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R P Rhoads
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J W Kim
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M E Van Amburgh
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R A Ehrhardt
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S J Frank
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Y R Boisclair
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despite elevated plasma growth hormone (GH; Block et al. 2001 , Rhoads et al. 2004 ). This fall in plasma IGF-I is thought to reflect decreased GH-stimulated IGF-I transcription in liver as a consequence of GH receptor (GHR) loss ( Radcliff et al

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0068
Volume/Issue:
Volume 195: Issue 1
Free access
Christopher J Delvecchio Department of Biochemistry and Biomedical Sciences, Faculty of Science, McMaster University, 1280 Main Street West, BSB Room 101, L85 4KI, Hamilton, Ontario, Canada

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John P Capone Department of Biochemistry and Biomedical Sciences, Faculty of Science, McMaster University, 1280 Main Street West, BSB Room 101, L85 4KI, Hamilton, Ontario, Canada

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Introduction The liver X receptors (LXRs; α and β subtypes) are members of the nuclear hormone receptor superfamily of ligand-activated transcription factors and are activated by oxidized cholesterol metabolites known as oxysterols ( Janowski et al

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0525
Volume/Issue:
Volume 197: Issue 1
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Paola Venditti Dipartimento delle Scienze Biologiche, Sezione di Fisiologia, Università di Napoli ‘Federico II’, Via Mezzocannone 8, I-80134 Napoli, Italy

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Angela Bari Dipartimento delle Scienze Biologiche, Sezione di Fisiologia, Università di Napoli ‘Federico II’, Via Mezzocannone 8, I-80134 Napoli, Italy

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Lisa Di Stefano Dipartimento delle Scienze Biologiche, Sezione di Fisiologia, Università di Napoli ‘Federico II’, Via Mezzocannone 8, I-80134 Napoli, Italy

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Sergio Di Meo Dipartimento delle Scienze Biologiche, Sezione di Fisiologia, Università di Napoli ‘Federico II’, Via Mezzocannone 8, I-80134 Napoli, Italy

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). Accumulating evidence has demonstrated that in the liver such effects are primarily due to increased tissue content of mitochondrial respiratory chain components ( Brand & Murphy 1987 ), including autoxidizable electron carriers ( Venditti et al . 2003 ). This

Article Type:
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DOI:
https://doi.org/10.1677/JOE-07-0625
Volume/Issue:
Volume 197: Issue 1
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Giuseppe Calamita Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy

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Maria Moreno Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy

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Domenico Ferri Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy

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Elena Silvestri Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy

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Patrizia Roberti Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy

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Luigi Schiavo Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy

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Patrizia Gena Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy

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Maria Svelto Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy

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Fernando Goglia Department of General and Environmental Physiology, Università degli Studi di Bari, Via Amendola 165/A, 70126 Bari, Italy
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy

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synthesis ( Roodyn 1965 , Goglia et al. 1988 ), the turnover ( Gross 1971 ), and the functional capacity of mitochondrial components. Liver mitochondria from hypothyroid rats have a decreased activity of membrane-associated electron transport enzymes and

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-06-0058
Volume/Issue:
Volume 192: Issue 1
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E. TUERKISCHER
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E. WERTHEIMER
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https://doi.org/10.1677/joe.0.0050229
Volume/Issue:
Volume 5: Issue 1
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María Cecilia Ramirez Instituto de Biología y Medicina Experimental-CONICET, Consejo Nacional de Investigaciones Científicas y Técnicas, Vuelta de Obligado 2490, Buenos Aires 1428, Argentina

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Guillermina María Luque Instituto de Biología y Medicina Experimental-CONICET, Consejo Nacional de Investigaciones Científicas y Técnicas, Vuelta de Obligado 2490, Buenos Aires 1428, Argentina

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Ana María Ornstein Instituto de Biología y Medicina Experimental-CONICET, Consejo Nacional de Investigaciones Científicas y Técnicas, Vuelta de Obligado 2490, Buenos Aires 1428, Argentina

