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Felipe de Oliveira Franco Laboratory of Adipose Tissue Biology, Center for Integrated Biotechnology, University of Mogi das Cruzes, Mogi das Cruzes, Brazil

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Magno Alves Lopes Laboratory of Adipose Tissue Biology, Center for Integrated Biotechnology, University of Mogi das Cruzes, Mogi das Cruzes, Brazil

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Felipe dos Santos Henriques Laboratory of Adipose Tissue Biology, Center for Integrated Biotechnology, University of Mogi das Cruzes, Mogi das Cruzes, Brazil

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Rodrigo Xavier das Neves Cancer Metabolism Research Group, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil

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Cesário Bianchi Filho Center for Clinical and Translational Research, University of Mogi das Cruzes, Mogi das Cruzes, Brazil

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Miguel Luiz Batista Jr Laboratory of Adipose Tissue Biology, Center for Integrated Biotechnology, University of Mogi das Cruzes, Mogi das Cruzes, Brazil
Cancer Metabolism Research Group, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil

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was similar ( Table 1 ). To evaluate cell turnover, we measured the mRNA levels of Pref1 (pre-adipocyte marker) because its expression is linked to adipogenesis inhibition ( O’Connell et al . 2011 ) and the mRNA levels of Adiponectin , which have

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0305
Volume/Issue:
Volume 232: Issue 3
Free access
Lili Guo Department of Endocrinology, Medical College, Department of Physiology, Department of Physical Education, Clinical Medical College, Yangzhou University, Nantong West Street No. 98, Yangzhou, Jiangsu 225001, China

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Penghua Fang Department of Endocrinology, Medical College, Department of Physiology, Department of Physical Education, Clinical Medical College, Yangzhou University, Nantong West Street No. 98, Yangzhou, Jiangsu 225001, China

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Mei Yu Department of Endocrinology, Medical College, Department of Physiology, Department of Physical Education, Clinical Medical College, Yangzhou University, Nantong West Street No. 98, Yangzhou, Jiangsu 225001, China

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Mingyi Shi Department of Endocrinology, Medical College, Department of Physiology, Department of Physical Education, Clinical Medical College, Yangzhou University, Nantong West Street No. 98, Yangzhou, Jiangsu 225001, China

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Ping Bo Department of Endocrinology, Medical College, Department of Physiology, Department of Physical Education, Clinical Medical College, Yangzhou University, Nantong West Street No. 98, Yangzhou, Jiangsu 225001, China
Department of Endocrinology, Medical College, Department of Physiology, Department of Physical Education, Clinical Medical College, Yangzhou University, Nantong West Street No. 98, Yangzhou, Jiangsu 225001, China

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Zhenwen Zhang Department of Endocrinology, Medical College, Department of Physiology, Department of Physical Education, Clinical Medical College, Yangzhou University, Nantong West Street No. 98, Yangzhou, Jiangsu 225001, China

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). Retinol-binding protein 4 (RBP4) and adiponectin ELISA kits were obtained from Uscn Life Science (Wuhan, China). Ala6–25Cys (SSPFPPRPTRAGRETQLLRSC) were custom synthesized by GL Biochem (Shanghai, China). Animals and i.c.v. injection All animal procedures

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0102
Volume/Issue:
Volume 223: Issue 3
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Rengasamy Palanivel Department of Biology, York University, Toronto, Ontario, Canada M3J 1P3

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Vivian Vu Department of Biology, York University, Toronto, Ontario, Canada M3J 1P3

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Min Park Department of Biology, York University, Toronto, Ontario, Canada M3J 1P3

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Xiangping Fang Department of Biology, York University, Toronto, Ontario, Canada M3J 1P3

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Gary Sweeney Department of Biology, York University, Toronto, Ontario, Canada M3J 1P3

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, Karmazyn et al . 2007 ). These include factors such as leptin, adiponectin, and TNF. It is also of interest to note that many of these factors have now been shown to be produced by the heart itself ( Purdham et al . 2004 , Pineiro et al . 2005 , Guo

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0336
Volume/Issue:
Volume 199: Issue 3
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Clare M Reynolds Liggins Institute, University of Auckland, Auckland, New Zealand

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Mark H Vickers Liggins Institute, University of Auckland, Auckland, New Zealand

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et al. 1994 , Ahima et al. 2000 ) led to profound changes in the field of metabolic physiology. Following the characterisation of leptin in 1994, a large number of adipokines have now been characterised including adiponectin, apelin, visfatin and

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-18-0686
Volume/Issue:
Volume 242: Issue 1
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Xiwen Xiong Department of Forensic Medicine, Xinxiang Medical University, Xinxiang, Henan, China

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Cuicui Zhang School of Basic Medical Sciences, Xinxiang Medical University, Xinxiang, Henan, China

