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percentage of DAB-positive cells within specific epithelial structures, i.e., TEBs, TD and ducts, Abs, and Lob 1. Data from different groups were analyzed by ANOVA and unpaired t -tests. Determination of gene expression profile by microarrays Total RNA from
Cell Biology and Biochemistry,
Neuropsychiatry, and
Pathology, Texas Tech University Health Sciences Center, Lubbock, Texas 79430, USA
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Cell Biology and Biochemistry,
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Pathology, Texas Tech University Health Sciences Center, Lubbock, Texas 79430, USA
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Cell Biology and Biochemistry,
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Pathology, Texas Tech University Health Sciences Center, Lubbock, Texas 79430, USA
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Cell Biology and Biochemistry,
Neuropsychiatry, and
Pathology, Texas Tech University Health Sciences Center, Lubbock, Texas 79430, USA
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Cell Biology and Biochemistry,
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Pathology, Texas Tech University Health Sciences Center, Lubbock, Texas 79430, USA
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Cell Biology and Biochemistry,
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Pathology, Texas Tech University Health Sciences Center, Lubbock, Texas 79430, USA
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Cell Biology and Biochemistry,
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Pathology, Texas Tech University Health Sciences Center, Lubbock, Texas 79430, USA
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membrane ( Clark et al. 1994 , Lin et al. 1995 , Wang et al. 1998 , Stocco 2001 ). In testicular Leydig cells, StAR gene expression is primarily regulated by luteinizing hormone (LH), but the mechanisms responsible for LH regulation of StAR gene
Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
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Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
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Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
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Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
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Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
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Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
Department of Physiology, Division of Cell and Molecular Biology, Department of Medicine, Division of Endocrinology and Metabolism, Department of Laboratory Medicine and Pathobiology, Department of Nutrition, University of Toronto, Toronto, Ontario, Canada M5S 1A8
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detection of expression of the Tcf7 , Tcf7l1 , and Lef1 genes are shown in Table 1 . PCR products were purified, inserted into TA cloning vectors, and verified by DNA sequencing. As shown in Fig. 3 , in addition to Tcf7l2 , both Tcf7 and Tcf7l1
Medical Microbiology and
Biochemistry, Faculty of Medicine and Health Sciences, United Arab Emirates University, PO Box 17666, Al Ain, United Arab Emirates
Department of Obstetrics and Gynecology, University Hospital, SE 221 85, Lund, Sweden
Zoonoses and Emerging Infections Group, Clinical Virology, University of Veterinary Medicine, Clinical Department of Diagnostic Imaging, Infectious Diseases and Clinical Pathology, Vienna, Veterinärplatz 1, A-1210 Vienna, Austria
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Medical Microbiology and
Biochemistry, Faculty of Medicine and Health Sciences, United Arab Emirates University, PO Box 17666, Al Ain, United Arab Emirates
Department of Obstetrics and Gynecology, University Hospital, SE 221 85, Lund, Sweden
Zoonoses and Emerging Infections Group, Clinical Virology, University of Veterinary Medicine, Clinical Department of Diagnostic Imaging, Infectious Diseases and Clinical Pathology, Vienna, Veterinärplatz 1, A-1210 Vienna, Austria
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Medical Microbiology and
Biochemistry, Faculty of Medicine and Health Sciences, United Arab Emirates University, PO Box 17666, Al Ain, United Arab Emirates
Department of Obstetrics and Gynecology, University Hospital, SE 221 85, Lund, Sweden
Zoonoses and Emerging Infections Group, Clinical Virology, University of Veterinary Medicine, Clinical Department of Diagnostic Imaging, Infectious Diseases and Clinical Pathology, Vienna, Veterinärplatz 1, A-1210 Vienna, Austria
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Medical Microbiology and
Biochemistry, Faculty of Medicine and Health Sciences, United Arab Emirates University, PO Box 17666, Al Ain, United Arab Emirates
Department of Obstetrics and Gynecology, University Hospital, SE 221 85, Lund, Sweden
Zoonoses and Emerging Infections Group, Clinical Virology, University of Veterinary Medicine, Clinical Department of Diagnostic Imaging, Infectious Diseases and Clinical Pathology, Vienna, Veterinärplatz 1, A-1210 Vienna, Austria
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Medical Microbiology and
Biochemistry, Faculty of Medicine and Health Sciences, United Arab Emirates University, PO Box 17666, Al Ain, United Arab Emirates
Department of Obstetrics and Gynecology, University Hospital, SE 221 85, Lund, Sweden
Zoonoses and Emerging Infections Group, Clinical Virology, University of Veterinary Medicine, Clinical Department of Diagnostic Imaging, Infectious Diseases and Clinical Pathology, Vienna, Veterinärplatz 1, A-1210 Vienna, Austria
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Medical Microbiology and
Biochemistry, Faculty of Medicine and Health Sciences, United Arab Emirates University, PO Box 17666, Al Ain, United Arab Emirates
Department of Obstetrics and Gynecology, University Hospital, SE 221 85, Lund, Sweden
Zoonoses and Emerging Infections Group, Clinical Virology, University of Veterinary Medicine, Clinical Department of Diagnostic Imaging, Infectious Diseases and Clinical Pathology, Vienna, Veterinärplatz 1, A-1210 Vienna, Austria
