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Unité de Recherches, Laboratoire de Neurochimie/LBPO, INSERM UMRS‐592, UPMC University Paris 06, INSERM U952, CNRS UMR 7224, Faculté des Sciences Biologiques/UMMTO, BP 17, Tizi-Ouzou, Algeria
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Unité de Recherches, Laboratoire de Neurochimie/LBPO, INSERM UMRS‐592, UPMC University Paris 06, INSERM U952, CNRS UMR 7224, Faculté des Sciences Biologiques/UMMTO, BP 17, Tizi-Ouzou, Algeria
Unité de Recherches, Laboratoire de Neurochimie/LBPO, INSERM UMRS‐592, UPMC University Paris 06, INSERM U952, CNRS UMR 7224, Faculté des Sciences Biologiques/UMMTO, BP 17, Tizi-Ouzou, Algeria
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Unité de Recherches, Laboratoire de Neurochimie/LBPO, INSERM UMRS‐592, UPMC University Paris 06, INSERM U952, CNRS UMR 7224, Faculté des Sciences Biologiques/UMMTO, BP 17, Tizi-Ouzou, Algeria
Unité de Recherches, Laboratoire de Neurochimie/LBPO, INSERM UMRS‐592, UPMC University Paris 06, INSERM U952, CNRS UMR 7224, Faculté des Sciences Biologiques/UMMTO, BP 17, Tizi-Ouzou, Algeria
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the antidiuretic hormone vasopressin (arginine vasopressin (AVP)) into the blood stream ( Antunes-Rodrigues et al . 2004 ). In Dp71-null mice, we described compensatory ectopic expression of DP140 in the magnocellular neurons of the hypothalamus. To
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Department of Physiology and Membrane Biology, School of Medicine, University of California, Davis, California, USA
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were maintained in group housing on a 12 h light:12 h darkness cycle with free access to water and standard rodent chow. Ucn3 -null mice were described previously ( Li et al. 2007 ). For all experiments, mice heterozygous for the Ucn3 -null allele
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times through regulated expression of PAPP-A and its associated proteolytic activity ( Pintar et al. 1998 ). Recently, we generated PAPP-A-null mice which, similar to IGF-II-null mice, were born as proportional dwarfs ( Conover et al. 2004 ). The
Institute for Molecular and Cell Biology, Rua do Campo Alegre, Porto, Portugal
ICBAS, University of Porto, Largo Professor Abel Salazar, Porto, Portugal
Molecular Endocrinology Unit, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC & UAM, Madrid, Spain
Department of Production & Systems Engineering, University of Minho, Campus Gualtar, Braga, Portugal
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Institute for Molecular and Cell Biology, Rua do Campo Alegre, Porto, Portugal
ICBAS, University of Porto, Largo Professor Abel Salazar, Porto, Portugal
Molecular Endocrinology Unit, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC & UAM, Madrid, Spain
Department of Production & Systems Engineering, University of Minho, Campus Gualtar, Braga, Portugal
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Institute for Molecular and Cell Biology, Rua do Campo Alegre, Porto, Portugal
ICBAS, University of Porto, Largo Professor Abel Salazar, Porto, Portugal
Molecular Endocrinology Unit, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC & UAM, Madrid, Spain
Department of Production & Systems Engineering, University of Minho, Campus Gualtar, Braga, Portugal
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Institute for Molecular and Cell Biology, Rua do Campo Alegre, Porto, Portugal
ICBAS, University of Porto, Largo Professor Abel Salazar, Porto, Portugal
Molecular Endocrinology Unit, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC & UAM, Madrid, Spain
Department of Production & Systems Engineering, University of Minho, Campus Gualtar, Braga, Portugal
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Institute for Molecular and Cell Biology, Rua do Campo Alegre, Porto, Portugal
ICBAS, University of Porto, Largo Professor Abel Salazar, Porto, Portugal
Molecular Endocrinology Unit, Instituto de Investigaciones Biomédicas Alberto Sols, CSIC & UAM, Madrid, Spain
Department of Production & Systems Engineering, University of Minho, Campus Gualtar, Braga, Portugal
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thyroid hormone homeostasis, we challenged TTR-null mice to cold exposure and removal of the thyroid gland, conditions of moderate and extremely increased hormone demand respectively. Materials and Methods Animals
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supported by the temperature-sensitive phenotype of Pacap null pups, which display reduced survival at a lower housing temperature ( Gray et al . 2002 ); however, the underlying mechanisms by which Pacap null mice are cold intolerant remain unknown. A
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Department of Research, The University of Connecticut School of Medicine, Saint Francis Hospital and Medical Center, 114 Woodland Street, Hartford, Connecticut 06105-1299, USA
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region of the osteocalcin gene. Cebpb null mutant mice are viable, possibly because of partial rescue from other members of the C/EBP family, or because C/EBPβ is dispensable during development. Cebpb null mice are affected by a lymphoproliferative
Laboratory of Metabolic Disease Research and Drug Development, Division of Biological Sciences, Ministry of Health Key Laboratory of Congenital Malformation, Shengjing Hospital, China Medical University, Shenyang, People's Republic of China
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Laboratory of Metabolic Disease Research and Drug Development, Division of Biological Sciences, Ministry of Health Key Laboratory of Congenital Malformation, Shengjing Hospital, China Medical University, Shenyang, People's Republic of China
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Laboratory of Metabolic Disease Research and Drug Development, Division of Biological Sciences, Ministry of Health Key Laboratory of Congenital Malformation, Shengjing Hospital, China Medical University, Shenyang, People's Republic of China
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role for the VDR in the regulation of adipogenesis and energy metabolism. 1,25(OH) 2 D 3 suppresses 3T3-L1 preadipocyte differentiation into mature adipocytes ( Blumberg et al . 2006 , Kong & Li 2006 ). Global VDR-null mice exhibit a lean phenotype
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initial failure of oocyte release did not affect luteinization and production of progesterone in the early stages of pregnancy, the normal mid-gestation rise in progesterone levels was not as pronounced in embryo-transferred RIP140-null mice. This is
Cardiovascular Endocrinology, Department of Physiology, MIMR-PHI Institute, 27–31 Wright St, Clayton 3168, Australia
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Cardiovascular Endocrinology, Department of Physiology, MIMR-PHI Institute, 27–31 Wright St, Clayton 3168, Australia
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MR ( McGraw et al . 2013 ). Aldosterone significantly increases the development of early atherosclerotic changes in the aorta as well as increasing the degree of inflammation of plaques in Apoe -null mice. The authors identified placental growth
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Bioscience Metabolism, Research and Early Development, Cardiovascular, Renal and Metabolism, Biopharmaceuticals R&D, AstraZeneca, Gothenburg, Sweden
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al. 2004 , Lang 2007 ), such as caveolae. Cav1 null mice exhibit decreased circulating levels of adiponectin as well as adipose tissue metabolic and mitochondrial dysfunction. Interestingly, the HMW form of adiponectin was specifically reduced in