mouse female fertility ( Mulac-Jericevic et al. 2000 ). Cyclic changes of P and estradiol control homeostasis of the oviduct epithelium as well as formation and beat frequency of the cilia ( Brenner 1969 , Wessel et al. 2004 ), although
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Stefan Cuoni Teilmann, Christian Alexandro Clement, Jørgen Thorup, Anne Grete Byskov, and Søren Tvorup Christensen
Si-Zeng Chen and Zheng-Guo Qiu
.p. administration of GH (0.5 mg/kg per day), INS (0.5 μg/kg per day), and/or IND (0.5 mg/kg per day) began on day 9 (at the onset of cachexia) and continued daily, according to mouse weight and the dosage table. Ten mice from each group were killed on day 16, with
Hong-Hui Wang, Qian Cui, Teng Zhang, Lei Guo, Ming-Zhe Dong, Yi Hou, Zhen-Bo Wang, Wei Shen, Jun-Yu Ma, and Qing-Yuan Sun
female reproductive process. The levels of these hormones change regularly in an estrous cycle, which lasts for different lengths of time in different species (for humans, 29.1 days of a menstrual cycle and mouse 4–5 days of an estrous cycle) ( Chiazze
Ioannis Simitsidellis, Arantza Esnal-Zuffiaure, Olympia Kelepouri, Elisabeth O’Flaherty, Douglas A Gibson, and Philippa T K Saunders
mouse model to test the impact of two SARMs, GTx-007 (also known as Andarine, S4: https://pubchem.ncbi.nlm.nih.gov/compound/9824562 ) and GTx-024 (Enobosarm, Ostarine: https://pubchem.ncbi.nlm.nih.gov/compound/11326715 ) on uterine tissue and to
Ikuko Ueki, Norio Abiru, Kentaro Kawagoe, and Yuji Nagayama
induced in susceptible mouse strains (e.g. BALB/c) by repetitive immunizations with recombinant adenovirus expressing the human full-length TSHR or its A-subunit ( Nagayama et al . 2002 , Chen et al . 2003 ). In these models, an in vitro T cell recall
Nele Cielen, Nele Heulens, Karen Maes, Geert Carmeliet, Chantal Mathieu, Wim Janssens, and Ghislaine Gayan-Ramirez
deficiency per se may play a role in skeletal muscle dysfunction in COPD, we examined the time-course effect of chronic vitamin D deficiency on skeletal muscle function in a smoking mouse model. We used a vitamin D-deficient mouse model with normalized
Jenny D Y Chow, Margaret E E Jones, Katja Prelle, Evan R Simpson, and Wah Chin Boon
estrogens from androgens ( Gruber et al . 2002 ), and the transcriptional actions of estrogens are mediated through ERα and ERβ ( Matthews & Gustafsson 2003 ). Mouse models of estrogen deficiency develop characteristics of MetS, for example, the aromatase
A Edlund, M Barghouth, M Hühn, M Abels, J S E Esguerra, I G Mollet, E Svedin, A Wendt, E Renström, E Zhang, N Wierup, B J Scholte, M Flodström-Tullberg, and L Eliasson
et al. 2016 ). In both human and mouse, F508del-CFTR is misprocessed in the endoplasmatic reticulum and the channel is subsequently degraded. However, a small portion of F508del-CFTR escapes degradation and reaches the plasma membrane where it has
K A Berghorn, P A Clark, B Encarnacion, C J DeRegis, J K Folger, M I Morasso, M J Soares, M W Wolfe, and M S Roberson
embryonic day (E) 9.5 leading ultimately to fetal death apparently due to abnormal placental/vascular morphogenesis ( Morasso et al. 1999 ). The establishment of the mouse placenta requires a large number of transcriptional regulators (reviewed by Cross
Hsien-Yeh Chou, Namasivayam Elangovan, Ying-Chu Lee, Hsiou Hsia Lin, and Sin-Tak Chu
). Anti-mouse IgG-horseradish peroxidase conjugate prepared from goats and anti-mouse IgG FITC-conjugated antibody were obtained from Sigma. The 24p3 protein was purified from diethyl-stilbestrol (DES)-stimulated female mice as has been previously
