C/EBPβ specifically in the lung epithelium ( Cebpb ΔLE mice) challenged with aerosolized LPS display a blunted recruitment of neutrophils to the airways, together with blunted induction of several inflammatory mediators, including the neutrophil
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Abraham B Roos and Magnus Nord
Eduardo Esteban-Zubero, Francisco Agustín García-Gil, Laura López-Pingarrón, Moisés Alejandro Alatorre-Jiménez, Pablo Iñigo-Gil, Dun-Xian Tan, José Joaquín García, and Russel J Reiter
expression of proinflammatory cytokines. 4-HNE is a chemoattractant for neutrophils ( Jaeschke 1996 ). Figure 1 Liver IRI physiopathology. Cold ischemia induces cellular ATP depletion, which results in the loss of electrolyte homeostasis and increases
Aarti D Rohira, David M Lonard, and Bert W O’Malley
macrophages and neutrophils are part of the innate immune system and serve as the first line of defense for eliminating pathogens ( Lingen 2001 ). They set up inflammatory signaling programs in response to an infection and eliminate pathogens through
A Feraco, A Armani, R Urbanet, A Nguyen Dinh Cat, V Marzolla, F Jaisser, and M Caprio
control-MR mice were observed in AT expression of adipokines such as Adiponectin , neutrophil gelatinase-associated lipocalin ( Ngal ) and Prostaglandin D2 Synthase ( Ptgds ), which has been recently identified as a specific MR target in AT ( Urbanet
You-Hua Xu, Chen-Lin Gao, Heng-Li Guo, Wen-Qian Zhang, Wei Huang, Shan-Shan Tang, Wen-Jun Gan, Yong Xu, Hua Zhou, and Quan Zhu
) or metformin (Met) for 5 weeks; at the end of the experiment, blood sample was collected, (A) WBCs, and inflammatory cell count including (B) neutrophils (NE), (C) abnormal lymphocytes (ALY) and (D) monocytes (MONO) were analyzed. ‘Normal’ indicates
Ana Tiganescu, Melanie Hupe, Yoshikazu Uchida, Theodora Mauro, Peter M Elias, and Walter M Holleran
regulate mouse macrophage phagocytosis ( Gilmour et al . 2006 ), human neutrophil survival ( Kardon et al . 2008 ), and murine mast cell degranulation ( Coutinho et al . 2013 ), although the beneficial/adverse effects of 11β-HSD1 blockade in various
Siobhan M Donnelly, Bao Tran Nguyen, Scott Rhyne, Jordan Estes, Subrina Jesmin, and Chishimba Nathan Mowa
neutrophils, lymphocytes, eosinophils, and monocytes, to the basolateral border of the invaded epithelial cells. Neutrophils will then migrate to the apical end of the epithelia and lumen, close or proximal to the invading bacteria, via the inter
Nele Cielen, Nele Heulens, Karen Maes, Geert Carmeliet, Chantal Mathieu, Wim Janssens, and Ghislaine Gayan-Ramirez
counts. Three hundred cells per mouse were counted to determine the number of neutrophils. Histopathology of the lungs The left lung was fixed in 6% v/v paraformaldehyde at a constant hydrostatic pressure of 25 cm H 2 O for 24 h and embedded in
Eun-Kyung Choi, Woon-Ki Kim, Ok-Joo Sul, Yun-Kyung Park, Eun-Sook Kim, Jae-Hee Suh, Rina Yu, and Hye-Seon Choi
CD11c cells as well as CD11b cells, which were significantly elevated by OVX. We cannot exclude the contribution of blood neutrophils to the elevated level of CD11bCD11c upon OVX, because blood neutrophils also express CD11bCD11c. However, their
Jon G Mabley, Pal Pacher, Kanneganti G K Murthy, William Williams, Garry J Southan, Andrew L Salzman, and Csaba Szabo
induced by multiple low doses of streptozotocin . Journal of Immunology 178 4623 – 4631 . Marton A Pacher P Murthy KG Nemeth ZH Hasko G Szabo C 2001 Anti-inflammatory effects of inosine in human monocytes, neutrophils and epithelial
