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James C Needell Barbara Davis Center for Childhood Diabetes, University of Colorado Denver, Aurora, Colorado, USA

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Madalyn N Brown Barbara Davis Center for Childhood Diabetes, University of Colorado Denver, Aurora, Colorado, USA

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Danny Zipris Barbara Davis Center for Childhood Diabetes, University of Colorado Denver, Aurora, Colorado, USA

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-induced adipose tissue inflammation in the development of T1D in the LEW1.WR1 rat. Our data demonstrate for the first time that infection with a diabetogenic virus leads to CD68 + macrophage infiltration into visceral but not subcutaneous adipose tissue observed

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0131
Volume/Issue:
Volume 238: Issue 1
Free access
Rebecca J Ainslie Institute for Regeneration and Repair, the University of Edinburgh, Edinburgh BioQuarter, Edinburgh, United Kingdom

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Ioannis Simitsidellis Institute for Regeneration and Repair, the University of Edinburgh, Edinburgh BioQuarter, Edinburgh, United Kingdom

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Phoebe M Kirkwood Institute for Regeneration and Repair, the University of Edinburgh, Edinburgh BioQuarter, Edinburgh, United Kingdom

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Douglas A Gibson Institute for Regeneration and Repair, the University of Edinburgh, Edinburgh BioQuarter, Edinburgh, United Kingdom

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in human alveolar macrophages ( Milewich et al. 1983 ) and synovial macrophages ( Cutolo et al. 1996 ), as well as murine T lymphocytes ( Samy et al. 2001 ). Mechanisms of androgen signalling Classically, androgen signalling is mediated

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-23-0398
Volume/Issue:
Volume 261: Issue 3
Open access
A Albalat
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C Liarte
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S MacKenzie
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L Tort
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J V Planas
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I Navarro
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& Goetz 2001 , Laing et al. 2001 ). Interestingly, TNFα mRNA expression has been reported in primary trout monocytes ( Zou et al. 2002 , MacKenzie et al. 2003 ) and in in vitro differentiated macrophages after stimulation with lipopolysaccharide

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.05940
Volume/Issue:
Volume 184: Issue 3
Free access
Rita E Roberts Center for Wound Healing and Tissue Regeneration, University of Illinois at Chicago, Chicago, Illinois, USA
Department of Kinesiology and Nutrition, University of Illinois at Chicago, Chicago, Illinois, USA
Research and Development Division, Jesse Brown Veterans Affairs Medical Center, Chicago, Illinois, USA

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Jacqueline Cavalcante-Silva Center for Wound Healing and Tissue Regeneration, University of Illinois at Chicago, Chicago, Illinois, USA
Department of Kinesiology and Nutrition, University of Illinois at Chicago, Chicago, Illinois, USA
Research and Development Division, Jesse Brown Veterans Affairs Medical Center, Chicago, Illinois, USA

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Rhonda D Kineman Research and Development Division, Jesse Brown Veterans Affairs Medical Center, Chicago, Illinois, USA
Section of Endocrinology, Diabetes, and Metabolism, Department of Medicine, University of Illinois at Chicago, Chicago, Illinois, USA

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Timothy J Koh Center for Wound Healing and Tissue Regeneration, University of Illinois at Chicago, Chicago, Illinois, USA
Department of Kinesiology and Nutrition, University of Illinois at Chicago, Chicago, Illinois, USA
Research and Development Division, Jesse Brown Veterans Affairs Medical Center, Chicago, Illinois, USA

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primary source of wound IGF-1 ( Wagner et al. 2003 ). However, IGF-1 is also expressed by keratinocytes, fibroblasts and macrophages in skin wounds ( Gartner et al. 1992 , Brown et al. 1997 , Todorovic et al. 2008 ). Thus, the relative

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-21-0298
Volume/Issue:
Volume 252: Issue 1
Restricted access
Rene F Chun Department of Orthopaedic Surgery, Molecular Biology Institute, David Geffen School of Medicine, UCLA, 615 Charles E Young Drive South, Los Angeles, California 90095, USA

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John S Adams Department of Orthopaedic Surgery, Molecular Biology Institute, David Geffen School of Medicine, UCLA, 615 Charles E Young Drive South, Los Angeles, California 90095, USA
Department of Orthopaedic Surgery, Molecular Biology Institute, David Geffen School of Medicine, UCLA, 615 Charles E Young Drive South, Los Angeles, California 90095, USA

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Martin Hewison Department of Orthopaedic Surgery, Molecular Biology Institute, David Geffen School of Medicine, UCLA, 615 Charles E Young Drive South, Los Angeles, California 90095, USA
Department of Orthopaedic Surgery, Molecular Biology Institute, David Geffen School of Medicine, UCLA, 615 Charles E Young Drive South, Los Angeles, California 90095, USA

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involved in mediating innate immune responses to Mycobacterium tuberculosis ( Liu et al . 2006 ). The DNA array analysis of macrophages activated with an M tuberculosis lipoprotein highlighted the induction of mRNA for both the vitamin D receptor (VDR

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0170
Volume/Issue:
Volume 198: Issue 2
Free access
Eun-Kyung Choi
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Woon-Ki Kim
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Ok-Joo Sul
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Yun-Kyung Park
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Eun-Sook Kim Department of Biological Sciences, Department of Endocrinology, Department of Pathology, Department of Food Science and Nutrition, University of Ulsan, Ulsan 680-749, Korea

