Search Results
Jee H Lee
Jee H Lee
Laboratory of Molecular Endocrinology and Diabetes Unit, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Biochemistry, Faculty of Medicine, University of Hong Kong, Pokfulam, Hong Kong SAR, China
Search for other papers by Jee H Lee in
Google Scholar
PubMed
Department of Biochemistry, Faculty of Medicine, University of Hong Kong, Pokfulam, Hong Kong SAR, China
Search for other papers by Jee H Lee in
Google Scholar
PubMed
Jamie L Volinic
Jamie L Volinic
Laboratory of Molecular Endocrinology and Diabetes Unit, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Biochemistry, Faculty of Medicine, University of Hong Kong, Pokfulam, Hong Kong SAR, China
Search for other papers by Jamie L Volinic in
Google Scholar
PubMed
Department of Biochemistry, Faculty of Medicine, University of Hong Kong, Pokfulam, Hong Kong SAR, China
Search for other papers by Jamie L Volinic in
Google Scholar
PubMed
Constanze Banz
Constanze Banz
Laboratory of Molecular Endocrinology and Diabetes Unit, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Biochemistry, Faculty of Medicine, University of Hong Kong, Pokfulam, Hong Kong SAR, China
Search for other papers by Constanze Banz in
Google Scholar
PubMed
Department of Biochemistry, Faculty of Medicine, University of Hong Kong, Pokfulam, Hong Kong SAR, China
Search for other papers by Constanze Banz in
Google Scholar
PubMed
Kwok-Ming Yao
Kwok-Ming Yao
Laboratory of Molecular Endocrinology and Diabetes Unit, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Biochemistry, Faculty of Medicine, University of Hong Kong, Pokfulam, Hong Kong SAR, China
Search for other papers by Kwok-Ming Yao in
Google Scholar
PubMed
Department of Biochemistry, Faculty of Medicine, University of Hong Kong, Pokfulam, Hong Kong SAR, China
Search for other papers by Kwok-Ming Yao in
Google Scholar
PubMed
Melissa K Thomas
Melissa K Thomas
Laboratory of Molecular Endocrinology and Diabetes Unit, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Biochemistry, Faculty of Medicine, University of Hong Kong, Pokfulam, Hong Kong SAR, China
Search for other papers by Melissa K Thomas in
Google Scholar
PubMed
Department of Biochemistry, Faculty of Medicine, University of Hong Kong, Pokfulam, Hong Kong SAR, China
Search for other papers by Melissa K Thomas in
Google Scholar
PubMed
complexes through interactions with a large repertoire of transcription factors and components of basal transcription machinery ( Chan & La Thangue 2001 , Vo & Goodman 2001 ). The intrinsic acetyltransferase activity of p300 augments the activation of gene
Gary B Silberstein
Gary B Silberstein
Department of Molecular, Cell and Developmental Biology, Sinsheimer Laboratories, University of California, Santa Cruz, California 95064, USA
Search for other papers by Gary B Silberstein in
Google Scholar
PubMed
Search for other papers by Gary B Silberstein in
Google Scholar
PubMed
Katharine Van Horn
Katharine Van Horn
Department of Molecular, Cell and Developmental Biology, Sinsheimer Laboratories, University of California, Santa Cruz, California 95064, USA
Search for other papers by Katharine Van Horn in
Google Scholar
PubMed
Search for other papers by Katharine Van Horn in
Google Scholar
PubMed
Eva Hrabeta-Robinson
Eva Hrabeta-Robinson
Department of Molecular, Cell and Developmental Biology, Sinsheimer Laboratories, University of California, Santa Cruz, California 95064, USA
Search for other papers by Eva Hrabeta-Robinson in
Google Scholar
PubMed
Search for other papers by Eva Hrabeta-Robinson in
Google Scholar
PubMed
Jennifer Compton
Jennifer Compton
Department of Molecular, Cell and Developmental Biology, Sinsheimer Laboratories, University of California, Santa Cruz, California 95064, USA
Search for other papers by Jennifer Compton in
Google Scholar
PubMed
Search for other papers by Jennifer Compton in
Google Scholar
PubMed
RANKL. RANKL is the ligand for receptor activator of nuclear factor-kappaB (RANK) and is induced by progesterone ( Fata et al. 2000 , Brisken et al. 2002 ). Pax-2 is a proto-oncogene transcription factor characterized by a paired domain and
