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Dipali Goyal School of Animal and Comparative Biomedical Sciences, College of Agriculture and Life Sciences, University of Arizona, Tucson, Arizona, USA

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Sean W Limesand School of Animal and Comparative Biomedical Sciences, College of Agriculture and Life Sciences, University of Arizona, Tucson, Arizona, USA

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Ravi Goyal School of Animal and Comparative Biomedical Sciences, College of Agriculture and Life Sciences, University of Arizona, Tucson, Arizona, USA

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, Jang et al. 2015 , Agarwal et al. 2018 , Ducsay et al. 2018 , Lecoutre et al. 2018 ). A factor that has received increasing attention, in this regard, is the idea of ‘developmental programming’ during fetal life, as a consequence of maternal

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-19-0009
Volume/Issue:
Volume 242: Issue 1
Free access
Umberto Simeoni Division of Pediatrics & DOHaD Laboratory, CHUV University Hospital and University of Lausanne, Lausanne, Switzerland

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Clive Osmond MRC Lifecourse Epidemiology Unit, University of Southampton, Southampton, UK

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Ricardo Garay Craven, Villemoisson-sur-Orge, France

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Christophe Buffat Laboratoire de Biochimie et de Biologie Moléculaire, Hôpital de la Conception, Marseille, France

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Farid Boubred Division of Neonatology, Assistance Publique, Hôpitaux de Marseille, Marseille, France

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Christophe Chagnaud Division of Radiology and Medical Imaging, Assistance Publique, Hôpitaux de Marseille, Marseille, France

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Elisabeth Jouve UPCET, Clinical Pharmacology, Assistance Publique, Hôpitaux de Marseille, Marseille, France

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Christine Audebert UPCET, Clinical Pharmacology, Assistance Publique, Hôpitaux de Marseille, Marseille, France

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Jean-Michel Antoine Danone Institute International, Paris, France

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Kent Thornburg Bob and Charlee Moore Institute for Nutrition and Wellness, Oregon Health & Science University, Portland, Oregon, USA

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range have been shown to be sufficient to influence the cardio-metabolic risk ( Barker et al. 1989 ). However little is known on developmental programming that occurs under the influence of variations in what is considered the normal environment, and

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0538
Volume/Issue:
Volume 244: Issue 2
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Alice S Green
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Paul J Rozance Department of Animal Sciences, Department of Pediatrics, University of Arizona, 1650 East Limberlost Drive, Tucson, Arizona 85719, USA

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Sean W Limesand
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role in building our understanding of developmental programing of β-cells. Although no model perfectly replicates human circumstances, all of the rodent and sheep models discussed here show a clear association between fetal growth restriction and β

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0399
Volume/Issue:
Volume 205: Issue 3
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Kazunari Nohara Division of Endocrinology, Division of Endocrinology, Department of Genetics and Development, INSERM U1048, Metabolism and Molecular Medicine and Comprehensive Center on Obesity, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA

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Suhuan Liu Division of Endocrinology, Division of Endocrinology, Department of Genetics and Development, INSERM U1048, Metabolism and Molecular Medicine and Comprehensive Center on Obesity, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA

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Matthew S Meyers Division of Endocrinology, Division of Endocrinology, Department of Genetics and Development, INSERM U1048, Metabolism and Molecular Medicine and Comprehensive Center on Obesity, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA

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Aurélie Waget Division of Endocrinology, Division of Endocrinology, Department of Genetics and Development, INSERM U1048, Metabolism and Molecular Medicine and Comprehensive Center on Obesity, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA

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Mathieu Ferron Division of Endocrinology, Division of Endocrinology, Department of Genetics and Development, INSERM U1048, Metabolism and Molecular Medicine and Comprehensive Center on Obesity, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA

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Gérard Karsenty Division of Endocrinology, Division of Endocrinology, Department of Genetics and Development, INSERM U1048, Metabolism and Molecular Medicine and Comprehensive Center on Obesity, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA

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Rémy Burcelin Division of Endocrinology, Division of Endocrinology, Department of Genetics and Development, INSERM U1048, Metabolism and Molecular Medicine and Comprehensive Center on Obesity, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA

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Franck Mauvais-Jarvis Division of Endocrinology, Division of Endocrinology, Department of Genetics and Development, INSERM U1048, Metabolism and Molecular Medicine and Comprehensive Center on Obesity, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA
Division of Endocrinology, Division of Endocrinology, Department of Genetics and Development, INSERM U1048, Metabolism and Molecular Medicine and Comprehensive Center on Obesity, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611, USA

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Introduction Polycystic ovary syndrome (PCOS) is the most common endocrine disorder in females of reproductive age and is believed to have a developmental origin in which maternal and fetal androgen excess during pregnancy programs both the

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0230
Volume/Issue:
Volume 219: Issue 3
Free access
Seokwon Jo Department of Integrative Biology & Physiology, University of Minnesota Medical School, Minneapolis, Minnesota, USA

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Emilyn U Alejandro Department of Integrative Biology & Physiology, University of Minnesota Medical School, Minneapolis, Minnesota, USA

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, Wang et al. 2015 ). The amount of pancreatic beta-cell mass is determined by the quantity and early developmental programming of pancreatic progenitor cells ( Stanger et al. 2007 ). This implies that the conditions experienced in the fetal

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-23-0069
Volume/Issue:
Volume 259: Issue 3
Open access
Fabio V Comim Imperial College London, Institute of Biodiversity, Institute of Reproductive and Developmental Biology, London W12 0NN, UK

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Kate Hardy Imperial College London, Institute of Biodiversity, Institute of Reproductive and Developmental Biology, London W12 0NN, UK

