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Ola Nilsson Developmental Endocrinology Branch and
Biometry and Mathematical Statistics Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA

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Robert D Mitchum Jr Developmental Endocrinology Branch and
Biometry and Mathematical Statistics Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA

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Lenneke Schrier Developmental Endocrinology Branch and
Biometry and Mathematical Statistics Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA

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Sandra P Ferns Developmental Endocrinology Branch and
Biometry and Mathematical Statistics Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA

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Kevin M Barnes Developmental Endocrinology Branch and
Biometry and Mathematical Statistics Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA

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James F Troendle Developmental Endocrinology Branch and
Biometry and Mathematical Statistics Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA

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Jeffrey Baron Developmental Endocrinology Branch and
Biometry and Mathematical Statistics Branch, National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA

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Introduction Overall body dimensions of mammals are primarily determined by growth of the skeleton, particularly longitudinal growth of the long bones and vertebrae. Longitudinal bone growth, in turn, results from chondrocyte

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06016
Volume/Issue:
Volume 186: Issue 1
Free access
James K Yeh Department of Medicine, Winthrop University Hospital, Mineola, New York 11501, USA
The Health Sciences Center, State University of New York, Stony Brook, New York 11794, USA

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Jodi F Evans Department of Medicine, Winthrop University Hospital, Mineola, New York 11501, USA
The Health Sciences Center, State University of New York, Stony Brook, New York 11794, USA

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Qing-Tian Niu Department of Medicine, Winthrop University Hospital, Mineola, New York 11501, USA
The Health Sciences Center, State University of New York, Stony Brook, New York 11794, USA

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John F Aloia Department of Medicine, Winthrop University Hospital, Mineola, New York 11501, USA
The Health Sciences Center, State University of New York, Stony Brook, New York 11794, USA

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Introduction Longitudinal growth is accomplished through the process of endochondral ossification, which occurs in the cartilaginous growth plate located at both ends of the vertebrae and long bones. It follows a sequential pattern

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06729
Volume/Issue:
Volume 191: Issue 3
Free access
Majdi Masarwi Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel
Felsenstein Medical Research Center, Petach Tikva, Israel

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Raanan Shamir Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel
Felsenstein Medical Research Center, Petach Tikva, Israel
Institute of Gastroenterology, Nutrition and Liver Diseases, Schneider Children’s Medical Center of Israel, Petach Tikva, Israel

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Moshe Phillip Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel
Felsenstein Medical Research Center, Petach Tikva, Israel
The Jesse Z and Sara Lea Shafer Institute for Endocrinology and Diabetes, National Center for Childhood Diabetes, Schneider Children’s Medical Center of Israel, Petach Tikva, Israel

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Galia Gat-Yablonski Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel
Felsenstein Medical Research Center, Petach Tikva, Israel
The Jesse Z and Sara Lea Shafer Institute for Endocrinology and Diabetes, National Center for Childhood Diabetes, Schneider Children’s Medical Center of Israel, Petach Tikva, Israel

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Introduction Temporary growth attenuation in children may be due to endocrinological, nutritional, medical or emotional disorders. Catch-up growth (CUG) is a phase of accelerated growth that follows the correction of these disorders, making it

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0028
Volume/Issue:
Volume 237: Issue 3
Free access
Siobhan M Donnelly
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Bao Tran Nguyen
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Scott Rhyne
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Jordan Estes
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Subrina Jesmin Department of Biology, Appalachian State University, Department of Internal Medicine, Tsukuba University, Rankin Science North Building N219, 572 River Street, Boone, North Carolina 28608, USA

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Chishimba Nathan Mowa
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Introduction During pregnancy, the uterine cervix undergoes a marked growth, particularly in the last half of pregnancy ( Downing & Sherwood 1985 , Hwang & Sherwood 1988 ), where uterine cervical weight gain increases as much as threefold in rats

