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Young Hoon Son Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

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Seok-Jin Lee Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

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Ki-Baek Lee Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

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Jin-Haeng Lee Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

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Eui Man Jeong Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea
Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

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Sun Gun Chung Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

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Sang-Chul Park Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

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In-Gyu Kim Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea
Department of Biochemistry and Molecular Biology, Institute of Human–Environment Interface Biology, Department of Rehabilitation Medicine, Seoul National University College of Medicine, 103 Daehak‐ro, Jongno‐Gu, Seoul 110‐799, Korea

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al . 2008 ). Conversely, Cav1 -null mice displayed skeletal muscle abnormalities due to tubular aggregate formation ( Schubert et al . 2007 ) and exercise intolerance ( Razani et al . 2001 ), demonstrating that insulin/IGF signaling depends on the

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E J Mackie School of Veterinary Science, University of Melbourne, Parkville, Victoria 3010, Australia

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L Tatarczuch School of Veterinary Science, University of Melbourne, Parkville, Victoria 3010, Australia

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M Mirams School of Veterinary Science, University of Melbourne, Parkville, Victoria 3010, Australia

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IHH-null/Gli3-null mice ( Ehlen et al . 2006 ). IHH binds to aggrecan through its chondroitin sulphate side chains, and in the mouse growth plate normal sulphation of chondroitin sulphate is required for normal IHH protein distribution and signalling

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Arnaud Lacombe Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

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Vincent Lelievre Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

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Charles E Roselli Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

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Jean-Marc Muller Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

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James A Waschek Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

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Eric Vilain Departments of Human Genetics, Pediatrics, and Urology, David Geffen School of Medicine at UCLA, Gonda Center, University of California, Room 6357, 695 Charles Young Drive South, Los Angeles, California 90095-7088, USA
Institut National de la Santé et de la Recherche Médicale U676, Hôpital Robert-Debré, 48 Boulevard Sérurier, F-75019 Paris, France
Department of Physiology and Pharmacology L334, Oregon Health & Science University, 3181 Southwest Sam Jackson Park Road, Portland, Oregon 97201-3098, USA
Institut de Physiologie et Biologie Cellulaires, Centre National de la Recherche Scientifique-Unité Mixte de Recherche, 6187 Pôle Biologie Santé, 40 Avenue du Recteur Pineau, 86022 Poitiers, France
Mental Retardation Research Center, University of California, Neurosciences Research Building, 655 Charles Young Drive South, Los Angeles, California 90095-7088, USA

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compared these data with what we previously observed in PACAP null mice ( Lacombe et al. 2006 ). We show that VIP-deficient males displayed lower serum concentrations of follicle-stimulating hormone (FSH) and a dramatic decrease in the serum

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A J W Hsueh Division of Reproductive Biology, Department of Obstetrics and Gynecology, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, California, 94305-5317, USA
Endocrinology Unit and EA 1533, University Pierre et Marie Curie, Genetics of Human Reproduction, Hôpital Saint-Antoine, 184 rue de Fg St Antoine, 75012, Paris, France

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P Bouchard Division of Reproductive Biology, Department of Obstetrics and Gynecology, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, California, 94305-5317, USA
Endocrinology Unit and EA 1533, University Pierre et Marie Curie, Genetics of Human Reproduction, Hôpital Saint-Antoine, 184 rue de Fg St Antoine, 75012, Paris, France

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I Ben-Shlomo Division of Reproductive Biology, Department of Obstetrics and Gynecology, Stanford University School of Medicine, 300 Pasteur Drive, Stanford, California, 94305-5317, USA
Endocrinology Unit and EA 1533, University Pierre et Marie Curie, Genetics of Human Reproduction, Hôpital Saint-Antoine, 184 rue de Fg St Antoine, 75012, Paris, France

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LGR4, 5, or 6. Although recent studies using LGR4- and LGR5-null mice indicated the important roles of LGR4 and LGR5 genes in embryonic development and perinatal survival ( Mazerbourg et al. 2004 a , Morita et al. 2004 ), the ligands for these

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Jacob C Garza Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

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Chung Sub Kim Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

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Jing Liu Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

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Wei Zhang Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

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Xin-Yun Lu Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, Texas 78229, USA

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, and medial amygdala can completely reverse increased food intake without affecting energy expenditure. Additionally, they show that reactivating MC4R specifically in the PVN in the adult MC4R-null mice causes a mild reduction of body weight ( Balthasar

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Shannon M Bailey Division of Molecular and Cellular Pathology, Division of Cardiovascular Diseases, Department of Pathology

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Uduak S Udoh Division of Molecular and Cellular Pathology, Division of Cardiovascular Diseases, Department of Pathology

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Martin E Young Division of Molecular and Cellular Pathology, Division of Cardiovascular Diseases, Department of Pathology

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null mice, concomitant with dysregulation of pyruvate dehydrogenase. Collectively, these observations suggest that circadian clocks within cardiac and skeletal myocytes significantly influence glucose utilization. Clock-mediated regulation of glucose

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Barbara C Fam Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Laura J Rose Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Rebecca Sgambellone Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Zheng Ruan Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Joseph Proietto Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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Sofianos Andrikopoulos Department of Medicine (Austin Health), Austin Hospital, University of Melbourne, Level 7, Lance Townsend Building, Studley Road, Heidelberg, Victoria 3084, Australia

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that such differences in the phenotype of deleting GLUT4 could be attributed to the peculiarity of the background strain. In addition, in vitro studies using isolated muscles from the Glut4 null mice exhibited different capacities for insulin

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Mohammed S Razzaque Department of Developmental Biology, Harvard School of Dental Medicine, Research and Education Building, Room # 304, 190 Longwood Avenue, Boston, Massachusetts 02115, USA

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Beate Lanske Department of Developmental Biology, Harvard School of Dental Medicine, Research and Education Building, Room # 304, 190 Longwood Avenue, Boston, Massachusetts 02115, USA

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determined, the clinical symptoms of hypophosphatemia in patients with ARHR are believed to be due to high circulatory levels of FGF23. Dmp-1 null mice have also high circulatory levels of Fgf23, and showed phenotypes resembling ARHR patients ( Feng et al

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Shoshana Yakar National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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Mary L Bouxsein National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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Ernesto Canalis National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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Hui Sun National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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Vaida Glatt National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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Caren Gundberg National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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Pinchas Cohen National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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David Hwang National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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Yves Boisclair National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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Derek LeRoith National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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Clifford J Rosen National Institute of Diabetes, Digestive and Kidney Diseases, Bethesda, Maryland, USA
Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA
St Francis Hospital and Medical Center, Hartford, Connecticut, USA
Yale University School of Medicine, New Haven, Connecticut, USA
Mattel Hospital for Children, Los Angeles California, USA
The Department of Animal Science, Cornell University, Ithaca, New York, USA
The Jackson Laboratory, Bar Harbor, Maine, USA
Maine Center for Osteoporosis Research and Education, St Joseph Hospital, Maine, USA

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retardation, as assessed by body length and femoral length, as well as a significant reduction in femoral vBMD ( Yakar et al. 2002 a , 2002 b ). Interestingly, Igf1 null mice also exhibit a cortical phenotype similar to LA mice, but have increased

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Giuseppina Maccarinelli
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Valeria Sibilia
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Antonio Torsello
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Francesca Raimondo
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Marina Pitto
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Andrea Giustina
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Carmela Netti
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Daniela Cocchi
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osteoblasts from calvaria. At variance with our data, Sun et al. (2003) reported no role for ghrelin in the maintenance of bone density since ghrelin-null mice had normal bone mineral density and bone mineral content. Interestingly enough, food

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