Search Results
Search for other papers by M Fraenkel in
Google Scholar
PubMed
Search for other papers by J Caloyeras in
Google Scholar
PubMed
Search for other papers by S-G Ren in
Google Scholar
PubMed
Search for other papers by S Melmed in
Google Scholar
PubMed
+/+ control animals is likely secondary to the pttg -null lipodystrophy, which is caused primarily by the low insulin levels. Several studies in humans and rodents have shown sexual dimorphism in plasma levels of adiponectin, with males having lower levels
Search for other papers by Obaro Evuarherhe in
Google Scholar
PubMed
Search for other papers by James Leggett in
Google Scholar
PubMed
Search for other papers by Eleanor Waite in
Google Scholar
PubMed
Search for other papers by Yvonne Kershaw in
Google Scholar
PubMed
Search for other papers by Stafford Lightman in
Google Scholar
PubMed
number: RJ4534). References Atkinson HC Waddell BJ 1997 Circadian variation in basal plasma corticosterone and adrenocorticotropin in the rat: sexual dimorphism and changes across the estrous cycle . Endocrinology 138 3842 – 3848 . Azooz OG
Search for other papers by Sophie A Clarke in
Google Scholar
PubMed
Search for other papers by Waljit S Dhillo in
Google Scholar
PubMed
. Although the effects of kisspeptin in ageing populations remain largely unexplored, the kisspeptin neuron population in the infundibular nucleus clearly increases with time. Interestingly, however, in the same manner that sexual dimorphism exists with
Department of Pharmacological and Biomolecular Sciences, University of Milan, Milan, Italy
Search for other papers by Sara Della Torre in
Google Scholar
PubMed
Department of Pharmacological and Biomolecular Sciences, University of Milan, Milan, Italy
Search for other papers by Gianpaolo Rando in
Google Scholar
PubMed
Department of Pharmacological and Biomolecular Sciences, University of Milan, Milan, Italy
Search for other papers by Clara Meda in
Google Scholar
PubMed
Search for other papers by Paolo Ciana in
Google Scholar
PubMed
Search for other papers by Luisa Ottobrini in
Google Scholar
PubMed
Department of Pharmacological and Biomolecular Sciences, University of Milan, Milan, Italy
Search for other papers by Adriana Maggi in
Google Scholar
PubMed
-temporal expression of ERα and ERβ was reported to contribute to organize sex differences that are not associated with reproduction, such as the stress response and cognition ( Mogi et al. 2015 ). It is conceivable that the sexual dimorphism observed in
Search for other papers by Rajat Kumar Das in
Google Scholar
PubMed
Search for other papers by Sarmistha Banerjee in
Google Scholar
PubMed
Search for other papers by Bernard H Shapiro in
Google Scholar
PubMed
hormone levels per se that are responsible for phenotypic sexual dimorphisms ranging from growth patterns to expression levels of hepatic enzymes ( Jansson et al . 1985 , Shapiro et al . 1995 ). In the rat, CYP responses to GH regulation are nearly as
Search for other papers by Yuefei Huang in
Google Scholar
PubMed
Search for other papers by Pei Yee Ting in
Google Scholar
PubMed
Search for other papers by Tham M Yao in
Google Scholar
PubMed
Search for other papers by Tsuyoshi Homma in
Google Scholar
PubMed
Search for other papers by Danielle Brooks in
Google Scholar
PubMed
Search for other papers by Isis Katayama Rangel in
Google Scholar
PubMed
Search for other papers by Gail K Adler in
Google Scholar
PubMed
Search for other papers by Jose R Romero in
Google Scholar
PubMed
Search for other papers by Jonathan S Williams in
Google Scholar
PubMed
Search for other papers by Luminita H Pojoga in
Google Scholar
PubMed
Search for other papers by Gordon H Williams in
Google Scholar
PubMed
