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Dieuwertje C E Spaanderman Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, the Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands

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Mark Nixon BHF Centre for Cardiovascular Science, The Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, United Kingdom

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Jacobus C Buurstede Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, the Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands

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Hetty H C M Sips Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, the Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands

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Maaike Schilperoort Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, the Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands

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Eline N Kuipers Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, the Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands

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Emma A Backer Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, the Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands

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Sander Kooijman Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, the Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands

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Patrick C N Rensen Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, the Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands

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Natalie Z M Homer BHF Centre for Cardiovascular Science, The Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, United Kingdom

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Brian R Walker BHF Centre for Cardiovascular Science, The Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, United Kingdom
Institute for Genetic Medicine, Newcastle University, Newcastle upon Tyne, United Kingdom

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Onno C Meijer Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, the Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands

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Jan Kroon Division of Endocrinology, Department of Medicine, Leiden University Medical Center, Leiden, the Netherlands
Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, the Netherlands

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the AR ( Arora et al . 2013 ), and these two receptors may even form heterodimers on glucocorticoid response elements ( Chen et al . 1997 ). However, beyond cancer the crosstalk between GR and AR has remained elusive. In this study, we explored if

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0503
Volume/Issue:
Volume 240: Issue 1
Free access
Greisa Vila
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Michaela Riedl
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Michael Resl
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Aart Jan van der Lely Division of Endocrinology and Metabolism, Division of Endocrinology, Department of Internal Medicine III, Medical University of Vienna, Waehringer Guertel 18-20, A-1090 Vienna, Austria

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Leo J Hofland Division of Endocrinology and Metabolism, Division of Endocrinology, Department of Internal Medicine III, Medical University of Vienna, Waehringer Guertel 18-20, A-1090 Vienna, Austria

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Martin Clodi
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Anton Luger
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neuroendocrine axis has been established: the gut–brain axis ( Murphy et al . 2006 ). Gut–brain signaling consists of bidirectional cross-talks between the hypothalamic nuclei and neuroendocrine cells of the gut, partially mediated by afferent and efferent

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0227
Volume/Issue:
Volume 203: Issue 1
Free access
Andréa M Caricilli Department of Internal Medicine, State University of Campinas, 13081-970 Campinas, SP, Brazil

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Paula H Nascimento Department of Internal Medicine, State University of Campinas, 13081-970 Campinas, SP, Brazil

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José R Pauli Department of Internal Medicine, State University of Campinas, 13081-970 Campinas, SP, Brazil

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Daniela M L Tsukumo Department of Internal Medicine, State University of Campinas, 13081-970 Campinas, SP, Brazil

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Lício A Velloso Department of Internal Medicine, State University of Campinas, 13081-970 Campinas, SP, Brazil

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José B Carvalheira Department of Internal Medicine, State University of Campinas, 13081-970 Campinas, SP, Brazil

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Mário J A Saad Department of Internal Medicine, State University of Campinas, 13081-970 Campinas, SP, Brazil

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modulator in the crosstalk between inflammation and metabolic pathway in DIO. However, in mice fed on standard chow, the inhibition of TLR2 did not cause an improvement in insulin sensitivity. This is probably due to the fact that TLR2 has a role in insulin

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0354
Volume/Issue:
Volume 199: Issue 3
Free access
Majdi Masarwi Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel
Felsenstein Medical Research Center, Petach Tikva, Israel

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Raanan Shamir Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel
Felsenstein Medical Research Center, Petach Tikva, Israel
Institute of Gastroenterology, Nutrition and Liver Diseases, Schneider Children’s Medical Center of Israel, Petach Tikva, Israel

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Moshe Phillip Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel
Felsenstein Medical Research Center, Petach Tikva, Israel
The Jesse Z and Sara Lea Shafer Institute for Endocrinology and Diabetes, National Center for Childhood Diabetes, Schneider Children’s Medical Center of Israel, Petach Tikva, Israel

