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Graham W Aberdeen Departments of Obstetrics, Gynecology, Reproductive Sciences and Physiology, University of Maryland School of Medicine, Baltimore, Maryland, USA

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Jeffery S Babischkin Departments of Obstetrics, Gynecology, Reproductive Sciences and Physiology, University of Maryland School of Medicine, Baltimore, Maryland, USA

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Gerald J Pepe Department of Physiological Sciences, Eastern Virginia Medical School, Norfolk, Virginia, USA

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Eugene D Albrecht Departments of Obstetrics, Gynecology, Reproductive Sciences and Physiology, University of Maryland School of Medicine, Baltimore, Maryland, USA

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growth factor (VEGF) has a well-established role in promoting angiogenesis in the adult, i.e. the formation and proliferation of capillaries ( Ferrara 1999 , Dvorak 2000 , Wagner 2011 ), and estrogen stimulates VEGF expression in many estrogen receptor

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-23-0364
Volume/Issue:
Volume 262: Issue 1
Open access
Joyce Emons
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Bas E Dutilh Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Eva Decker Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Heide Pirzer Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Carsten Sticht Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Norbert Gretz Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Gudrun Rappold Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Ewan R Cameron Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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James C Neil Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Gary S Stein Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Andre J van Wijnen Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Jan Maarten Wit
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Janine N Post Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Marcel Karperien Department of Paediatrics, Centre for Molecular and Biomolecular Informatics, Department of Human Molecular Genetics, Medical Research Center, Molecular Oncology Laboratory, Department of Cell Biology, Department of Tissue Regeneration, Leiden University Medical Center, 2300 ZA Leiden, The Netherlands

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Introduction Longitudinal growth occurs at the epiphyseal growth plate, a thin layer of cartilage between epiphyseal and metaphyseal bone at the distal ends of the long bones. In the normal growth plate, immature cells are located toward the

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-10-0219
Volume/Issue:
Volume 209: Issue 2
Free access
Soo-Hyun Kim
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Jeremy Turnbull Division of Biomedical Sciences, Institute of Integrative Biology, St George's Medical School, University of London, Cranmer Terrace, London SW17 0RE, UK

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Scott Guimond Division of Biomedical Sciences, Institute of Integrative Biology, St George's Medical School, University of London, Cranmer Terrace, London SW17 0RE, UK

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, largely through their ability to bind multiple interacting partners such as other ECM proteins, growth factors, signal receptors and the adhesion molecules, which is mediated by the multiple, specific domains present within each protein. The best example

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-10-0377
Volume/Issue:
Volume 209: Issue 2
Free access
Stina Häggström Rudolfsson Surgical and Perioperative Sciences, Medical Biosciences, Urology and Andrology and

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Anders Bergh Surgical and Perioperative Sciences, Medical Biosciences, Urology and Andrology and

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Introduction Androgens are the principal regulators of prostate growth and homeostasis. Androgen receptors are present on prostate epithelial and stroma cells, and androgens influence prostate growth by effects both in the epithelium and in the

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-07-0272
Volume/Issue:
Volume 196: Issue 1
Free access
David E Maridas Maine Medical Center Research Institute, Scarborough, Maine, USA

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Victoria E DeMambro Maine Medical Center Research Institute, Scarborough, Maine, USA

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Phuong T Le Maine Medical Center Research Institute, Scarborough, Maine, USA

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Kenichi Nagano Harvard School of Dental Medicine, Boston, Massachusetts, USA

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Roland Baron Harvard School of Dental Medicine, Boston, Massachusetts, USA

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Subburaman Mohan VA Loma Linda Healthcare System, Loma Linda, California, USA

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Clifford J Rosen Maine Medical Center Research Institute, Scarborough, Maine, USA

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Introduction IGF signaling is a crucial pathway controlling muscle, fat and bone growth and is regulated by 6 binding proteins, IGFBP1–6. The IGF-binding proteins can enhance or inhibit IGF action locally ( Gustafsson et al . 1999 , Tahimic

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0673
Volume/Issue:
Volume 233: Issue 1
Open access
Pablo Mendez Instituto Cajal, CSIC, E-28002 Madrid, Spain
Departamento de Biología Celular, Facultad de Biología, Universidad Complutense, E-28040 Madrid, Spain