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Yang Zhang Department of Biochemistry and Molecular Biology, Indiana University School of Medicine, Indianapolis, USA

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Rui Fan School of Basic Medical Sciences, Xinxiang Medical University, Xinxiang, Henan, China

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Xinlai Qian School of Basic Medical Sciences, Xinxiang Medical University, Xinxiang, Henan, China

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X Charlie Dong Department of Biochemistry and Molecular Biology, Indiana University School of Medicine, Indianapolis, USA

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key regulator of energy metabolism by secreting a diverse range of adipokines, such as leptin and adiponectin ( Galic et al. 2010 ). Therefore, adipose tissue is considered as highly important for systemic metabolic homeostasis. The sirtuin

Article Type:
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DOI:
https://doi.org/10.1530/JOE-17-0033
Volume/Issue:
Volume 233: Issue 3
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Gulizar Issa Ameen Department of Cellular and Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, UK

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Silvia Mora Department of Cellular and Molecular Physiology, Institute of Translational Medicine, University of Liverpool, Liverpool, UK

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that help regulate biological functions in other cells and organs. Some of these proteins like adiponectin increase insulin sensitivity in peripheral organs, some arepro-inflammatory such as IL6 or TNFa and inhibit insulin action and others like leptin

Article Type:
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DOI:
https://doi.org/10.1530/JOE-17-0359
Volume/Issue:
Volume 236: Issue 1
Open access
R G Ahmed Division of Anatomy and Embryology, Zoology Department, Faculty of Science, Beni-Suef University, Beni-Suef, PO 62514, Egypt

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been shown to disrupt adiponectin ( Mullerova et al . 2008 ), leptin ( Nieminen et al . 2000 ), tumor necrosis factor (TNFα; Arsenescu et al . 2008 ), and insulin ( Everett et al . 2011 ) concentrations, body weight, energy expenditure and fat

Article Type:
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DOI:
https://doi.org/10.1530/JOE-13-0302
Volume/Issue:
Volume 219: Issue 3
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Bernard Beck INSERM, Université Henri Poincaré, U724, Faculté de Médecine, BP 184 F 54505 Vandœuvre-les-Nancy, France
INSERM, Université Henri Poincaré, U724, Faculté de Médecine, BP 184 F 54505 Vandœuvre-les-Nancy, France

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Sébastien Richy INSERM, Université Henri Poincaré, U724, Faculté de Médecine, BP 184 F 54505 Vandœuvre-les-Nancy, France

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adiponectin ( Havel 2004 , Kadowaki & Yamauchi 2005 ). It also modulates gastric hormones involved in feeding regulation like ghrelin ( Beck et al . 2002 , Erdmann et al . 2003 , Greenman et al . 2004 , Moesgaard et al . 2004 , Sanchez et al . 2004

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0468
Volume/Issue:
Volume 196: Issue 1
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M S Fernandes Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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V Pierron Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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D Michalovich Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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S Astle Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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S Thornton Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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H Peltoketo Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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E W-F Lam Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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B Gellersen Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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I Huhtaniemi Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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J Allen Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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J J Brosens Institute of Reproductive and Developmental Biology, Wolfson & Weston Research Centre for Family Health, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Inpharmatica Ltd, London Bioscience Innovation Centre, 2 Royal College Street, London NW1 0NH, UK
Biomedical Research Institute, Department of Biological Sciences, Warwick Medical School, University of Warwick, Coventry CV4 7AL, UK
Cancer Research-UK Labs and Section of Cancer Cell Biology, Department of Cancer Medicine, Imperial College London, Hammersmith Hospital, London W12 0NN, UK
Endokrinologikum Hamburg, Falkenried 88, 20251 Hamburg, Germany

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because several of its members are steroid or adiponectin (AdipoQ) receptors ( Lyons et al. 2004 ). Initial studies indicated that the expression of mPRs is highly tissue-specific, with mPRα being the predominant isoform in reproductive tissues

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06242
Volume/Issue:
Volume 187: Issue 1
Free access
Min Kyong Moon Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea
Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea

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In-Kyong Jeong Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea

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Tae Jung Oh Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea

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Hwa Young Ahn Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea
Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea

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Hwan Hee Kim Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea

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Young Joo Park Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea

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Hak Chul Jang Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea
Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea

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Kyong Soo Park Department of Internal Medicine, Department of Internal Medicine, Department of Internal Medicine, Clinical Research Institute, Department of Internal Medicine, Seoul National University College of Medicine, 101 Daehak-ro, Jongno-gu, Seoul 110-744, Republic of Korea

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, Alonso-Magdalena et al . 2006 ), decreased production or secretion of adiponectin from adipocytes, the establishment of insulin-sensitizing adipocytokine ( Hugo et al . 2008 , Kidani et al . 2010 ), enhanced adipocyte differentiation and lipid

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0714
Volume/Issue:
Volume 226: Issue 1
Free access