Tsun-Jui Liu
Tsun-Jui Liu
Department of Medicine, Biological Chemistry, Physiology and Biophysics, Center for Diabetes Research and Treatment, University of California, Irvine, California 92697, USA
Taichung Veterans General Hospital and Institute of Clinical Medicine, National Yang-Ming University, Taipei, Taiwan
Search for other papers by Tsun-Jui Liu in
Google Scholar
PubMed
Taichung Veterans General Hospital and Institute of Clinical Medicine, National Yang-Ming University, Taipei, Taiwan
Search for other papers by Tsun-Jui Liu in
Google Scholar
PubMed
Hui-Chin Lai
Hui-Chin Lai
Department of Medicine, Biological Chemistry, Physiology and Biophysics, Center for Diabetes Research and Treatment, University of California, Irvine, California 92697, USA
Taichung Veterans General Hospital and Institute of Clinical Medicine, National Yang-Ming University, Taipei, Taiwan
Search for other papers by Hui-Chin Lai in
Google Scholar
PubMed
Taichung Veterans General Hospital and Institute of Clinical Medicine, National Yang-Ming University, Taipei, Taiwan
Search for other papers by Hui-Chin Lai in
Google Scholar
PubMed
Chih-Tai Ting
Chih-Tai Ting
Department of Medicine, Biological Chemistry, Physiology and Biophysics, Center for Diabetes Research and Treatment, University of California, Irvine, California 92697, USA
Taichung Veterans General Hospital and Institute of Clinical Medicine, National Yang-Ming University, Taipei, Taiwan
Search for other papers by Chih-Tai Ting in
Google Scholar
PubMed
Taichung Veterans General Hospital and Institute of Clinical Medicine, National Yang-Ming University, Taipei, Taiwan
Search for other papers by Chih-Tai Ting in
Google Scholar
PubMed
Ping H Wang
Ping H Wang
Department of Medicine, Biological Chemistry, Physiology and Biophysics, Center for Diabetes Research and Treatment, University of California, Irvine, California 92697, USA
Taichung Veterans General Hospital and Institute of Clinical Medicine, National Yang-Ming University, Taipei, Taiwan
Search for other papers by Ping H Wang in
Google Scholar
PubMed
Taichung Veterans General Hospital and Institute of Clinical Medicine, National Yang-Ming University, Taipei, Taiwan
Search for other papers by Ping H Wang in
Google Scholar
PubMed
signaling in cardiomyocytes will help elucidate how hormonal signaling modulates myocardial function. Insulin and IGF-I could prevent skeletal muscle cell atrophy and promote myogenesis through forkhead transcription factors (FOXO; Hribal et al
Marcello Maggiolini
Marcello Maggiolini
Search for other papers by Marcello Maggiolini in
Google Scholar
PubMed
Search for other papers by Marcello Maggiolini in
Google Scholar
PubMed
Didier Picard
Didier Picard
Department of Pharmaco-Biology, Département de Biologie Cellulaire, University of Calabria, 87030 Rende, Italy
Search for other papers by Didier Picard in
Google Scholar
PubMed
Search for other papers by Didier Picard in
Google Scholar
PubMed
that GPR30 signaling triggers lead to the induced expression of c-fos in macrophages ( Kanda & Watanabe 2003 a ) and activation of the transcription factor CREB in keratinocytes ( Kanda & Watanabe 2003 b , 2004 ). These in turn activate the
Aarti D Rohira
Aarti D Rohira
Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA
Search for other papers by Aarti D Rohira in
Google Scholar
PubMed
Search for other papers by Aarti D Rohira in
Google Scholar
PubMed
David M Lonard
David M Lonard
Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA
Search for other papers by David M Lonard in
Google Scholar
PubMed
Search for other papers by David M Lonard in
Google Scholar
PubMed
Bert W O’Malley
Bert W O’Malley
Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA
Search for other papers by Bert W O’Malley in
Google Scholar
PubMed
Search for other papers by Bert W O’Malley in
Google Scholar
PubMed
production which acts by regulating stromal COUP-TFII signaling to promote decidualization. In another example, conditional deletion of the ER gene in the uterine epithelial compartment leads to loss of LIF production. Furthermore, the transcription factor
