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neurobiology of stress and adaptation: central role of the brain. Physiological Reviews 87 873–904. Copyright 2007 the American Physiological Society. Within this broader view of neuroendocrinology in relation to brain–body communication, we modified the
Department of Physiology, Naval Medical University, Shanghai, China
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NHC Key Laboratory of Hormones and Development, Tianjin Key Laboratory of Metabolic Diseases, Tianjin Medical University Chu Hsien-I Memorial Hospital and Tianjin Institute of Endocrinology, Tianjin, China
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adaptation. Lipoprotein lipase (LPL) is dramatically upregulated during BAT activation to promote uptake of free fatty acids from circulation. β-oxidation can directly provide acetyl-CoA for thermogenesis. However, de novo lipogenesis (DNL) genes, such as
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insulin secretion by endurance exercise is related to the activation of the AMPK–UCP2 pathway, and if these adaptations depend on exercise frequency. Materials and Methods Ethical approval All the experiments described herein were approved by the State
Nottingham Digestive Disease Centre and Biomedical Research Centre, School of Medicine, University of Nottingham, Nottingham, UK
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humans, excess adiposity is the consequence of a much smaller change in energy balance over a prolonged period of time and is typically accompanied by a range of behavioural adaptations such as reduced activity and more frequent snacking ( Weinsier et al
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School of Literature, Science, and Arts, University of Michigan, Ann Arbor, Michigan, USA
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Neuroscience Graduate Program, University of Michigan, Ann Arbor, Michigan, USA
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Instituto de Investigaciones en Ingeniería Genética y Biología Molecular, Consejo Nacional de Investigaciones Científicas y Técnicas and Facultad de Ciencias Exactas y Naturales, Universidad de Buenos, Buenos Aires, Argentina
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neuropeptides in the hypothalamus, transient hyperleptinemia and reduced transport of leptin into the brain ( Gustafson et al . 2019 ). The maternal adaptations to pregnancy are mediated largely by the placenta, which separates the maternal and fetal
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higher LDH levels. Higher physical capacity is a normal adaptation to chronic exercise that was observed in both trained groups. Interestingly, the skeletal muscle glycogen levels were not affected by training in any group, though liver glycogen levels
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-GAS ( Fig. 3A and F ), suggesting that the adaptation mechanism to exercise-induced stress and damage has been compromised in Chga -KO-GAS. Figure 3 Evaluation of exercise-induced signaling by Western blot in WT and Chga -KO GAS muscle. Mice were
Department of Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1 NL-6525ED, Nijmegen, The Netherlands
Division of Pediatrics, Department of Clinical and Molecular Medicine, University of Bergen, Haukeland University Hospital, N-5021, Bergen, Norway
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Department of Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1 NL-6525ED, Nijmegen, The Netherlands
Division of Pediatrics, Department of Clinical and Molecular Medicine, University of Bergen, Haukeland University Hospital, N-5021, Bergen, Norway
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Department of Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1 NL-6525ED, Nijmegen, The Netherlands
Division of Pediatrics, Department of Clinical and Molecular Medicine, University of Bergen, Haukeland University Hospital, N-5021, Bergen, Norway
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Department of Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1 NL-6525ED, Nijmegen, The Netherlands
Division of Pediatrics, Department of Clinical and Molecular Medicine, University of Bergen, Haukeland University Hospital, N-5021, Bergen, Norway
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Department of Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1 NL-6525ED, Nijmegen, The Netherlands
Division of Pediatrics, Department of Clinical and Molecular Medicine, University of Bergen, Haukeland University Hospital, N-5021, Bergen, Norway
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Department of Animal Physiology, Faculty of Science, Radboud University Nijmegen, Toernooiveld 1 NL-6525ED, Nijmegen, The Netherlands
Division of Pediatrics, Department of Clinical and Molecular Medicine, University of Bergen, Haukeland University Hospital, N-5021, Bergen, Norway
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most important mechanism is the increase in branchial Na + /K + -ATPase activity to provide the fish with SW-type chloride cells ( McCormick 1996 ); isoform switch may be a second phenomenon in this adaptation. Ágústsson et al. (2003) measured an
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first to show that ARC is required for β-cell survival and sufficient insulin secretion in vivo in response to a genetic model of diabetogenic stress. The role of ARC in β-cell adaptation to high fat diet (HFD) feeding, a physiologically relevant
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). Intermittent SD refers to repeated exposure to a larger, dominant conspecific, which according to traditional definitions would seem to represent a repeated homotypic stressor. However, it is believed that repeated SD does not result in adaptation as it rather