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Damasia Becu-Villalobos Instituto de Biología y Medicina Experimental-CONICET, Consejo Nacional de Investigaciones Científicas y Técnicas, Vuelta de Obligado 2490, Buenos Aires 1428, Argentina

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metabolism, thus increasing the susceptibility to develop later disease. In rodents, GH regulates the sexually dimorphic patterns of a large number of liver-expressed genes, including many receptors, signaling molecules, and enzymes of steroid and drug

Article Type:
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https://doi.org/10.1677/JOE-10-0276
Volume/Issue:
Volume 207: Issue 3
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Abdullah Cim
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Greta J Sawyer
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Xiaohong Zhang
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Haibin Su
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Louise Collins
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Peter Jones Department of Hepatology and Transplantation, Diabetes Research Group, Department of Medical and Molecular Genetics, InvivoGen, Institut für Zellbiologie, King's College London School of Medicine, James Black Centre, 125 Coldharbour Lane, London SE5 9NU, UK

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Michael Antoniou Department of Hepatology and Transplantation, Diabetes Research Group, Department of Medical and Molecular Genetics, InvivoGen, Institut für Zellbiologie, King's College London School of Medicine, James Black Centre, 125 Coldharbour Lane, London SE5 9NU, UK

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Jean-Paul Reynes Department of Hepatology and Transplantation, Diabetes Research Group, Department of Medical and Molecular Genetics, InvivoGen, Institut für Zellbiologie, King's College London School of Medicine, James Black Centre, 125 Coldharbour Lane, London SE5 9NU, UK

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Hans-Joachim Lipps Department of Hepatology and Transplantation, Diabetes Research Group, Department of Medical and Molecular Genetics, InvivoGen, Institut für Zellbiologie, King's College London School of Medicine, James Black Centre, 125 Coldharbour Lane, London SE5 9NU, UK

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John W Fabre
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transducing hepatocytes towards a pancreas phenotype in vivo . Ferber et al . (2000) gave standard E1/E4-deleted adenoviral vectors encoding rat Pdx1 i.v. to mice and found insulin-producing endocrine cells in the liver. These cells produced multiple

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0033
Volume/Issue:
Volume 214: Issue 3
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Antonia Giacco Department of Science and Technology, University of Sannio, Benevento, Italy

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Teresa Peluso Department of Science and Technology, University of Sannio, Benevento, Italy

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Federica Cioffi Department of Science and Technology, University of Sannio, Benevento, Italy

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Stefania Iervolino Department of Science and Technology, University of Sannio, Benevento, Italy

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Giovanna Mercurio Department of Science and Technology, University of Sannio, Benevento, Italy

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Luca Roberto Biogem Scarl, Istituto di Ricerche Genetiche ‘Gaetano Salvatore',Ariano Irpino, Italy

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Carla Reale Biogem Scarl, Istituto di Ricerche Genetiche ‘Gaetano Salvatore',Ariano Irpino, Italy

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Marco Colella Department of Science and Technology, University of Sannio, Benevento, Italy
Biogem Scarl, Istituto di Ricerche Genetiche ‘Gaetano Salvatore',Ariano Irpino, Italy

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Mario De Felice Institute of Experimental Endocrinology and Oncology (IEOS), CNR, Naples, Italy
Department of Molecular Medicine and Medical Biotechnologies, University of Naples Federico II, Naples, Italy

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Maria Moreno Department of Science and Technology, University of Sannio, Benevento, Italy

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Concetta Ambrosino Department of Science and Technology, University of Sannio, Benevento, Italy
Biogem Scarl, Istituto di Ricerche Genetiche ‘Gaetano Salvatore',Ariano Irpino, Italy
Institute of Experimental Endocrinology and Oncology (IEOS), CNR, Naples, Italy

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Elena Silvestri Department of Science and Technology, University of Sannio, Benevento, Italy