Lei Ye
Lei Ye
Shanghai Clinical Center for Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Second Medical University, 197 Ruijin Er Lu, Shanghai 200025, People’s Republic of China
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Lei Ye in
Google Scholar
PubMed
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Lei Ye in
Google Scholar
PubMed
Xiaoying Li
Xiaoying Li
Shanghai Clinical Center for Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Second Medical University, 197 Ruijin Er Lu, Shanghai 200025, People’s Republic of China
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Xiaoying Li in
Google Scholar
PubMed
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Xiaoying Li in
Google Scholar
PubMed
Xiangyin Kong
Xiangyin Kong
Shanghai Clinical Center for Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Second Medical University, 197 Ruijin Er Lu, Shanghai 200025, People’s Republic of China
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Xiangyin Kong in
Google Scholar
PubMed
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Xiangyin Kong in
Google Scholar
PubMed
Weiqing Wang
Weiqing Wang
Shanghai Clinical Center for Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Second Medical University, 197 Ruijin Er Lu, Shanghai 200025, People’s Republic of China
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Weiqing Wang in
Google Scholar
PubMed
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Weiqing Wang in
Google Scholar
PubMed
Yufang Bi
Yufang Bi
Shanghai Clinical Center for Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Second Medical University, 197 Ruijin Er Lu, Shanghai 200025, People’s Republic of China
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Yufang Bi in
Google Scholar
PubMed
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Yufang Bi in
Google Scholar
PubMed
Landian Hu
Landian Hu
Shanghai Clinical Center for Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Second Medical University, 197 Ruijin Er Lu, Shanghai 200025, People’s Republic of China
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Landian Hu in
Google Scholar
PubMed
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Landian Hu in
Google Scholar
PubMed
Bin Cui
Bin Cui
Shanghai Clinical Center for Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Second Medical University, 197 Ruijin Er Lu, Shanghai 200025, People’s Republic of China
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Bin Cui in
Google Scholar
PubMed
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Bin Cui in
Google Scholar
PubMed
Xi Li
Xi Li
Shanghai Clinical Center for Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Second Medical University, 197 Ruijin Er Lu, Shanghai 200025, People’s Republic of China
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Xi Li in
Google Scholar
PubMed
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Xi Li in
Google Scholar
PubMed
Guang Ning
Guang Ning
Shanghai Clinical Center for Endocrine and Metabolic Diseases, Shanghai Institute of Endocrine and Metabolic Diseases, Ruijin Hospital, Shanghai Second Medical University, 197 Ruijin Er Lu, Shanghai 200025, People’s Republic of China
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Guang Ning in
Google Scholar
PubMed
Health Science Center, Shanghai Institute for Biological Sciences, Chinese Academy of Sciences and Shanghai Second Medical University, Shanghai 200025, People’s Republic of China
Search for other papers by Guang Ning in
Google Scholar
PubMed
defined in the human POMC promoter, domain IV (−376 to −417) had the distinctive property of being active in DMS-79 cells but not in AtT-20 cells ( Picon et al. 1995 ). It was reported that E2 transcription factor binding was required for the activity of
Hong-Wei Wang
Hong-Wei Wang
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Hong-Wei Wang in
Google Scholar
PubMed
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Hong-Wei Wang in
