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Koichi Suzuki and Leonard D Kohn

of the thyroid follicular cell, is a potent suppressor of thyroid-specific gene expression, and that this action of Tg overcomes TSH-mediated effects. Thus, it appears that Tg is a feedback suppressor of its own expression, as well as of TPO, NIS and

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Won Bae Kim, Christopher J Lewis, Kelly D McCall, Ramiro Malgor, Aimee D Kohn, Randall T Moon, and Leonard D Kohn

), sodium iodide symporter, and the thyroid-stimulating hormone receptor (TSHR) genes, all of which are under the regulatory control of TSH and insulin/insulin-like growth factor-I (IGF-I). Moreover, the growth of FRTL-5 cells is regulated by several

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Bert De Groef, Sylvia V H Grommen, and Veerle M Darras

even more dramatic surge of plasma 3,5,3′-tri-iodothyronine (T 3 ). The underlying cause of augmented TH secretion shortly before hatching is unknown. Some authors have speculated that elevated thyroidal and pituitary sensitivity to thyrotrophin (TSH

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Isabela Teixeira Bonomo, Patrícia Cristina Lisboa, Magna Cottini Fonseca Passos, Simone Bezerra Alves, Adelina Martha Reis, and Egberto Gaspar de Moura

, and liver. Thyrotropin (TSH), T 3 , and T 4 serum concentrations TSH was measured by specific RIA, using a kit for rat TSH supplied by the NIDDKD (NIH) and data were expressed in terms of the reference preparation provided (RP-3). The intra

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Michael Muchow, Ioannis Bossis, and Tom E Porter

Introduction Thyroid hormones are involved in the regulation of growth, metabolism, heat production, gonadal development, molting, migration, and hatching in birds ( McNabb 2000 ). Thyroid-stimulating hormone (TSH) plays an important

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Patricia C Lisboa, Ellen P S Conceição, Elaine de Oliveira, and Egberto G Moura

understanding regarding the degree of hypothyroidism previously reported in EO animal models. Because circulating thyrotropin (TSH) was unaltered in adult small litter (SL) rats, we evaluated the thyrotropin release hormone (TRH) in the hypothalamus, the TSH in

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Michelle P Marassi, Rodrigo S Fortunato, Alba C Matos da Silva, Valmara S Pereira, Denise P Carvalho, Doris Rosenthal, and Vânia M Corrêa da Costa

ascertained ( Donda et al. 1990 ). Cheron et al. (1980) reported lower serum T3 and higher serum thyroid-stimulating hormone (TSH) in adults than in 21- and 30-day-old pre-pubertal male (Pm) rats, without any significant difference in serum T4

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Camila Calvino, Luana L Souza, Ricardo H Costa-e-Sousa, Norma A S Almeida, Isis H Trevenzoli, and Carmen C Pazos-Moura

TSH concentration in rats, potentially due to leptin action at the hypothalamus, as the direct pituitary effect of leptin on TSH release was inhibitory, potentially as a result of an autocrine–paracrine effect exerted by locally produced leptin

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Juan C Solis-S, Patricia Villalobos, Aurea Orozco, Guadalupe Delgado, Andres Quintanar-Stephano, Pablo Garcia-Solis, Hebert L Hernandez-Montiel, Ludivina Robles-Osorio, and Carlos Valverde-R

in intact rats reported that TSH administration increases rat tDh (rtDh) activity ( Roche et al . 1953 , Maayan & Rosenberg 1963 ). Similarly, thyroidal ID1 activity ( Erickson et al . 1982 ) and mRNA ( Toyoda et al . 1992 ) are increased by TSH

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Shona Wood and Andrew Loudon

β sub-units of thyroid-stimulating hormone (TSH). These cells lack receptors for the hypothalamic thyrotropin-releasing hormone (TRH) ( Bockmann et al . 1997 ), and do not respond to conventional hypothalamic outputs. In mammals, the MT1 receptor co