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). In the present article, we report on the quantitative transcription of the OT gene and the identification of OT in the uterus of pregnant rats and describe the dependence of OT gene expression on sex steroids in ovariectomized rats
Obstetrics and Gynecology, McGill University, 3655 Promenade Sir-William-Osler, Montréal, Québec, Canada H3G 1Y6
Toxicology Research Division, Health Products and Foods Branch, Food Directorate, Health Canada, Ottawa, Ontario, Canada
Reproductive Biology Unit, Departments of Cellular and Molecular Medicine and Obstetrics and Gynecology, University of Ottawa, Sir Frederick G Banting Research Centre, 2202D1 Tunney’s Pasture, Ottawa, Ontario, Canada K1A 0L2
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Obstetrics and Gynecology, McGill University, 3655 Promenade Sir-William-Osler, Montréal, Québec, Canada H3G 1Y6
Toxicology Research Division, Health Products and Foods Branch, Food Directorate, Health Canada, Ottawa, Ontario, Canada
Reproductive Biology Unit, Departments of Cellular and Molecular Medicine and Obstetrics and Gynecology, University of Ottawa, Sir Frederick G Banting Research Centre, 2202D1 Tunney’s Pasture, Ottawa, Ontario, Canada K1A 0L2
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Obstetrics and Gynecology, McGill University, 3655 Promenade Sir-William-Osler, Montréal, Québec, Canada H3G 1Y6
Toxicology Research Division, Health Products and Foods Branch, Food Directorate, Health Canada, Ottawa, Ontario, Canada
Reproductive Biology Unit, Departments of Cellular and Molecular Medicine and Obstetrics and Gynecology, University of Ottawa, Sir Frederick G Banting Research Centre, 2202D1 Tunney’s Pasture, Ottawa, Ontario, Canada K1A 0L2
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by complex region-specific gene expression profiles ( Jervis & Robaire 2001 , Cornwall et al. 2002 ). Theunique patternsofgene expression alongtheduct contribute to the evolving repertoire of components that make up the highly specialized luminal
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of the steroidogenic cytochrome P450 genes in endocrine tissues, such as the adrenal cortex, testis, and ovary ( Morohashi et al . 1992 ). Previously, Crawford et al . (1997) using stable ectopic expression of NF5A1 in mouse embryonic stem (ES
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using a blocker, verapamil ( Manna et al . 1999 ). However, observations on the roles of L-type Ca 2+ channels in Star gene expression and steroidogenesis were not consistent in the previous studies. While blocking Ca 2+ influx through L-type Ca 2
Agrarian Sector, Federal University of Paraná, Veterinary Hospital, Curitiba, Brazil
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Department of Anatomy, Federal University of Pernambuco, Recife, Brazil
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molecular analysis by real-time PCR. Analysis of the gene expression of tyrosine hydroxylase, Drd 1 a , and Drd 2 in the nucleus accumbens and striatum Gene expression analysis was performed in groups (TH): UC n = 5, TC ( n = 4), UHF ( n
Immunology Research Center, Department of Immunology, Department of Hematology, Endocrinology Department, Gazi University, 06500 Ankara, Turkey
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investigate the expression and biological function(s) of the TSHR in hBMSCs. TSH-induced gene expression was explored particularly. Materials and Methods Isolation and maintenance of mesenchymal stem cells hBMSCs were isolated by plastic adherence according to
Departments of Physiology and Pharmacology, Obstetrics and Gynecology, The Children's Health Research Institute, The Lawson Health Research Institute, The University of Western Ontario, London, Ontario, Canada N6A 5C1
Departments of Physiology and Pharmacology, Obstetrics and Gynecology, The Children's Health Research Institute, The Lawson Health Research Institute, The University of Western Ontario, London, Ontario, Canada N6A 5C1
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Departments of Physiology and Pharmacology, Obstetrics and Gynecology, The Children's Health Research Institute, The Lawson Health Research Institute, The University of Western Ontario, London, Ontario, Canada N6A 5C1
Departments of Physiology and Pharmacology, Obstetrics and Gynecology, The Children's Health Research Institute, The Lawson Health Research Institute, The University of Western Ontario, London, Ontario, Canada N6A 5C1
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Departments of Physiology and Pharmacology, Obstetrics and Gynecology, The Children's Health Research Institute, The Lawson Health Research Institute, The University of Western Ontario, London, Ontario, Canada N6A 5C1
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Departments of Physiology and Pharmacology, Obstetrics and Gynecology, The Children's Health Research Institute, The Lawson Health Research Institute, The University of Western Ontario, London, Ontario, Canada N6A 5C1
Departments of Physiology and Pharmacology, Obstetrics and Gynecology, The Children's Health Research Institute, The Lawson Health Research Institute, The University of Western Ontario, London, Ontario, Canada N6A 5C1
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Departments of Physiology and Pharmacology, Obstetrics and Gynecology, The Children's Health Research Institute, The Lawson Health Research Institute, The University of Western Ontario, London, Ontario, Canada N6A 5C1
Departments of Physiology and Pharmacology, Obstetrics and Gynecology, The Children's Health Research Institute, The Lawson Health Research Institute, The University of Western Ontario, London, Ontario, Canada N6A 5C1
Departments of Physiology and Pharmacology, Obstetrics and Gynecology, The Children's Health Research Institute, The Lawson Health Research Institute, The University of Western Ontario, London, Ontario, Canada N6A 5C1
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implicated in regulating genes involved in the metabolism and transport of cholesterol ( Lehmann et al . 1997 , Venkateswaran et al . 2000 ) and in enhancing the expression of lipogenic enzymes ( Repa et al . 2000 ). Recent studies have also demonstrated