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Galia Gat-Yablonski Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel
Felsenstein Medical Research Center, Petach Tikva, Israel
The Jesse Z and Sara Lea Shafer Institute for Endocrinology and Diabetes, National Center for Childhood Diabetes, Schneider Children’s Medical Center of Israel, Petach Tikva, Israel

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. 1997 ), and in GP chondrocytes ( Oz et al . 2001 , Eshet et al . 2004 ). The aim of this study was to investigate the crosstalk between leptin, which is increased by re-feeding after food restriction ( Pando et al . 2014 ), and sex hormones, which

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0028
Volume/Issue:
Volume 237: Issue 3
Free access
L Francisco Lorenzo-Martín Centro de Investigación del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Instituto de Biología Molecular y Celular del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Centro de Investigación Biomédica en Red de Cáncer (CIBERONC), CSIC-University of Salamanca, Salamanca, Spain

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Mauricio Menacho-Márquez Centro de Investigación del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Instituto de Biología Molecular y Celular del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Centro de Investigación Biomédica en Red de Cáncer (CIBERONC), CSIC-University of Salamanca, Salamanca, Spain

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Salvatore Fabbiano Centro de Investigación del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Instituto de Biología Molecular y Celular del Cáncer, CSIC-University of Salamanca, Salamanca, Spain

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Omar Al-Massadi Departamento de Fisioloxía, University of Santiago de Compostela, Santiago de Compostela, Spain
Centro de Investigación en Medicina Molecular e Enfermidades Crónicas, University of Santiago de Compostela, Santiago de Compostela, Spain
Centro de Investigación Biomédica en Red de Cáncer sobre la Fisiopatología de la Obesidad y Nutrición (CIBEROBN), University of Santiago de Compostela, Santiago de Compostela, Spain

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Antonio Abad Centro de Investigación del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Instituto de Biología Molecular y Celular del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Centro de Investigación Biomédica en Red de Cáncer (CIBERONC), CSIC-University of Salamanca, Salamanca, Spain

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Sonia Rodríguez-Fdez Centro de Investigación del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Instituto de Biología Molecular y Celular del Cáncer, CSIC-University of Salamanca, Salamanca, Spain

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María A Sevilla Centro de Investigación del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Instituto de Biología Molecular y Celular del Cáncer, CSIC-University of Salamanca, Salamanca, Spain

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María J Montero Centro de Investigación del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Instituto de Biología Molecular y Celular del Cáncer, CSIC-University of Salamanca, Salamanca, Spain

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Carlos Diéguez Departamento de Fisioloxía, University of Santiago de Compostela, Santiago de Compostela, Spain
Centro de Investigación en Medicina Molecular e Enfermidades Crónicas, University of Santiago de Compostela, Santiago de Compostela, Spain
Centro de Investigación Biomédica en Red de Cáncer sobre la Fisiopatología de la Obesidad y Nutrición (CIBEROBN), University of Santiago de Compostela, Santiago de Compostela, Spain

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Rubén Nogueiras Departamento de Fisioloxía, University of Santiago de Compostela, Santiago de Compostela, Spain
Centro de Investigación en Medicina Molecular e Enfermidades Crónicas, University of Santiago de Compostela, Santiago de Compostela, Spain
Centro de Investigación Biomédica en Red de Cáncer sobre la Fisiopatología de la Obesidad y Nutrición (CIBEROBN), University of Santiago de Compostela, Santiago de Compostela, Spain

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Xosé R Bustelo Centro de Investigación del Cáncer, CSIC-University of Salamanca, Salamanca, Spain
Instituto de Biología Molecular y Celular del Cáncer, CSIC-University of Salamanca, Salamanca, Spain

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Introduction Physiological and metabolic balance in the organism requires extensive crosstalk between the central nervous system and peripheral organs. Efferent signals to the periphery are mediated by the sympathetic (SNS) and parasympathetic