aldosterone effect. Sexual dimorphism has been documented previously for blood pressure levels and regulation ( Yong et al . 1993 , Himmelmann et al . 1994 ). In humans, hypertension is more common in young males than in premenopausal females; the
Search for other papers by Lucas Monje in
Google Scholar
PubMed
Search for other papers by Jorgelina Varayoud in
Google Scholar
PubMed
Search for other papers by Enrique H Luque in
Google Scholar
PubMed
Search for other papers by Jorge G Ramos in
Google Scholar
PubMed
time point studied. At PND8, ERα 5′UTR variant expression in the POA showed a great sexual dimorphism, since control females showed higher levels of ERα-OT, ERα-O, and ERα-E1 transcripts than male pups ( P < 0.05, Fig. 4A–C ). DES
Search for other papers by T V Novoselova in
Google Scholar
PubMed
Search for other papers by R Larder in
Google Scholar
PubMed
Search for other papers by D Rimmington in
Google Scholar
PubMed
Search for other papers by C Lelliott in
Google Scholar
PubMed
Search for other papers by E H Wynn in
Google Scholar
PubMed
Search for other papers by R J Gorrigan in
Google Scholar
PubMed
Search for other papers by P H Tate in
Google Scholar
PubMed
Search for other papers by L Guasti in
Google Scholar
PubMed
Search for other papers by The Sanger Mouse Genetics Project in
Google Scholar
PubMed
Search for other papers by S O’Rahilly in
Google Scholar
PubMed
Search for other papers by A J L Clark in
Google Scholar
PubMed
Search for other papers by D W Logan in
Google Scholar
PubMed
Search for other papers by A P Coll in
Google Scholar
PubMed
Search for other papers by L F Chan in
Google Scholar
PubMed
either global P -values for genotype adjusted for multiple correction testing, or (in the cases of sexual dimorphism) the P -value is the impact of genotype for that sex. Animal husbandry The care and use of all animals were carried out in
Search for other papers by Chandrika D Mahalingam in
Google Scholar
PubMed
Search for other papers by Bharat Reddy Sampathi in
Google Scholar
PubMed
Search for other papers by Sonali Sharma in
Google Scholar
PubMed
Search for other papers by Tanuka Datta in
Google Scholar
PubMed
Search for other papers by Varsha Das in
Google Scholar
PubMed
Search for other papers by Abdul B Abou-Samra in
Google Scholar
PubMed
Division of Endocrinology, Barbara Ann Karmanos Cancer Institute, Cardiovascular Research Institute, Department of Internal Medicine, Wayne State University School of Medicine, 1107 Elliman Clinical Research Building, 421 East Canfield Avenue, Detroit, Michigan 48201, USA
Division of Endocrinology, Barbara Ann Karmanos Cancer Institute, Cardiovascular Research Institute, Department of Internal Medicine, Wayne State University School of Medicine, 1107 Elliman Clinical Research Building, 421 East Canfield Avenue, Detroit, Michigan 48201, USA
Search for other papers by Nabanita S Datta in
Google Scholar
PubMed
and death in astrocytes and possibly contributes to sexual dimorphisms in brain development ( Zhang et al . 2002 ). MAPKs regulate cyclin D1 ( Terada et al . 1999 ), an unique regulator of cell cycle progression and cell proliferation ( Sherr
Search for other papers by Lianne Abrahams in
Google Scholar
PubMed
Search for other papers by Nina M Semjonous in
Google Scholar
PubMed
Search for other papers by Phil Guest in
Google Scholar
PubMed
Search for other papers by Agnieszka Zielinska in
Google Scholar
PubMed
Search for other papers by Beverly Hughes in
Google Scholar
PubMed
Search for other papers by Gareth G Lavery in
Google Scholar
PubMed
Search for other papers by Paul M Stewart in
Google Scholar
PubMed
, although the ultimate reason for the observed sexual dimorphism remains obscure. 11β-HSD1/H6PDH HETs and double HETs have a urinary 11-DHC profile that is comparable to that of WT animals ( Fig. 2 ). Loss of one 11β-HSD1 and/or one H6PDH allele therefore