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Diletta Piccirilli Department of Biomedicine and Prevention, University of Rome Tor Vergata, Rome, Italy

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Enke Baldini Department of Surgical Sciences, ‘Sapienza’ University of Rome, Rome, Italy

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Micol Massimiani Department of Biomedicine and Prevention, University of Rome Tor Vergata, Rome, Italy

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Antonella Camaioni Department of Biomedicine and Prevention, University of Rome Tor Vergata, Rome, Italy

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Antonietta Salustri Department of Biomedicine and Prevention, University of Rome Tor Vergata, Rome, Italy

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Roberta Bernardini Department of Biology, NSTA, II University of Rome, Tor Vergata, Italy

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Marco Centanni Department of Medico-Surgical Sciences and Biotechnologies, ‘Sapienza’ University of Rome, Latina, Italy

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Salvatore Ulisse Department of Surgical Sciences, ‘Sapienza’ University of Rome, Rome, Italy

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Costanzo Moretti Department of Systems’ Medicine University of Rome Tor Vergata, UOC of Endocrinology and Diabetes, Section of Reproductive Endocrinology Fatebenefratelli Hospital, ‘Isola Tiberina’, Rome, Italy

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Luisa Campagnolo Department of Biomedicine and Prevention, University of Rome Tor Vergata, Rome, Italy

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blastocysts ( Fig. 8 ). These data suggest that TH regulates the bidirectional crosstalk between the receptive endometrium and the ready-to-implant embryo. Figure 6 TH supplementation stimulates mouse blastocyst hatching in vitro . (A) Schematic

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R C M Simmen Department of Physiology and Biophysics, Interdisciplinary Biomedical Sciences Program, Arkansas Children's Nutrition Center, Buck Institute for Age Research, University of Arkansas for Medical Sciences, 4301 W. Markham Street, Little Rock, Arkansas 72202, USA
Department of Physiology and Biophysics, Interdisciplinary Biomedical Sciences Program, Arkansas Children's Nutrition Center, Buck Institute for Age Research, University of Arkansas for Medical Sciences, 4301 W. Markham Street, Little Rock, Arkansas 72202, USA
Department of Physiology and Biophysics, Interdisciplinary Biomedical Sciences Program, Arkansas Children's Nutrition Center, Buck Institute for Age Research, University of Arkansas for Medical Sciences, 4301 W. Markham Street, Little Rock, Arkansas 72202, USA

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J M P Pabona Department of Physiology and Biophysics, Interdisciplinary Biomedical Sciences Program, Arkansas Children's Nutrition Center, Buck Institute for Age Research, University of Arkansas for Medical Sciences, 4301 W. Markham Street, Little Rock, Arkansas 72202, USA

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M C Velarde Department of Physiology and Biophysics, Interdisciplinary Biomedical Sciences Program, Arkansas Children's Nutrition Center, Buck Institute for Age Research, University of Arkansas for Medical Sciences, 4301 W. Markham Street, Little Rock, Arkansas 72202, USA

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C Simmons Department of Physiology and Biophysics, Interdisciplinary Biomedical Sciences Program, Arkansas Children's Nutrition Center, Buck Institute for Age Research, University of Arkansas for Medical Sciences, 4301 W. Markham Street, Little Rock, Arkansas 72202, USA

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O Rahal Department of Physiology and Biophysics, Interdisciplinary Biomedical Sciences Program, Arkansas Children's Nutrition Center, Buck Institute for Age Research, University of Arkansas for Medical Sciences, 4301 W. Markham Street, Little Rock, Arkansas 72202, USA
Department of Physiology and Biophysics, Interdisciplinary Biomedical Sciences Program, Arkansas Children's Nutrition Center, Buck Institute for Age Research, University of Arkansas for Medical Sciences, 4301 W. Markham Street, Little Rock, Arkansas 72202, USA

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F A Simmen Department of Physiology and Biophysics, Interdisciplinary Biomedical Sciences Program, Arkansas Children's Nutrition Center, Buck Institute for Age Research, University of Arkansas for Medical Sciences, 4301 W. Markham Street, Little Rock, Arkansas 72202, USA
Department of Physiology and Biophysics, Interdisciplinary Biomedical Sciences Program, Arkansas Children's Nutrition Center, Buck Institute for Age Research, University of Arkansas for Medical Sciences, 4301 W. Markham Street, Little Rock, Arkansas 72202, USA

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and/or progesterone signaling. Given the experimental data that KLFs may promote or attenuate endocrine-responsive cancers, the possibility that KLFs exert their effects through crosstalk with ESR1 and PGR signaling pathways was anticipated ( Zhang et

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Hai-Fan Yu College of Veterinary Medicine, Jilin University, Changchun, People’s Republic of China

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Zhan-Peng Yue College of Veterinary Medicine, Jilin University, Changchun, People’s Republic of China