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Jane Robinson Imperial College London, Institute of Biodiversity, Institute of Reproductive and Developmental Biology, London W12 0NN, UK

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Stephen Franks Imperial College London, Institute of Biodiversity, Institute of Reproductive and Developmental Biology, London W12 0NN, UK

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3966 – 3973 . ( doi:10.1016/j.febslet.2009.10.036 ) Ortega HH Salvetti NR Padmanabhan V 2009 Developmental programming: prenatal androgen excess disrupts ovarian steroid receptor balance . Reproduction 137 865 – 877 . ( doi:10.1530/REP-08

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0150
Volume/Issue:
Volume 225: Issue 1
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Christopher M Mulligan Section of Neonatology, Department of Pediatrics, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA

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Jacob E Friedman Section of Neonatology, Department of Pediatrics, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA

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advanced fibrosis, respectively, compared with those of normal birth weight. Evidence suggests that the transgenerational passage of the maternal obese phenotype with NAFLD, or developmental programming of the offspring, involves the innate immune system

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-17-0303
Volume/Issue:
Volume 235: Issue 1
Free access
Simon Lecoutre Univ. Lille, EA4489, Équipe Malnutrition Maternelle et Programmation des Maladies Métaboliques, F59000 Lille, France

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Barbara Deracinois Univ. Lille, EA4489, Équipe Malnutrition Maternelle et Programmation des Maladies Métaboliques, F59000 Lille, France

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Christine Laborie Univ. Lille, EA4489, Équipe Malnutrition Maternelle et Programmation des Maladies Métaboliques, F59000 Lille, France

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Delphine Eberlé Univ. Lille, EA4489, Équipe Malnutrition Maternelle et Programmation des Maladies Métaboliques, F59000 Lille, France

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Céline Guinez Univ. Lille, EA4489, Équipe Malnutrition Maternelle et Programmation des Maladies Métaboliques, F59000 Lille, France

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Polina E Panchenko INRA, UMR1198 Biologie du Développement et Reproduction, F-78350 Jouy-en-Josas, France

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Jean Lesage Univ. Lille, EA4489, Équipe Malnutrition Maternelle et Programmation des Maladies Métaboliques, F59000 Lille, France

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Didier Vieau Univ. Lille, EA4489, Équipe Malnutrition Maternelle et Programmation des Maladies Métaboliques, F59000 Lille, France

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Claudine Junien INRA, UMR1198 Biologie du Développement et Reproduction, F-78350 Jouy-en-Josas, France
UVSQ, Université Versailles-Saint-Quentin-en-Yvelines, Versailles, France

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Anne Gabory INRA, UMR1198 Biologie du Développement et Reproduction, F-78350 Jouy-en-Josas, France

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Christophe Breton Univ. Lille, EA4489, Équipe Malnutrition Maternelle et Programmation des Maladies Métaboliques, F59000 Lille, France

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‘developmental programming’ or ‘conditioning’ ( Barker 2004 , Hanson & Gluckman 2014 ), alterations of nutrient supply in the fetus or neonate result in long-term programming of individual body weight (BW) setpoint. Epidemiological studies initially showed that

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0037
Volume/Issue:
Volume 230: Issue 1
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Márta Szaszák
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Hung-Dar Chen
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Hao-Chia Chen
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Albert Baukal
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László Hunyady Section on Hormonal Regulation, Department of Physiology, Endocrinology and Reproduction Research Branch, Program in Developmental Endocrinology and Genetics, Eunice Kennedy Shriver National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892-4510, USA

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Kevin J Catt
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Intramural Research Program, Eunice Kennedy Shriver National Institute of Child Health and Human Development. Acknowledgements We thank Tamas Balla and Balazs Mihalik for their valuable discussions. Microscopy imaging was performed at the Microscopy

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0190
Volume/Issue:
Volume 199: Issue 2
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Bo He Department of Pharmacology, Wuhan University School of Basic Medical Sciences, Wuhan, China
School of Pharmaceutical Sciences and Yunnan Key Laboratory of Pharmacology for Natural Products, Kunming Medical University, Kunming, China

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Yinxian Wen Department of Orthopedic Surgery, Zhongnan Hospital of Wuhan University, Wuhan, China
Hubei Provincial Key Laboratory of Developmentally Originated Disease, Wuhan, China

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Shuwei Hu Department of Pharmacology, Wuhan University School of Basic Medical Sciences, Wuhan, China

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Guihua Wang Department of Pharmacology, Wuhan University School of Basic Medical Sciences, Wuhan, China

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Wen Hu Department of Pharmacology, Wuhan University School of Basic Medical Sciences, Wuhan, China

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Jacques Magdalou UMR 7561 CNRS-Université de Lorraine, Faculté de Médicine, Vandoeuvre-lès-Nancy, Nancy, France

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Liaobin Chen Department of Orthopedic Surgery, Zhongnan Hospital of Wuhan University, Wuhan, China
Hubei Provincial Key Laboratory of Developmentally Originated Disease, Wuhan, China

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Hui Wang Department of Pharmacology, Wuhan University School of Basic Medical Sciences, Wuhan, China
Hubei Provincial Key Laboratory of Developmentally Originated Disease, Wuhan, China

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( Pei et al. 2017 ). Thus, intrauterine programming of the glucocorticoid-insulin-like growth factor 1 (GC-IGF1) axis induced by PCE may mediate liver developmental toxicology in offspring. However, whether PCE-induced liver developmental dysfunction

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-19-0066
Volume/Issue:
Volume 242: Issue 3
Free access