Search Results

You are looking at 41 - 50 of 353 items for :

  • "null mice" x
  • Refine by access: All content x
Clear All
Young Hoon Son Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

Search for other papers by Young Hoon Son in
Google Scholar
PubMed
Close
,
Seok-Jin Lee Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

Search for other papers by Seok-Jin Lee in
Google Scholar
PubMed
Close
,
Ki-Baek Lee Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

Search for other papers by Ki-Baek Lee in
Google Scholar
PubMed
Close
,
Jin-Haeng Lee Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

Search for other papers by Jin-Haeng Lee in
Google Scholar
PubMed
Close
,
Eui Man Jeong Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea
Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

Search for other papers by Eui Man Jeong in
Google Scholar
PubMed
Close
,
Sun Gun Chung Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

Search for other papers by Sun Gun Chung in
Google Scholar
PubMed
Close
,
Sang-Chul Park Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

Search for other papers by Sang-Chul Park in
Google Scholar
PubMed
Close
, and
In-Gyu Kim Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea
Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

Search for other papers by In-Gyu Kim in
Google Scholar
PubMed
Close

al . 2008 ). Conversely, Cav1 -null mice displayed skeletal muscle abnormalities due to tubular aggregate formation ( Schubert et al . 2007 ) and exercise intolerance ( Razani et al . 2001 ), demonstrating that insulin/IGF signaling depends on the

Open access
E J Mackie School of Veterinary Science, University of Melbourne, Parkville, Victoria 3010, Australia

Search for other papers by E J Mackie in
Google Scholar
PubMed
Close
,
L Tatarczuch School of Veterinary Science, University of Melbourne, Parkville, Victoria 3010, Australia

Search for other papers by L Tatarczuch in
Google Scholar
PubMed
Close
, and
M Mirams School of Veterinary Science, University of Melbourne, Parkville, Victoria 3010, Australia

Search for other papers by M Mirams in
Google Scholar
PubMed
Close

IHH-null/Gli3-null mice ( Ehlen et al . 2006 ). IHH binds to aggrecan through its chondroitin sulphate side chains, and in the mouse growth plate normal sulphation of chondroitin sulphate is required for normal IHH protein distribution and signalling

Free access
Arnaud Lacombe Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

Search for other papers by Arnaud Lacombe in
Google Scholar
PubMed
Close
,
Vincent Lelievre Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

Search for other papers by Vincent Lelievre in
Google Scholar
PubMed
Close
,
Charles E Roselli Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

Search for other papers by Charles E Roselli in
Google Scholar
PubMed
Close
,
Jean-Marc Muller Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

Search for other papers by Jean-Marc Muller in
Google Scholar
PubMed
Close
,
James A Waschek Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

Search for other papers by James A Waschek in
Google Scholar
PubMed
Close
, and
Eric Vilain Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

Search for other papers by Eric Vilain in
Google Scholar
PubMed
Close

compared these data with what we previously observed in PACAP null mice ( Lacombe et al. 2006 ). We show that VIP-deficient males displayed lower serum concentrations of follicle-stimulating hormone (FSH) and a dramatic decrease in the serum

Free access
A J W Hsueh Division of Reproductive Biology, Department of Obstetrics and Gynecology, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, California, 94305-5317, USA
Endocrinology Unit and EA 1533, University Pierre et Marie Curie, Genetics of Human Reproduction, Hôpital Saint-Antoine, 184 rue de Fg St Antoine, 75012, Paris, France

Search for other papers by A J W Hsueh in
Google Scholar
PubMed
Close
,
P Bouchard Division of Reproductive Biology, Department of Obstetrics and Gynecology, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, California, 94305-5317, USA
Endocrinology Unit and EA 1533, University Pierre et Marie Curie, Genetics of Human Reproduction, Hôpital Saint-Antoine, 184 rue de Fg St Antoine, 75012, Paris, France

Search for other papers by P Bouchard in
Google Scholar
PubMed
Close
, and
I Ben-Shlomo Division of Reproductive Biology, Department of Obstetrics and Gynecology, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, California, 94305-5317, USA
Endocrinology Unit and EA 1533, University Pierre et Marie Curie, Genetics of Human Reproduction, Hôpital Saint-Antoine, 184 rue de Fg St Antoine, 75012, Paris, France

Search for other papers by I Ben-Shlomo in
Google Scholar
PubMed
Close

LGR4, 5, or 6. Although recent studies using LGR4- and LGR5-null mice indicated the important roles of LGR4 and LGR5 genes in embryonic development and perinatal survival ( Mazerbourg et al. 2004 a , Morita et al. 2004 ), the ligands for these

Free access
Jacob C Garza Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

Search for other papers by Jacob C Garza in
Google Scholar
PubMed
Close
,
Chung Sub Kim Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

Search for other papers by Chung Sub Kim in
Google Scholar
PubMed
Close
,
Jing Liu Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

Search for other papers by Jing Liu in
Google Scholar
PubMed
Close
,
Wei Zhang Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

Search for other papers by Wei Zhang in
Google Scholar
PubMed
Close
, and
Xin-Yun Lu Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

