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Faculté de Médécine, Centre Commun de Cytométrie en Flux, Université Jean Monnet, 42023 St Etienne, France
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Faculté de Médécine, Centre Commun de Cytométrie en Flux, Université Jean Monnet, 42023 St Etienne, France
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Faculté de Médécine, Centre Commun de Cytométrie en Flux, Université Jean Monnet, 42023 St Etienne, France
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Faculté de Médécine, Centre Commun de Cytométrie en Flux, Université Jean Monnet, 42023 St Etienne, France
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potential importance of the GDNF/RET pathway in male reproductive physiopathology, we determined the localization of GDNF and its two receptors, GFR1α and RET, during postnatal development of the rat testis. We determined mRNA and protein levels in isolated
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disappears upon estradiol administration ( Duckles et al . 2006 ). Thus, we explored functional alterations in rat heart mitochondria at one, two and three months after oophorectomy. Oxygen consumption measurements were performed to analyze the progressive
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-1β in rat pancreatic islet cells. In addition, we explored the expression level of potentially related apoptotic and anti-apoptotic molecules in rat islet cells treated with these various cell death inducers and different glucose concentrations. Our
Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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Department of Biological and Environmental Sciences, University of Sannio, Via Port’Arsa 11, 82100 Benevento, Italy
Department of Zoology, University of Bari, Bari, Italy
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a ), mitochondria possess facilitated pathways for water diffusion other than the AQP8 water channels ( Calamita et al. 2006 ). In rat, liver AQP8 has also been suggested to play a role in amino acid metabolism and detoxification of ammonia by
Laboratory of Hormones and Receptors, Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, 32 Vavilova Str., 119991, Moscow, Russian Federation
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Laboratory of Hormones and Receptors, Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, 32 Vavilova Str., 119991, Moscow, Russian Federation
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Laboratory of Hormones and Receptors, Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, 32 Vavilova Str., 119991, Moscow, Russian Federation
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Laboratory of Hormones and Receptors, Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, 32 Vavilova Str., 119991, Moscow, Russian Federation
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Laboratory of Hormones and Receptors, Engelhardt Institute of Molecular Biology, Russian Academy of Sciences, 32 Vavilova Str., 119991, Moscow, Russian Federation
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Introduction Several prolactin receptor (PrlR) isoforms have been identified in mammals. In rat, they have been shown to be produced via alternative splicing of PrlR pre-mRNA: the formation of long and short isoforms result from
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Rheoscience A/S, Faculty of Pharmaceutical Sciences, Neurology Service, Department of Neurology and Neurosciencies, NovoNordisk A/S, Rødovre 2610, Copenhagen, Denmark
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) out-bred rat models ( Levin et al . 1997 ). While some short-term studies have been performed to characterize the development of obesity and to address the presence of some of the associated metabolic disturbances ( Levin et al . 1997 , Levin
Department of Physiology and Biophysics, Faculty of Medicine and Health sciences, University of Sherbrooke, Sherbrooke, 3001, 12th Avenue North, Quebec, Canada J1H 5N4
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Department of Physiology and Biophysics, Faculty of Medicine and Health sciences, University of Sherbrooke, Sherbrooke, 3001, 12th Avenue North, Quebec, Canada J1H 5N4
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Department of Physiology and Biophysics, Faculty of Medicine and Health sciences, University of Sherbrooke, Sherbrooke, 3001, 12th Avenue North, Quebec, Canada J1H 5N4
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Department of Physiology and Biophysics, Faculty of Medicine and Health sciences, University of Sherbrooke, Sherbrooke, 3001, 12th Avenue North, Quebec, Canada J1H 5N4
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Introduction The adult rat adrenal cortex is composed of three concentric layers, the zona glomerulosa, the zona fasciculata and the zona reticularis, all of which present different morphological and functional properties. The zona
Laboratory of Cellular Biology, Department of Biology, Universidade Federal de Juiz de Fora, Juiz de Fora, Minas Gerais, Brazil
Laboratory of Structural Biology and Reproduction, Department of Morphology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil
Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Endocrine Neurobiology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest H-1083, Hungary
Division of Endocrinology, Diabetes, and Metabolism, Tufts-New England Medical Center, Boston, Massachusetts, USA
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Laboratory of Cellular Biology, Department of Biology, Universidade Federal de Juiz de Fora, Juiz de Fora, Minas Gerais, Brazil
Laboratory of Structural Biology and Reproduction, Department of Morphology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil
Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Endocrine Neurobiology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest H-1083, Hungary
Division of Endocrinology, Diabetes, and Metabolism, Tufts-New England Medical Center, Boston, Massachusetts, USA
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Laboratory of Cellular Biology, Department of Biology, Universidade Federal de Juiz de Fora, Juiz de Fora, Minas Gerais, Brazil
Laboratory of Structural Biology and Reproduction, Department of Morphology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil
Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Endocrine Neurobiology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest H-1083, Hungary
Division of Endocrinology, Diabetes, and Metabolism, Tufts-New England Medical Center, Boston, Massachusetts, USA
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Laboratory of Cellular Biology, Department of Biology, Universidade Federal de Juiz de Fora, Juiz de Fora, Minas Gerais, Brazil
Laboratory of Structural Biology and Reproduction, Department of Morphology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil
Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Endocrine Neurobiology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest H-1083, Hungary
Division of Endocrinology, Diabetes, and Metabolism, Tufts-New England Medical Center, Boston, Massachusetts, USA
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Laboratory of Cellular Biology, Department of Biology, Universidade Federal de Juiz de Fora, Juiz de Fora, Minas Gerais, Brazil
Laboratory of Structural Biology and Reproduction, Department of Morphology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil
Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Endocrine Neurobiology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest H-1083, Hungary
Division of Endocrinology, Diabetes, and Metabolism, Tufts-New England Medical Center, Boston, Massachusetts, USA
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Laboratory of Cellular Biology, Department of Biology, Universidade Federal de Juiz de Fora, Juiz de Fora, Minas Gerais, Brazil
Laboratory of Structural Biology and Reproduction, Department of Morphology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil
Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Endocrine Neurobiology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest H-1083, Hungary
Division of Endocrinology, Diabetes, and Metabolism, Tufts-New England Medical Center, Boston, Massachusetts, USA
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Laboratory of Cellular Biology, Department of Biology, Universidade Federal de Juiz de Fora, Juiz de Fora, Minas Gerais, Brazil
Laboratory of Structural Biology and Reproduction, Department of Morphology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil
Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Endocrine Neurobiology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest H-1083, Hungary
Division of Endocrinology, Diabetes, and Metabolism, Tufts-New England Medical Center, Boston, Massachusetts, USA
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Laboratory of Cellular Biology, Department of Biology, Universidade Federal de Juiz de Fora, Juiz de Fora, Minas Gerais, Brazil
Laboratory of Structural Biology and Reproduction, Department of Morphology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil
Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Endocrine Neurobiology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest H-1083, Hungary
Division of Endocrinology, Diabetes, and Metabolism, Tufts-New England Medical Center, Boston, Massachusetts, USA
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Laboratory of Cellular Biology, Department of Biology, Universidade Federal de Juiz de Fora, Juiz de Fora, Minas Gerais, Brazil
Laboratory of Structural Biology and Reproduction, Department of Morphology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil
Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Endocrine Neurobiology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest H-1083, Hungary
Division of Endocrinology, Diabetes, and Metabolism, Tufts-New England Medical Center, Boston, Massachusetts, USA
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Laboratory of Cellular Biology, Department of Biology, Universidade Federal de Juiz de Fora, Juiz de Fora, Minas Gerais, Brazil
Laboratory of Structural Biology and Reproduction, Department of Morphology, Universidade Federal de Minas Gerais, Belo Horizonte, Brazil
Division of Endocrinology, Diabetes and Hypertension, Brigham and Women’s Hospital and Harvard Medical School, Boston, Massachusetts, USA
Department of Endocrine Neurobiology, Institute of Experimental Medicine, Hungarian Academy of Sciences, Budapest H-1083, Hungary
Division of Endocrinology, Diabetes, and Metabolism, Tufts-New England Medical Center, Boston, Massachusetts, USA
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testis was considered for years to be a thyroid hormone unresponsive organ, a concept based on data showing minimal levels of testicular thyroid hormone receptors (TRs) and lack of thyroid hormone effects on oxygen consumption in adult rat testes
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-up growth is still unclear. In the present work, we characterized an in vitro model that mimics the clinically observed catch-up growth after cessation of glucocorticoid exposure. For this purpose, we optimized a well-known model of fetal rat metatarsal
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, Perry & Ridgway 2005 ), and it is vital to lipid metabolism in eukaryotic cells. SMS2 and its enzymatic activity have been extensively investigated in other systems, such as in chick liver ( Lyman et al. 1976 ), rat colon ( Dudeja et al. 1986 ), and