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0469
Volume/Issue:
Volume 217: Issue 1
Free access
Andrei S Chagin Pediatric Endocrinology Laboratory, Department of Women's and Children's Health, Karolinska Institutet, Stockholm SE-17176, Sweden

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Elham Karimian Pediatric Endocrinology Laboratory, Department of Women's and Children's Health, Karolinska Institutet, Stockholm SE-17176, Sweden

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Katja Sundström Pediatric Endocrinology Laboratory, Department of Women's and Children's Health, Karolinska Institutet, Stockholm SE-17176, Sweden

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Emma Eriksson Pediatric Endocrinology Laboratory, Department of Women's and Children's Health, Karolinska Institutet, Stockholm SE-17176, Sweden

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Lars Sävendahl Pediatric Endocrinology Laboratory, Department of Women's and Children's Health, Karolinska Institutet, Stockholm SE-17176, Sweden

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Introduction Longitudinal bone growth occurs at the growth plate by a process called endochondral ossification where a continuously formed cartilage template is substituted by newly formed bone tissue ( Kronenberg 2003 , Chagin & Savendahl 2007

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-09-0307
Volume/Issue:
Volume 204: Issue 1
Free access
Claire L Wood Division of Functional Genetics and Development, Roslin Institute, University of Edinburgh, Edinburgh, UK
Translational and Clinical Research Institute, Newcastle University, Newcastle upon Tyne, UK

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Rob van ‘t Hof Institute of Ageing and Chronic Disease, University of Liverpool, Liverpool, UK

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Scott Dillon Division of Functional Genetics and Development, Roslin Institute, University of Edinburgh, Edinburgh, UK

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Volker Straub John Walton Muscular Dystrophy Research Centre, Newcastle University and Newcastle Hospitals NHS Foundation Trust, Newcastle upon Tyne, UK

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Sze C Wong Developmental Endocrinology Research Group, School of Medicine, University of Glasgow, Glasgow, UK

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S Faisal Ahmed Developmental Endocrinology Research Group, School of Medicine, University of Glasgow, Glasgow, UK

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Colin Farquharson Division of Functional Genetics and Development, Roslin Institute, University of Edinburgh, Edinburgh, UK

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established pharmacological intervention proven to stabilise muscle strength for a finite period of time ( Manzur et al. 2008 , Moxley et al. 2010 , Birnkrant et al. 2018 ). The side-effects of GC, however, are well recognised, with growth retardation

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-21-0388
Volume/Issue:
Volume 253: Issue 2
Open access
V E MacRae Bone Biology Group, Division of Gene Function and Development, Roslin Institute, Edinburgh EH25 9PS, UK
Bone and Endocrine Research Group, Royal Hospital for Sick Children, Glasgow G3 8SJ, UK

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C Farquharson Bone Biology Group, Division of Gene Function and Development, Roslin Institute, Edinburgh EH25 9PS, UK
Bone and Endocrine Research Group, Royal Hospital for Sick Children, Glasgow G3 8SJ, UK

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S F Ahmed Bone Biology Group, Division of Gene Function and Development, Roslin Institute, Edinburgh EH25 9PS, UK
Bone and Endocrine Research Group, Royal Hospital for Sick Children, Glasgow G3 8SJ, UK

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Introduction Adverse longitudinal growth in children with chronic inflammatory diseases is common, and can be attributed to a combination of systemic factors that include poor nutrition, excess glucocorticoids, sex steroid deficiency

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06609
Volume/Issue:
Volume 189: Issue 2
Free access
Sonnet S Jonker Center for Developmental Health, Knight Cardiovascular Institute, Oregon Health & Science University, Portland, Oregon, USA

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Daniel Kamna Center for Developmental Health, Knight Cardiovascular Institute, Oregon Health & Science University, Portland, Oregon, USA

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Dan LoTurco Department of Pediatrics, Perinatal Research Center, Anschutz Medical Campus, University of Colorado, Aurora, Colorado, USA

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Jenai Kailey Department of Pediatrics, Perinatal Research Center, Anschutz Medical Campus, University of Colorado, Aurora, Colorado, USA