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Iñigo Azcoitia Instituto Cajal, CSIC, E-28002 Madrid, Spain
Departamento de Biología Celular, Facultad de Biología, Universidad Complutense, E-28040 Madrid, Spain

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Luis Miguel Garcia-Segura Instituto Cajal, CSIC, E-28002 Madrid, Spain
Departamento de Biología Celular, Facultad de Biología, Universidad Complutense, E-28040 Madrid, Spain

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). In this review we focus on one of the mechanisms of action of oestradiol in the brain: the interaction with the signalling of insulin-like growth factor-I (IGF-I). First, we present several lines of evidence of crosstalk between oestradiol and IGF

Article Type:
Review Article
DOI:
https://doi.org/10.1677/joe.1.06058
Volume/Issue:
Volume 185: Issue 1
Free access
P. Davies
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C. L. Eaton
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Introduction

The prostate gland attracts attention and achieves notoriety because of the disorders of growth which beset it during the later stages of life of the human male. Improving longevity increases the incidence of these age-related diseases (Carter & Coffey, 1990). More than 50% of men in the U.K. and U.S.A. have some degree of benign prostatic hyperplasia (BPH), presaging a major impact in terms of morbidity, mortality and health-care costs (Carter, Carter & Isaacs, 1990a; Carter & Coffey, 1990; Griffiths, Davies, Eaton et al. 1991). Even more alarmingly, carcinoma of the prostate is the fourth most common cause of death from malignant disease in the U.K., being responsible for approximately 4000 deaths each year (Griffiths et al. 1991). In the U.S.A., approximately 100 000 new cases present each year, with 28 000 deaths (Carter et al. 1990a; Carter & Coffey, 1990). The prostate gland is the frequently chosen model

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.0.1310005
Volume/Issue:
Volume 131: Issue 1
Restricted access
J Santiago-Moreno
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A Gómez-Brunet
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A Toledano-Díaz
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R Salas-Vega Department of Animal Reproduction, Consejería de Medio Ambiente, INIA, Avenida Puerta de Hierro Km 5.9, Madrid 28040, Spain

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F Gómez-Guillamón Department of Animal Reproduction, Consejería de Medio Ambiente, INIA, Avenida Puerta de Hierro Km 5.9, Madrid 28040, Spain

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A López-Sebastián
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activity ( Hafez 1952 ). The annual arrest of horn growth in wild bovids coincides with the time when males are producing their highest levels of testosterone, i.e. in the autumn, when the photoperiod is shortening and when spermatogenesis is at its height

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0016
Volume/Issue:
Volume 214: Issue 2
Free access
Matthias Haase Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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Matthias Schott Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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Stefan R Bornstein Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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Ludwik K Malendowicz Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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Werner A Scherbaum Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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Holger S Willenberg Department of Endocrinology, Diabetes and Rheumatology, University Hospital Duesseldorf, Moorenstr. 5, D-40225 Duesseldorf, Germany
University of Technology Dresden, University Clinic III, Dresden, Germany
School of Medicine, Department of Histology and Embryology, Poznan, Poland

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assumed to play a fundamental role in adrenal organogenesis, adrenal tissue growth and/or adrenal differentiation. The human adrenal cortex arises from a combined adrenal and gonadal precursor at 4 weeks of gestation as a condensation of coelomic

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-06-0083
Volume/Issue:
Volume 192: Issue 2
Free access
Ken KY Ho Garvan Institute of Medical Research, St. Vincent’s Hospital and the UNSW Sydney, Sydney, New South Wales, Australia

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Anthony J O’Sullivan St. George Hospital and the Faculty of Medicine and Health, UNSW Sydney, Sydney, New South Wales, Australia

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Morton G Burt Southern Adelaide Diabetes and Endocrine, Flinders Medical Centre and College of Medicine and Public Health, and Flinders University, Adelaide, South Australia, Australia

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Hormone replacement therapy is a tenet of endocrinology. The recognition that growth hormone (GH) is biologically important beyond promoting growth in childhood has only been accepted recently despite its isolation over 70 years ago. In addition

Article Type:
Review Article
Collections:
Endocrine Society of Australia special collection
DOI:
https://doi.org/10.1530/JOE-22-0197
Volume/Issue:
Volume 257: Issue 2
Free access