Google Scholar
PubMed
Michelle Muguira
Michelle Muguira
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Michelle Muguira in
Google Scholar
PubMed
Search for other papers by Michelle Muguira in
Google Scholar
PubMed
Wei-Dong Liu
Wei-Dong Liu
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Wei-Dong Liu in
Google Scholar
PubMed
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Wei-Dong Liu in
Google Scholar
PubMed
Tao Zhang
Tao Zhang
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Tao Zhang in
Google Scholar
PubMed
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Tao Zhang in
Google Scholar
PubMed
Chiachen Chen
Chiachen Chen
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Chiachen Chen in
Google Scholar
PubMed
Search for other papers by Chiachen Chen in
Google Scholar
PubMed
Rebecca Aucoin
Rebecca Aucoin
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Rebecca Aucoin in
Google Scholar
PubMed
Search for other papers by Rebecca Aucoin in
Google Scholar
PubMed
Mary B Breslin
Mary B Breslin
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Mary B Breslin in
Google Scholar
PubMed
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Mary B Breslin in
Google Scholar
PubMed
Michael S Lan
Michael S Lan
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Michael S Lan in
Google Scholar
PubMed
The Research Institute for Children, Departments of Pediatrics and Genetics, Children's Hospital, 200 Henry Clay Avenue, Research and Education Building, Room 2211, New Orleans, Louisiana 70118, USA
Search for other papers by Michael S Lan in
Google Scholar
PubMed
Introduction Insulin gene transcription is regulated by both the ubiquitous and the β-cell-specific transcription factors ( Melloul et al . 2002 , Brink 2003 ). In β-cells, multiple regulatory elements in the basal insulin promoter control insulin
Anna Milanesi
Anna Milanesi
Division of Endocrinology, Department of Neurosurgery, Department of Urology, Maxime Dunitz Neurosurgical Institute, Cedars-Sinai Medical Center, 8631 West 3rd Street, Suite 800 East, Los Angeles, California 90048, USA
Search for other papers by Anna Milanesi in
Google Scholar
PubMed
Search for other papers by Anna Milanesi in
Google Scholar
PubMed
Jang-Won Lee
Jang-Won Lee
Division of Endocrinology, Department of Neurosurgery, Department of Urology, Maxime Dunitz Neurosurgical Institute, Cedars-Sinai Medical Center, 8631 West 3rd Street, Suite 800 East, Los Angeles, California 90048, USA
Search for other papers by Jang-Won Lee in
Google Scholar
PubMed
Search for other papers by Jang-Won Lee in
Google Scholar
PubMed
Qijin Xu
Qijin Xu
Division of Endocrinology, Department of Neurosurgery, Department of Urology, Maxime Dunitz Neurosurgical Institute, Cedars-Sinai Medical Center, 8631 West 3rd Street, Suite 800 East, Los Angeles, California 90048, USA
Search for other papers by Qijin Xu in
Google Scholar
PubMed
Search for other papers by Qijin Xu in
Google Scholar
PubMed
Laura Perin
Laura Perin
Division of Endocrinology, Department of Neurosurgery, Department of Urology, Maxime Dunitz Neurosurgical Institute, Cedars-Sinai Medical Center, 8631 West 3rd Street, Suite 800 East, Los Angeles, California 90048, USA
Search for other papers by Laura Perin in
Google Scholar
PubMed
Search for other papers by Laura Perin in
Google Scholar
PubMed
John S Yu
John S Yu
Division of Endocrinology, Department of Neurosurgery, Department of Urology, Maxime Dunitz Neurosurgical Institute, Cedars-Sinai Medical Center, 8631 West 3rd Street, Suite 800 East, Los Angeles, California 90048, USA
Search for other papers by John S Yu in
Google Scholar
PubMed
Search for other papers by John S Yu in
Google Scholar
PubMed
differentiate into pancreatic, exocrine, ductal and endocrine cells in culture. A recent publication showed that suppression of nestin expression in embryonic stem cells by gene silencing reduced endodermal and pancreatic transcription factor expression ( Kim
Jay H Lo
Jay H Lo
Department of Molecular and Cell Biology, University of Connecticut, 91 N. Eagleville Road, U-3125, Storrs, Connecticut 06269, USA