Search for other papers by Xin-Yun Lu in
Google Scholar
PubMed
Close

, and medial amygdala can completely reverse increased food intake without affecting energy expenditure. Additionally, they show that reactivating MC4R specifically in the PVN in the adult MC4R-null mice causes a mild reduction of body weight ( Balthasar

Free access
Shannon M Bailey Division of Molecular and Cellular Pathology, Division of Cardiovascular Diseases, Department of Pathology

Search for other papers by Shannon M Bailey in
Google Scholar
PubMed
Close
,
Uduak S Udoh Division of Molecular and Cellular Pathology, Division of Cardiovascular Diseases, Department of Pathology

Search for other papers by Uduak S Udoh in
Google Scholar
PubMed
Close
, and
Martin E Young Division of Molecular and Cellular Pathology, Division of Cardiovascular Diseases, Department of Pathology

Search for other papers by Martin E Young in
Google Scholar
PubMed
Close

null mice, concomitant with dysregulation of pyruvate dehydrogenase. Collectively, these observations suggest that circadian clocks within cardiac and skeletal myocytes significantly influence glucose utilization. Clock-mediated regulation of glucose

Free access
Barbara C Fam Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

Search for other papers by Barbara C Fam in
Google Scholar
PubMed
Close
,
Laura J Rose Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

Search for other papers by Laura J Rose in
Google Scholar
PubMed
Close
,
Rebecca Sgambellone Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

Search for other papers by Rebecca Sgambellone in
Google Scholar
PubMed
Close
,
Zheng Ruan Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

Search for other papers by Zheng Ruan in
Google Scholar
PubMed
Close
,
Joseph Proietto Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

Search for other papers by Joseph Proietto in
Google Scholar
PubMed
Close
, and
Sofianos Andrikopoulos Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

Search for other papers by Sofianos Andrikopoulos in
Google Scholar
PubMed
Close

that such differences in the phenotype of deleting GLUT4 could be attributed to the peculiarity of the background strain. In addition, in vitro studies using isolated muscles from the Glut4 null mice exhibited different capacities for insulin

Free access
Mohammed S Razzaque Department of Developmental Biology, Harvard School of Dental Medicine, Research and Education Building, Room # 304, 190 Longwood Avenue, Boston, Massachusetts 02115, USA

Search for other papers by Mohammed S Razzaque in
Google Scholar
PubMed
Close
and
Beate Lanske Department of Developmental Biology, Harvard School of Dental Medicine, Research and Education Building, Room # 304, 190 Longwood Avenue, Boston, Massachusetts 02115, USA

Search for other papers by Beate Lanske in
Google Scholar
PubMed
Close

determined, the clinical symptoms of hypophosphatemia in patients with ARHR are believed to be due to high circulatory levels of FGF23. Dmp-1 null mice have also high circulatory levels of Fgf23, and showed phenotypes resembling ARHR patients ( Feng et al

Free access
Shoshana Yakar National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by Shoshana Yakar in
Google Scholar
PubMed
Close
,
Mary L Bouxsein National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by Mary L Bouxsein in
Google Scholar
PubMed
Close
,
Ernesto Canalis National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by Ernesto Canalis in
Google Scholar
PubMed
Close
,
Hui Sun National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by Hui Sun in
Google Scholar
PubMed
Close
,
Vaida Glatt National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by Vaida Glatt in
Google Scholar
PubMed
Close
,
Caren Gundberg National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by Caren Gundberg in
Google Scholar
PubMed
Close
,
Pinchas Cohen National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by Pinchas Cohen in
Google Scholar
PubMed
Close
,
David Hwang National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by David Hwang in
Google Scholar
PubMed
Close
,
Yves Boisclair National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by Yves Boisclair in
Google Scholar
PubMed
Close
,
Derek LeRoith National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by Derek LeRoith in
Google Scholar
PubMed
Close
, and
Clifford J Rosen National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

Search for other papers by Clifford J Rosen in
Google Scholar
PubMed
Close

retardation, as assessed by body length and femoral length, as well as a significant reduction in femoral vBMD ( Yakar et al. 2002 a , 2002 b ). Interestingly, Igf1 null mice also exhibit a cortical phenotype similar to LA mice, but have increased

Free access
Giuseppina Maccarinelli
Search for other papers by Giuseppina Maccarinelli in
Google Scholar
PubMed
Close
,
Valeria Sibilia
Search for other papers by Valeria Sibilia in
Google Scholar
PubMed
Close
,
Antonio Torsello
Search for other papers by Antonio Torsello in
Google Scholar
PubMed
Close
,
Francesca Raimondo
Search for other papers by Francesca Raimondo in
Google Scholar
PubMed
Close
,
Marina Pitto
Search for other papers by Marina Pitto in
Google Scholar
PubMed
Close
,
Andrea Giustina
Search for other papers by Andrea Giustina in
Google Scholar
PubMed
Close
,
Carmela Netti
Search for other papers by Carmela Netti in
Google Scholar
PubMed
Close
, and
Daniela Cocchi
Search for other papers by Daniela Cocchi in
Google Scholar
PubMed
Close

osteoblasts from calvaria. At variance with our data, Sun et al. (2003) reported no role for ghrelin in the maintenance of bone density since ghrelin-null mice had normal bone mineral density and bone mineral content. Interestingly enough, food

Free access