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Introduction In humans and rodents, alterations in thyroid function have been associated with the pathogenesis of liver diseases and insulin resistance (IR) ( Iwen et al. 2013 , Giacco et al. 2019 ), predisposing, in turn, to diabetes as

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-22-0053
Volume/Issue:
Volume 253: Issue 3
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Kasper Faarkrog Høyer Department of Clinical Medicine, The MR Research Center, Aarhus University Hospital, Aarhus, Denmark
Novo Nordisk Foundation Center for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark

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Christoffer Laustsen Department of Clinical Medicine, The MR Research Center, Aarhus University Hospital, Aarhus, Denmark

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Steffen Ringgaard Department of Clinical Medicine, The MR Research Center, Aarhus University Hospital, Aarhus, Denmark

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Haiyun Qi Department of Clinical Medicine, The MR Research Center, Aarhus University Hospital, Aarhus, Denmark

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Christian Østergaard Mariager Department of Clinical Medicine, The MR Research Center, Aarhus University Hospital, Aarhus, Denmark

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Thomas Svava Nielsen Novo Nordisk Foundation Center for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark

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Ulrik Kræmer Sundekilde Department of Food Science, Aarhus University, Aarslev, Denmark

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Jonas T Treebak Novo Nordisk Foundation Center for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark

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Niels Jessen Department of Biomedicine, Research Laboratory for Biochemical Pathology, Aarhus University, Aarhus, Denmark
Department of Clinical Pharmacology, Aarhus University Hospital, Aarhus, Denmark
Steno Diabetes Center Aarhus, Aarhus University Hospital, Aarhus, Denmark

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Hans Stødkilde-Jørgensen Department of Clinical Medicine, The MR Research Center, Aarhus University Hospital, Aarhus, Denmark

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Introduction The liver plays a crucial role in the regulation of whole-body glucose homeostasis and ensures a sufficient supply of substrates irrespective of food intake. During fasting, it is the primary source of endogenous glucose

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-19-0159
Volume/Issue:
Volume 242: Issue 3
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Paula J Brunton Division of Neurobiology, Endocrinology Unit, Laboratory of Neuroendocrinology, The Roslin Institute and R(D)SVS, University of Edinburgh, Easter Bush Campus, Midlothian, EH25 9RG, UK

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Katie M Sullivan Division of Neurobiology, Endocrinology Unit, Laboratory of Neuroendocrinology, The Roslin Institute and R(D)SVS, University of Edinburgh, Easter Bush Campus, Midlothian, EH25 9RG, UK

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David Kerrigan Division of Neurobiology, Endocrinology Unit, Laboratory of Neuroendocrinology, The Roslin Institute and R(D)SVS, University of Edinburgh, Easter Bush Campus, Midlothian, EH25 9RG, UK

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John A Russell Division of Neurobiology, Endocrinology Unit, Laboratory of Neuroendocrinology, The Roslin Institute and R(D)SVS, University of Edinburgh, Easter Bush Campus, Midlothian, EH25 9RG, UK

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Jonathan R Seckl Division of Neurobiology, Endocrinology Unit, Laboratory of Neuroendocrinology, The Roslin Institute and R(D)SVS, University of Edinburgh, Easter Bush Campus, Midlothian, EH25 9RG, UK

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Amanda J Drake Division of Neurobiology, Endocrinology Unit, Laboratory of Neuroendocrinology, The Roslin Institute and R(D)SVS, University of Edinburgh, Easter Bush Campus, Midlothian, EH25 9RG, UK

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expression of the glucocorticoid receptor (GR) in both liver and adipose tissue ( Nyirenda et al . 1998 , Cleasby et al . 2003 ) and with increased hepatic expression of genes known to be important in regulating gluconeogenesis, such as phosphoenolpyruvate

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0540
Volume/Issue:
Volume 217: Issue 2
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