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0623
Volume/Issue:
Volume 240: Issue 3
Free access
Md Nurul Islam Division of Neurology, Respirology, Endocrinology and Metabolism, Department of Internal Medicine, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan

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Yuichiro Mita Division of Neurology, Respirology, Endocrinology and Metabolism, Department of Internal Medicine, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan
Systems Life Sciences Laboratory, Department of Medical Life Systems, Faculty of Life and Medical Sciences, Doshisha University, Kyoto, Japan

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Keisuke Maruyama Division of Neurology, Respirology, Endocrinology and Metabolism, Department of Internal Medicine, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan

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Ryota Tanida Division of Neurology, Respirology, Endocrinology and Metabolism, Department of Internal Medicine, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan
Department of Sports and Fitness, Faculty of Wellness, Shigakkan University, Aichi, Japan

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Weidong Zhang Division of Neurology, Respirology, Endocrinology and Metabolism, Department of Internal Medicine, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan

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Hideyuki Sakoda Division of Neurology, Respirology, Endocrinology and Metabolism, Department of Internal Medicine, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan

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Masamitsu Nakazato Division of Neurology, Respirology, Endocrinology and Metabolism, Department of Internal Medicine, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan
CREST (Japan) Agency for Medical Research and Development (A-MED) 1-7-1 Otemachi, Tokyo, Japan

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that peripheral administration of LEAP2 suppressed ghrelin-induced GH secretion and food intake in mice ( Ge et al. 2018 ). Therefore, both central and peripheral LEAP2 antagonizes ghrelin functions. Crosstalk between organs is important for

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-19-0102
Volume/Issue:
Volume 244: Issue 1
Open access
Henrik Oster Institute of Neurobiology, University of Lübeck, Lübeck, Germany

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high-fat diet disrupts diurnal food intake profiles and rhythms of gene expression in several tissues and even slows down the SCN pacemaker ( Kohsaka et al. 2007 ). This crosstalk has important implications for real-life situations of chronodisruption

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-20-0124
Volume/Issue:
Volume 247: Issue 1
Free access
Stefanie Ruhs Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Halle, Germany

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Alexander Nolze Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Halle, Germany

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Ralf Hübschmann Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Halle, Germany

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Claudia Grossmann Julius Bernstein Institute of Physiology, Martin Luther University Halle-Wittenberg, Halle, Germany

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. (2) Additionally, aldosterone and MR can initiate a crosstalk with other cytosolic signaling pathways, like for example NFAT and CREB signaling, which ultimately may affect genomic signaling. (3) Genomic MR signaling may be influenced by epigenetic

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-16-0659
Volume/Issue:
Volume 234: Issue 1
Free access
Mari van de Vyver Division of Clinical Pharmacology, Department of Medicine, Faculty of Medicine and Health Sciences, Stellenbosch University, Cape Town, South Africa

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, Kurita et al. 2021 ). Under pathological conditions, there is bilateral crosstalk between immune regulation and aberrant metabolism resulting in persistent inflammation in the absence of infection ( Furman et al. 2019 ). This phenomenon is referred to

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-22-0271
Volume/Issue:
Volume 257: Issue 1
Free access
Timothy J Dreyer Department of Comparative Biomedical Sciences, The Royal Veterinary College, London, UK

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Jacob AC Keen Department of Comparative Biomedical Sciences, The Royal Veterinary College, London, UK

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Leah M Wells Department of Comparative Biomedical Sciences, The Royal Veterinary College, London, UK

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Scott J Roberts Department of Comparative Biomedical Sciences, The Royal Veterinary College, London, UK

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mineralisation, suggesting crosstalk between vascular and bone tissue ( Mace et al. 2022 ). De Maré et al. showed that SOST −/− mice with adenine diet-induced CKD had significantly higher calcium content in their aorta compared to wild type ( De Maré et

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-22-0209
Volume/Issue:
Volume 257: Issue 2
Free access