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Jae-Hee Suh Department of Biological Sciences, Department of Endocrinology, Department of Pathology, Department of Food Science and Nutrition, University of Ulsan, Ulsan 680-749, Korea

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Rina Yu Department of Biological Sciences, Department of Endocrinology, Department of Pathology, Department of Food Science and Nutrition, University of Ulsan, Ulsan 680-749, Korea

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Hye-Seon Choi
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observed in OVX. Increased fat leads to the accumulation of adipose tissue macrophage (ATM), which plays a critical role in chronic inflammation and development of insulin resistance (IR) ( Shoelson et al . 2007 ). Exacerbation of insulin sensitivity is

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-13-0341
Volume/Issue:
Volume 220: Issue 1
Free access
Corinne A Schuyler Ralph H. Johnson Veterans Affairs Medical Center, Division of Endocrinology, Charleston, South Carolina 29401, USA

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Nga N Ta Ralph H. Johnson Veterans Affairs Medical Center, Division of Endocrinology, Charleston, South Carolina 29401, USA

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Yanchun Li Ralph H. Johnson Veterans Affairs Medical Center, Division of Endocrinology, Charleston, South Carolina 29401, USA

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Maria F Lopes-Virella Ralph H. Johnson Veterans Affairs Medical Center, Division of Endocrinology, Charleston, South Carolina 29401, USA
Ralph H. Johnson Veterans Affairs Medical Center, Division of Endocrinology, Charleston, South Carolina 29401, USA

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Yan Huang Ralph H. Johnson Veterans Affairs Medical Center, Division of Endocrinology, Charleston, South Carolina 29401, USA
Ralph H. Johnson Veterans Affairs Medical Center, Division of Endocrinology, Charleston, South Carolina 29401, USA

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that are prone to rupture consist of a larger intimal lesion and an increased number of macrophages. Analysis of gene expression in atherosclerotic plaques showed that when compared with stable plaques, vulnerable plaques have higher expression of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-10-0420
Volume/Issue:
Volume 210: Issue 1
Free access
Susanne Granholm Department of Oral Cell Biology, Umeå University, S-901 87 Umeå, Sweden

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Pernilla Lundberg Department of Oral Cell Biology, Umeå University, S-901 87 Umeå, Sweden

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Ulf H Lerner Department of Oral Cell Biology, Umeå University, S-901 87 Umeå, Sweden

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haematopoietic origin and are most closely related to the monocyte/macrophage precursor cells ( Takahashi et al . 2002 ). Activation of the c-Fms receptor on these cells by macrophage colony-stimulating factor (M-CSF), released by stromal cells, or osteoblasts

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0338
Volume/Issue:
Volume 195: Issue 3
Free access
Li Juan He Division of Nephrology, Nanfang Hospital, Southern Medical University, 1838 North Guangzhou Avenue, Guangzhou 510515, People's Republic of China

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Min Liang Division of Nephrology, Nanfang Hospital, Southern Medical University, 1838 North Guangzhou Avenue, Guangzhou 510515, People's Republic of China

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Fan Fan Hou Division of Nephrology, Nanfang Hospital, Southern Medical University, 1838 North Guangzhou Avenue, Guangzhou 510515, People's Republic of China

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Zhi Jian Guo Division of Nephrology, Nanfang Hospital, Southern Medical University, 1838 North Guangzhou Avenue, Guangzhou 510515, People's Republic of China

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Di Xie Division of Nephrology, Nanfang Hospital, Southern Medical University, 1838 North Guangzhou Avenue, Guangzhou 510515, People's Republic of China

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Xun Zhang Division of Nephrology, Nanfang Hospital, Southern Medical University, 1838 North Guangzhou Avenue, Guangzhou 510515, People's Republic of China

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the development of diabetic nephropathy ( Schrijvers et al . 2004 ). Increasing evidence shows that aberrant inflammatory responses, such as macrophage infiltration and overexpression of proinflammatory cytokines, are present in the diabetic kidney

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0481
Volume/Issue:
Volume 200: Issue 3
Free access
Yingxin Xian Department of Endocrinology and Metabolism, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China
Guangdong Provincial Key Laboratory of Diabetology, Guangzhou, China

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Zonglan Chen Department of Endocrinology and Metabolism, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China
Guangdong Provincial Key Laboratory of Diabetology, Guangzhou, China

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Hongrong Deng Department of Endocrinology and Metabolism, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China
Guangdong Provincial Key Laboratory of Diabetology, Guangzhou, China

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Mengyin Cai Department of Endocrinology and Metabolism, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China
Guangdong Provincial Key Laboratory of Diabetology, Guangzhou, China

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Hua Liang Department of Endocrinology and Metabolism, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China
Guangdong Provincial Key Laboratory of Diabetology, Guangzhou, China

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Wen Xu Department of Endocrinology and Metabolism, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China
Guangdong Provincial Key Laboratory of Diabetology, Guangzhou, China

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Jianping Weng Department of Endocrinology and Metabolism, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China
Guangdong Provincial Key Laboratory of Diabetology, Guangzhou, China

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Fen Xu Department of Endocrinology and Metabolism, The Third Affiliated Hospital of Sun Yat-sen University, Guangzhou, China
Guangdong Provincial Key Laboratory of Diabetology, Guangzhou, China

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tissue from humans with obesity and obese mice models is infiltrated with increased numbers of macrophages provides a major mechanistic advance into understanding obesity-associated inflammation ( Olefsky & Glass 2010 ). These adipose tissue macrophages

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0639
Volume/Issue:
Volume 242: Issue 2
Free access