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Laura D Brown Department of Pediatrics, Perinatal Research Center, Anschutz Medical Campus, University of Colorado, Aurora, Colorado, USA

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Introduction Intrauterine growth restriction (IUGR) results from inadequate placental function limiting fetal nutrient delivery, impairing normal growth and development ( Marconi & Paolini 2008 ). Infants affected by placental insufficiency

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0382
Volume/Issue:
Volume 239: Issue 2
Free access
Maryam Iravani Department of Women’s and Children’s Health, Karolinska Institutet, Stockholm, Sweden

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Marie Lagerquist Centre for Bone and Arthritis Research, Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden

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Claes Ohlsson Centre for Bone and Arthritis Research, Institute of Medicine, Sahlgrenska Academy at University of Gothenburg, Gothenburg, Sweden

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Lars Sävendahl Department of Women’s and Children’s Health, Karolinska Institutet, Stockholm, Sweden

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Introduction Longitudinal bone growth takes place in the growth plate, consisting of three layers: resting zone, proliferative zone and the hypertrophic zone. Bone growth is regulated by estrogens, acting either indirectly via the GH

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0263
Volume/Issue:
Volume 232: Issue 3
Free access
E A Parker Developmental Endocrinology Branch, National Institutes of Health, National Institute of Child Health and Human Development, Building 10/CRC, Rm 1-3330, MSC 1103, 10 Center drive, Bethesda, Maryland 20892, USA
Pediatric Endocrinology Unit, Department of Woman and Child Health, Karolinska Institute, Karolinska University Hospital, SE-171 76 Stockholm, Sweden

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A Hegde Developmental Endocrinology Branch, National Institutes of Health, National Institute of Child Health and Human Development, Building 10/CRC, Rm 1-3330, MSC 1103, 10 Center drive, Bethesda, Maryland 20892, USA
Pediatric Endocrinology Unit, Department of Woman and Child Health, Karolinska Institute, Karolinska University Hospital, SE-171 76 Stockholm, Sweden

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M Buckley Developmental Endocrinology Branch, National Institutes of Health, National Institute of Child Health and Human Development, Building 10/CRC, Rm 1-3330, MSC 1103, 10 Center drive, Bethesda, Maryland 20892, USA
Pediatric Endocrinology Unit, Department of Woman and Child Health, Karolinska Institute, Karolinska University Hospital, SE-171 76 Stockholm, Sweden

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K M Barnes Developmental Endocrinology Branch, National Institutes of Health, National Institute of Child Health and Human Development, Building 10/CRC, Rm 1-3330, MSC 1103, 10 Center drive, Bethesda, Maryland 20892, USA
Pediatric Endocrinology Unit, Department of Woman and Child Health, Karolinska Institute, Karolinska University Hospital, SE-171 76 Stockholm, Sweden

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J Baron Developmental Endocrinology Branch, National Institutes of Health, National Institute of Child Health and Human Development, Building 10/CRC, Rm 1-3330, MSC 1103, 10 Center drive, Bethesda, Maryland 20892, USA
Pediatric Endocrinology Unit, Department of Woman and Child Health, Karolinska Institute, Karolinska University Hospital, SE-171 76 Stockholm, Sweden

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O Nilsson Developmental Endocrinology Branch, National Institutes of Health, National Institute of Child Health and Human Development, Building 10/CRC, Rm 1-3330, MSC 1103, 10 Center drive, Bethesda, Maryland 20892, USA
Pediatric Endocrinology Unit, Department of Woman and Child Health, Karolinska Institute, Karolinska University Hospital, SE-171 76 Stockholm, Sweden

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Introduction The mammalian growth plate is a specialized cartilaginous structure at which longitudinal bone growth occurs. The growth plate is organized into three zones, the resting zone (RZ), the proliferative zone (PZ), and the

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0012
Volume/Issue:
Volume 194: Issue 1
Free access