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tissue ischemia ( Tilton et al. 1997 , Duh & Aiello 1999 , Cruz et al. 2002 ). Moreover, vascular endothelial growth factor (VEGF) is known to be an endothelial mitogen and a potent vasopermeability factor ( Ferrara 1999 ). Recent evidence
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endothelial growth factor (VEGF) elicits angio-genesis by inducing endothelial proliferation, migration and tube formation, and plays a critical role in the regulation of vascular permeability and angiogenesis. This process involves the activation of multiple
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Department of Paediatrics, Department of Women's and Children's Health, Division of Endocrinology, Department of Tissue Regeneration, Department of Endocrinology and Metabolism, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands
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endochondral ossification is when blood vessels enter from the primary spongiosum and osteoblasts invade from the bone marrow to lay down trabecular bone ( Kember 1993 , Hunziker 1994 ). Vascular endothelial growth factor (VEGF) is a potent mediator of
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Introduction The endocrine gland-derived vascular endothelial growth factor (EG-VEGF) belongs to the AVIT protein family. The first described member of this family was originally isolated from snake venom and frog skin secretions. The amino
INSERM, IFR105 Université Paris 7, INSERM, U553, Hémostase, Endothélium et Angiogénèse, Hôpital Saint Louis, 1 Avenue Claude Vellefaux, 75475 Paris Cedex 10, France
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INSERM, IFR105 Université Paris 7, INSERM, U553, Hémostase, Endothélium et Angiogénèse, Hôpital Saint Louis, 1 Avenue Claude Vellefaux, 75475 Paris Cedex 10, France
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INSERM, IFR105 Université Paris 7, INSERM, U553, Hémostase, Endothélium et Angiogénèse, Hôpital Saint Louis, 1 Avenue Claude Vellefaux, 75475 Paris Cedex 10, France
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INSERM, IFR105 Université Paris 7, INSERM, U553, Hémostase, Endothélium et Angiogénèse, Hôpital Saint Louis, 1 Avenue Claude Vellefaux, 75475 Paris Cedex 10, France
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and progesterone are the stimuli of vascular endothelial growth factor (VEGF) expression in breast cancer cells and tumors ( Nakamura et al . 1996 , Hyder et al . 1998 , Ruohola et al . 1999 , Buteau-Lozano et al . 2002 , Stoner et al
Institut National de la Santé et de la Recherche Médicale, Commissariat à l'Energie Atomique, Université Joseph Fourier, Unité 878, Grenoble, France
Institut National de la Santé et de la Recherche Médicale, Commissariat à l'Energie Atomique, Université Joseph Fourier, Unité 878, Grenoble, France
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Institut National de la Santé et de la Recherche Médicale, Commissariat à l'Energie Atomique, Université Joseph Fourier, Unité 878, Grenoble, France
Institut National de la Santé et de la Recherche Médicale, Commissariat à l'Energie Atomique, Université Joseph Fourier, Unité 878, Grenoble, France
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Institut National de la Santé et de la Recherche Médicale, Commissariat à l'Energie Atomique, Université Joseph Fourier, Unité 878, Grenoble, France
Institut National de la Santé et de la Recherche Médicale, Commissariat à l'Energie Atomique, Université Joseph Fourier, Unité 878, Grenoble, France
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Institut National de la Santé et de la Recherche Médicale, Commissariat à l'Energie Atomique, Université Joseph Fourier, Unité 878, Grenoble, France
Institut National de la Santé et de la Recherche Médicale, Commissariat à l'Energie Atomique, Université Joseph Fourier, Unité 878, Grenoble, France
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Institut National de la Santé et de la Recherche Médicale, Commissariat à l'Energie Atomique, Université Joseph Fourier, Unité 878, Grenoble, France
Institut National de la Santé et de la Recherche Médicale, Commissariat à l'Energie Atomique, Université Joseph Fourier, Unité 878, Grenoble, France
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expression of angiogenic factors, such as vascular endothelial growth factor-A (VEGF-A), is high in these organs despite the absence of active angiogenesis, suggesting that these factors play an important role in the homeostasis and the hormonal control of
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retinopathy is predominantly caused by vascular endothelial growth factor (VEGF)-mediated stress fiber formation and vascular endothelial (VE)-cadherin disruption ( Spindler et al. 2010 , Lim et al. 2014 ). Recently, we demonstrated that reactive oxygen
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vasculature is a cause or consequence of follicle dominance remains to be determined. The principal angiogenic factor with an established role in follicular angiogenesis is vascular endothelial growth factor (VEGF). VEGF mRNA and protein have been
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follicular capillary bed in the theca interna. Impaired normal vascular formation may cause insufficiency of the CL in the luteal phase or in the early stage of pregnancy. The vascular endothelial growth factor (VEGF) plays a critical role in the regulation
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factor, vascular endothelial growth factor (VEGF) from glandular epithelial cells in castrated animals ( Joseph et al . 1997 , Häggström et al . 1999 ), and that neutralization of VEGF bioactivity with a soluble VEGF receptor chimerical protein, mflt(1