promotes cell survival and proliferation by phosphorylating the FOXO family of transcription factors ( Brunet et al. 2001 ). SGK1 has an important role enhancing transepithelial Na + reabsorption ( Chen et
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( Chu & Ferro 2005 ) and thus possibly influence TRα promoter activity. Other binding sites for transcription factors on the TRα promoter have been described, such as early growth-response (Egr)-1 site (also known as Krox-24; Laudet et al
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Endocrine Signalling Group, Barts and the London School of Medicine and Dentistry, Department of Medicine, Cardiovascular and Inflammation Group, Laboratory for Integrated Neurosciences and Endocrinology, Veterinary Basic Sciences, Royal Veterinary College, University of London, Royal College Street, London NW1 0TU, UK
Endocrine Signalling Group, Barts and the London School of Medicine and Dentistry, Department of Medicine, Cardiovascular and Inflammation Group, Laboratory for Integrated Neurosciences and Endocrinology, Veterinary Basic Sciences, Royal Veterinary College, University of London, Royal College Street, London NW1 0TU, UK
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our preliminary in silico screen of potential transcription factor-binding sites ( Fig. 3 A), at least three distinct complexes were detected, which upon antibody interrogation appeared to contain Sp1/Sp3 proteins in homo- and heterodimer
Graduate School of the Chinese Academy of Sciences, 19 Yu-quan Road, Beijing 10009, China
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Graduate School of the Chinese Academy of Sciences, 19 Yu-quan Road, Beijing 10009, China
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Graduate School of the Chinese Academy of Sciences, 19 Yu-quan Road, Beijing 10009, China
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Graduate School of the Chinese Academy of Sciences, 19 Yu-quan Road, Beijing 10009, China
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Graduate School of the Chinese Academy of Sciences, 19 Yu-quan Road, Beijing 10009, China
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cholesterol from mitochondrial outer membrane to inner membrane, where cytochrome P450 side-chain cleavage enzyme (P450 scc) resides ( Christenson & Strauss 2000 , Stocco 2000 ). StAR expression is regulated by several transcription factors such as DAX-1
Biology, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China
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Biology, The Chinese University of Hong Kong, Shatin, N.T., Hong Kong, China
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.aist.go.jp/research/db/TFSEARCH.html ) revealed a number of putative transcription factor-binding sites on the 5′-flanking region of the seabream ghrelin gene (Fig. 5 ). Functional mapping of the seabream ghrelin promoter The promoter activity of the 5
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impairing cellular functioning ( Elsner et al . 2011 ). However, the mechanisms underlying β-cell dysfunction and the resulting apoptosis via those factors have not been fully characterized ( Donath et al . 1999 , Kim et al . 2005 , Lablanche et al
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Tyr 1138 engages the STAT3 transcription factor. LepRb→STAT3 signaling represents the primary mechanism by which leptin regulates energy balance, although the target genes of STAT3 in LepRb neurons remain undiscovered. Leptin also recruits the IRS2→PI
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member of the nuclear hormone receptor superfamily of transcription factors, is expressed in the hypothalamic–pituitary–adrenal/gonadal axis and has been demonstrated to have roles in the regulation of steroidogenesis ( Zazopoulos et al. 1997 , Lalli
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AP-HP Hôpital Pitié-Salpêtrière-Charles Foix, Nutrition Department, Paris, France
AP-HP Hôpital Pitié-Salpêtrière-Charles Foix, Diabetology-Metabolism Department, Paris, France
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AP-HP Hôpital Pitié-Salpêtrière-Charles Foix, Nutrition Department, Paris, France
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in vivo and in vitro have shown that HNF-4α plays pleiotropic roles in liver functions and is a central transcription factor at the crossroads between epithelial morphogenesis and functions ( Battle et al. 2006 , Ribeiro et al. 2007 , Hwang
UMR‐1132 Inserm, Université Paris Diderot, Hôpital Lariboisière, 2 Rue Ambroise Paré, 75475 Paris Cedex 10, France
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UMR‐1132 Inserm, Université Paris Diderot, Hôpital Lariboisière, 2 Rue Ambroise Paré, 75475 Paris Cedex 10, France
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UMR‐1132 Inserm, Université Paris Diderot, Hôpital Lariboisière, 2 Rue Ambroise Paré, 75475 Paris Cedex 10, France
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UMR‐1132 Inserm, Université Paris Diderot, Hôpital Lariboisière, 2 Rue Ambroise Paré, 75475 Paris Cedex 10, France
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to the inhibition of glycogen synthase kinase 3β and decreased phosphorylation, stabilisation and subsequent translocation of β-catenin into the nucleus. This results in the binding of β-catenin to T-cell-specific transcription factor