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Kai Wang College of Veterinary Medicine, Jilin University, Changchun, People’s Republic of China

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Zhan-Qing Yang College of Veterinary Medicine, Jilin University, Changchun, People’s Republic of China

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Hong-Liang Zhang College of Veterinary Medicine, Jilin University, Changchun, People’s Republic of China

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Shuang Geng College of Veterinary Medicine, Jilin University, Changchun, People’s Republic of China

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Bin Guo College of Veterinary Medicine, Jilin University, Changchun, People’s Republic of China

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-transfected stromal cells, whereas silencing of Gja1 abolished the induction of Acvr1 on Hand2 expression, implying that Gja1 may play an important role in the crosstalk between Acvr1 and Hand2 . It has been well established that initiation of the

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Nan Yang Manchester Centre for Nuclear Hormone Research in Disease and Institute of Human Development, Institute of Cardiovascular Sciences, Faculty of Medical and Human Sciences, University of Manchester, AV Hill Building, Oxford Road, Manchester, M13 9PT, UK

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Giorgio Caratti Manchester Centre for Nuclear Hormone Research in Disease and Institute of Human Development, Institute of Cardiovascular Sciences, Faculty of Medical and Human Sciences, University of Manchester, AV Hill Building, Oxford Road, Manchester, M13 9PT, UK

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Louise M Ince Manchester Centre for Nuclear Hormone Research in Disease and Institute of Human Development, Institute of Cardiovascular Sciences, Faculty of Medical and Human Sciences, University of Manchester, AV Hill Building, Oxford Road, Manchester, M13 9PT, UK

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Toryn M Poolman Manchester Centre for Nuclear Hormone Research in Disease and Institute of Human Development, Institute of Cardiovascular Sciences, Faculty of Medical and Human Sciences, University of Manchester, AV Hill Building, Oxford Road, Manchester, M13 9PT, UK

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Peter J Trebble Manchester Centre for Nuclear Hormone Research in Disease and Institute of Human Development, Institute of Cardiovascular Sciences, Faculty of Medical and Human Sciences, University of Manchester, AV Hill Building, Oxford Road, Manchester, M13 9PT, UK

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Cathy M Holt Manchester Centre for Nuclear Hormone Research in Disease and Institute of Human Development, Institute of Cardiovascular Sciences, Faculty of Medical and Human Sciences, University of Manchester, AV Hill Building, Oxford Road, Manchester, M13 9PT, UK

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David W Ray Manchester Centre for Nuclear Hormone Research in Disease and Institute of Human Development, Institute of Cardiovascular Sciences, Faculty of Medical and Human Sciences, University of Manchester, AV Hill Building, Oxford Road, Manchester, M13 9PT, UK

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Laura C Matthews Manchester Centre for Nuclear Hormone Research in Disease and Institute of Human Development, Institute of Cardiovascular Sciences, Faculty of Medical and Human Sciences, University of Manchester, AV Hill Building, Oxford Road, Manchester, M13 9PT, UK

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as to permit cross-talk. We investigated several endogenous GR-transactivated genes. Gc-induced expression of MT1X and FKBP5 was attenuated by serum, while GILZ and PER1 remains unaffected. Interestingly, sequence analysis of the MT1X gene

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Luana Lopes Souza
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Aline Cordeiro
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Lorraine Soares Oliveira
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Gabriela Silva Monteiro de Paula
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Larissa Costa Faustino
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Tania Maria Ortiga-Carvalho
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Karen Jesus Oliveira Biophysics Institute Carlos Chagas Filho, Department of Physiology and Pharmacology, Federal University of Rio de Janeiro, Centro de Ciências da Saúde, Avenida Carlos Chagas Filho, 373, Bloco G, Cidade Universitária - Ilha do Fundão, Rio de Janeiro - RJ 21941-902, Brazil

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Carmen Cabanelas Pazos-Moura
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designate significant differences between groups ( P <0.05 or less). Discussion This study highlights in vivo cross-talking mechanisms between the n-3 PUFA from FO and the TH signaling in lipid metabolism. Previously, we had reported that rats fed an FO

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Esther Isorna Departamento de Fisiología (Fisiología Animal II), Facultad de Biología, Universidad Complutense de Madrid, Madrid, Spain

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Nuria de Pedro Departamento de Fisiología (Fisiología Animal II), Facultad de Biología, Universidad Complutense de Madrid, Madrid, Spain

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Ana I Valenciano Departamento de Fisiología (Fisiología Animal II), Facultad de Biología, Universidad Complutense de Madrid, Madrid, Spain

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Ángel L Alonso-Gómez Departamento de Fisiología (Fisiología Animal II), Facultad de Biología, Universidad Complutense de Madrid, Madrid, Spain

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María J Delgado Departamento de Fisiología (Fisiología Animal II), Facultad de Biología, Universidad Complutense de Madrid, Madrid, Spain