Search for other papers by Jay H Lo in
Google Scholar
PubMed
Search for other papers by Jay H Lo in
Google Scholar
PubMed
Pinwen Peter Chiou
Pinwen Peter Chiou
Department of Molecular and Cell Biology, University of Connecticut, 91 N. Eagleville Road, U-3125, Storrs, Connecticut 06269, USA
Search for other papers by Pinwen Peter Chiou in
Google Scholar
PubMed
Search for other papers by Pinwen Peter Chiou in
Google Scholar
PubMed
C M Lin
C M Lin
Department of Molecular and Cell Biology, University of Connecticut, 91 N. Eagleville Road, U-3125, Storrs, Connecticut 06269, USA
Search for other papers by C M Lin in
Google Scholar
PubMed
Search for other papers by C M Lin in
Google Scholar
PubMed
Thomas T Chen
Thomas T Chen
Department of Molecular and Cell Biology, University of Connecticut, 91 N. Eagleville Road, U-3125, Storrs, Connecticut 06269, USA
Search for other papers by Thomas T Chen in
Google Scholar
PubMed
Search for other papers by Thomas T Chen in
Google Scholar
PubMed
by first binding to the GH receptor (GHR) and then triggering signal transduction cascades resulting in physiological responses ( Argetsinger & Carter-Su 1996 ). Although several transcription factors were believed to be involved in GH signaling
Verónica Torres-Estay
Verónica Torres-Estay
Departamento de Fisiología, Urología, Center for Integrative Medicine and Innovative Sciences, Department of Urology, Pontificia Universidad Católica de Chile, Santiago de Chile, Chile
Search for other papers by Verónica Torres-Estay in
Google Scholar
PubMed
Search for other papers by Verónica Torres-Estay in
Google Scholar
PubMed
Daniela V Carreño
Daniela V Carreño
Departamento de Fisiología, Urología, Center for Integrative Medicine and Innovative Sciences, Department of Urology, Pontificia Universidad Católica de Chile, Santiago de Chile, Chile
Search for other papers by Daniela V Carreño in
Google Scholar
PubMed
Search for other papers by Daniela V Carreño in
Google Scholar
PubMed
Ignacio F San Francisco
Ignacio F San Francisco
Departamento de Fisiología, Urología, Center for Integrative Medicine and Innovative Sciences, Department of Urology, Pontificia Universidad Católica de Chile, Santiago de Chile, Chile
Search for other papers by Ignacio F San Francisco in
Google Scholar
PubMed
Search for other papers by Ignacio F San Francisco in
Google Scholar
PubMed
Paula Sotomayor
Paula Sotomayor
Departamento de Fisiología, Urología, Center for Integrative Medicine and Innovative Sciences, Department of Urology, Pontificia Universidad Católica de Chile, Santiago de Chile, Chile
Search for other papers by Paula Sotomayor in
Google Scholar
PubMed
Search for other papers by Paula Sotomayor in
Google Scholar
PubMed
Alejandro S Godoy
Alejandro S Godoy
Departamento de Fisiología, Urología, Center for Integrative Medicine and Innovative Sciences, Department of Urology, Pontificia Universidad Católica de Chile, Santiago de Chile, Chile
Departamento de Fisiología, Urología, Center for Integrative Medicine and Innovative Sciences, Department of Urology, Pontificia Universidad Católica de Chile, Santiago de Chile, Chile
Search for other papers by Alejandro S Godoy in
Google Scholar
PubMed
Departamento de Fisiología, Urología, Center for Integrative Medicine and Innovative Sciences, Department of Urology, Pontificia Universidad Católica de Chile, Santiago de Chile, Chile
Search for other papers by Alejandro S Godoy in
Google Scholar
PubMed
Gary J Smith
Gary J Smith
Departamento de Fisiología, Urología, Center for Integrative Medicine and Innovative Sciences, Department of Urology, Pontificia Universidad Católica de Chile, Santiago de Chile, Chile
Search for other papers by Gary J Smith in
Google Scholar
PubMed
Search for other papers by Gary J Smith in
Google Scholar
PubMed
( Wen et al . 2013 ) and that the induction of VEGF was mediated by binding of the transcription factor AR and SP1 to the core promoter region of VEGF ( Eisermann et al . 2013 ). Androgen deprivation therapy (ADT), the standard treatment for advanced