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may be related to the daily rhythms of clock genes; indeed, daily variations in the hypothalamic expression of orexin in goldfish have been related to clock genes oscillations ( Hoskins & Volkoff 2012 a ), and cross-talking between orexin signalling

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Lin Xia
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Zhongqiu Wang Center for Molecular Metabolism, Department of Radiology, Department of Biochemistry and Molecular Biology, Nanjing University of Science and Technology, B508, #364, 200 Xiaolingwei Street, Nanjing 210094, China

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Ying Zhang Center for Molecular Metabolism, Department of Radiology, Department of Biochemistry and Molecular Biology, Nanjing University of Science and Technology, B508, #364, 200 Xiaolingwei Street, Nanjing 210094, China

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Xiao Yang
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Yibei Zhan
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Rui Cheng
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Shiming Wang
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Jianfa Zhang
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, the elevation in the level of plasma 5′-AMP (pAMP) is a potential upstream regulator of hyperglycemia in type 2 diabetes ( Zhang et al . 2012 ). The question as to whether a crosstalk exists between insulin signaling and pAMP remains unknown. In this

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Julie Rodriguez Metabolism and Nutrition Research Group, Louvain Drug Research Institute, UCLouvain, Université catholique de Louvain, Brussels, Belgium

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Nathalie M Delzenne Metabolism and Nutrition Research Group, Louvain Drug Research Institute, UCLouvain, Université catholique de Louvain, Brussels, Belgium

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R Tremaroli V Kovatcheva-Datchary P Cani PD Backhed F 2015 Crosstalk between gut microbiota and dietary lipids aggravates WAT inflammation through TLR signaling . Cell Metabolism 22 658 – 668 . ( https://doi.org/10.1016/j.cmet.2015

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Arturo Hernandez Center for Molecular Medicine, Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine, USA
Graduate School for Biomedical Science and Engineering, University of Maine, Orono, Maine, USA
Department of Medicine, Tufts University School of Medicine, Boston, Massachusetts, USA

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M Elena Martinez Center for Molecular Medicine, Maine Medical Center Research Institute, Maine Medical Center, Scarborough, Maine, USA

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and cross-talk to ensure adequate cell function and hormone action in testicular tissue to support the maturation of germ cells into functional sperm ( Makela et al . 2019 ). The disruption of these processes may impair spermatogenesis and compromise

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Sujith Rajan Division of Pharmacology, CSIR-Central Drug Research Institute, Lucknow, India
Academy of Scientific and Innovative Research, CSIR-CDRI, Lucknow, India

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Ganesh Panzade Studio of Computational Biology and Bioinformatics, Biotechnology Division, CSIR-Institute of Himalayan Bioresource Technology, Palampur, India
Academy of Scientific and Innovative Research, CSIR-IHBT, Palampur, India

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Ankita Srivastava Division of Pharmacology, CSIR-Central Drug Research Institute, Lucknow, India
Academy of Scientific and Innovative Research, CSIR-CDRI, Lucknow, India

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Kripa Shankar Division of Pharmacology, CSIR-Central Drug Research Institute, Lucknow, India

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Rajesh Pandey CSIR Ayurgenomics Unit-TRISUTRA, CSIR-Institute of Genomics and Integrative Biology, New Delhi, India

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Durgesh Kumar Division of Pharmacology, CSIR-Central Drug Research Institute, Lucknow, India
Academy of Scientific and Innovative Research, CSIR-CDRI, Lucknow, India

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Sanchita Gupta Division of Pharmacology, CSIR-Central Drug Research Institute, Lucknow, India
Academy of Scientific and Innovative Research, CSIR-CDRI, Lucknow, India

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Abhishek Gupta Division of Pharmacology, CSIR-Central Drug Research Institute, Lucknow, India

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Salil Varshney Division of Pharmacology, CSIR-Central Drug Research Institute, Lucknow, India
Academy of Scientific and Innovative Research, CSIR-CDRI, Lucknow, India

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Muheeb Beg Division of Pharmacology, CSIR-Central Drug Research Institute, Lucknow, India

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Raj Kumar Mishra SIPS Superspeciality Hospital, Lucknow, India

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Ravi Shankar Studio of Computational Biology and Bioinformatics, Biotechnology Division, CSIR-Institute of Himalayan Bioresource Technology, Palampur, India
Academy of Scientific and Innovative Research, CSIR-IHBT, Palampur, India

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Anil Gaikwad Division of Pharmacology, CSIR-Central Drug Research Institute, Lucknow, India
Academy of Scientific and Innovative Research, CSIR-CDRI, Lucknow, India

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profile in IR adipocytes. Further, we found that the miR-876-3p is a critical determinant of adiponectin expression by virtue of target within adiponectin 3′UTR. These effects of miR-876-3p could be greatly consolidated and augmented by crosstalk between

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