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M J F Newson Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology (LINE), School of Clinical Sciences, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK

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G R Pope Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology (LINE), School of Clinical Sciences, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK

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E M Roberts Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology (LINE), School of Clinical Sciences, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK

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S J Lolait Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology (LINE), School of Clinical Sciences, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK

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A-M O'Carroll Henry Wellcome Laboratories for Integrative Neuroscience and Endocrinology (LINE), School of Clinical Sciences, University of Bristol, Dorothy Hodgkin Building, Whitson Street, Bristol BS1 3NY, UK

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GL 1980 Rat vasopressin response to insulin-induced hypoglycemia . Endocrinology 107 1975 – 1979 . ( doi:10.1210/endo-107-6-1975 ) Beishuizen A Thijs LG 2003 Endotoxin and the hypothalamo–pituitary–adrenal (HPA) axis . Journal of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0375
Volume/Issue:
Volume 216: Issue 1
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Joëlle Dupont PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Sophie Tesseraud PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Michel Derouet PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Anne Collin PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Nicole Rideau PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Sabine Crochet PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Estelle Godet PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Estelle Cailleau-Audouin PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Sonia Métayer-Coustard PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Michel J Duclos PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Christian Gespach PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Tom E Porter PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Larry A Cogburn PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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Jean Simon PRC (UMR 6175), Station de Recherches Avicoles (UR 83), INSERM, Department of Animal and Avian Sciences, Department of Animal and Food Sciences, INRA, 37380 Nouzilly, France

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the fasted state, the control of glycemia is in contrast dependent on glucagon since fasted pancreatectomized chickens tend to develop hypoglycemia rather than hyperglycemia ( Simon 1989 ). Similar conclusions have been obtained in ducks ( Mirsky et

Article Type:
Research Article
DOI:
https://doi.org/10.1677/JOE-08-0055
Volume/Issue:
Volume 197: Issue 3
Free access
Susan Gray Department of Pediatrics, David Geffen School of Medicine at UCLA, Los Angeles, California 90095, USA
Department of Pediatrics, Brown University Medical School and Women and Infants’ Hospital of Rhode Island, Providence, Rhode Island 02912, USA
Department of Pediatrics, University of Pittsburgh School of Medicine and Magee-Womens Research Institute, Pittsburgh, Pennsylvania 15261, USA

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Barbara S Stonestreet Department of Pediatrics, David Geffen School of Medicine at UCLA, Los Angeles, California 90095, USA
Department of Pediatrics, Brown University Medical School and Women and Infants’ Hospital of Rhode Island, Providence, Rhode Island 02912, USA
Department of Pediatrics, University of Pittsburgh School of Medicine and Magee-Womens Research Institute, Pittsburgh, Pennsylvania 15261, USA

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Shanthie Thamotharan Department of Pediatrics, David Geffen School of Medicine at UCLA, Los Angeles, California 90095, USA
Department of Pediatrics, Brown University Medical School and Women and Infants’ Hospital of Rhode Island, Providence, Rhode Island 02912, USA
Department of Pediatrics, University of Pittsburgh School of Medicine and Magee-Womens Research Institute, Pittsburgh, Pennsylvania 15261, USA

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Grazyna B Sadowska Department of Pediatrics, David Geffen School of Medicine at UCLA, Los Angeles, California 90095, USA
Department of Pediatrics, Brown University Medical School and Women and Infants’ Hospital of Rhode Island, Providence, Rhode Island 02912, USA
Department of Pediatrics, University of Pittsburgh School of Medicine and Magee-Womens Research Institute, Pittsburgh, Pennsylvania 15261, USA

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Molly Daood Department of Pediatrics, David Geffen School of Medicine at UCLA, Los Angeles, California 90095, USA
Department of Pediatrics, Brown University Medical School and Women and Infants’ Hospital of Rhode Island, Providence, Rhode Island 02912, USA
Department of Pediatrics, University of Pittsburgh School of Medicine and Magee-Womens Research Institute, Pittsburgh, Pennsylvania 15261, USA

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Jon Watchko Department of Pediatrics, David Geffen School of Medicine at UCLA, Los Angeles, California 90095, USA
Department of Pediatrics, Brown University Medical School and Women and Infants’ Hospital of Rhode Island, Providence, Rhode Island 02912, USA
Department of Pediatrics, University of Pittsburgh School of Medicine and Magee-Womens Research Institute, Pittsburgh, Pennsylvania 15261, USA

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Sherin U Devaskar Department of Pediatrics, David Geffen School of Medicine at UCLA, Los Angeles, California 90095, USA
Department of Pediatrics, Brown University Medical School and Women and Infants’ Hospital of Rhode Island, Providence, Rhode Island 02912, USA
Department of Pediatrics, University of Pittsburgh School of Medicine and Magee-Womens Research Institute, Pittsburgh, Pennsylvania 15261, USA

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resulted from nutrient restriction as a consequence of maternal insulin infusions with maternal hypoglycemia, fetal hypoglycemia and fetal hypoinsulinemia, fetal skeletal muscle GLUT 1 and GLUT 4 concentrations did not change ( Das et al. 1999

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06589
Volume/Issue:
Volume 189: Issue 2
Free access
Astrid C Hauge-Evans Diabetes Research Group, Division of Diabetes and Nutritional Sciences, King's College London, 2.27 Henrietta Raphael Building, Guy's Campus, London SE1 1UL, UK

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James Bowe Diabetes Research Group, Division of Diabetes and Nutritional Sciences, King's College London, 2.27 Henrietta Raphael Building, Guy's Campus, London SE1 1UL, UK

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Zara J Franklin Diabetes Research Group, Division of Diabetes and Nutritional Sciences, King's College London, 2.27 Henrietta Raphael Building, Guy's Campus, London SE1 1UL, UK

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Zoheb Hassan Diabetes Research Group, Division of Diabetes and Nutritional Sciences, King's College London, 2.27 Henrietta Raphael Building, Guy's Campus, London SE1 1UL, UK

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Peter M Jones Diabetes Research Group, Division of Diabetes and Nutritional Sciences, King's College London, 2.27 Henrietta Raphael Building, Guy's Campus, London SE1 1UL, UK

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-Iken D Routh VH Fisher SJ 2010 Brain insulin action regulates hypothalamic glucose sensing and the counterregulatory response to hypoglycemia . Diabetes 59 2271 – 2280 . ( doi:10.2337/db10-0401 ) Gey GO Gey MK 1936 Maintenance of

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0709
Volume/Issue:
Volume 225: Issue 1
Free access
Ewa Ocłoń Department of Animal Physiology and Endocrinology, University of Agriculture in Krakow, Krakow, Poland

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Anna Latacz Department of Animal Physiology and Endocrinology, University of Agriculture in Krakow, Krakow, Poland

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Joanna Zubel–Łojek Department of Animal Physiology and Endocrinology, University of Agriculture in Krakow, Krakow, Poland

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Krystyna Pierzchała–Koziec Department of Animal Physiology and Endocrinology, University of Agriculture in Krakow, Krakow, Poland

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association with compromised insulin-mediated control of hepatic gluconeogenesis. By contrast, mutations associated with impaired hepatic Pck1 expression have been reported to induce hypoglycemia. Interestingly, Karolina and coworkers reported that miR-144

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-15-0495
Volume/Issue:
Volume 229: Issue 3
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Ken Takao Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Katsumi Iizuka Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Yanyan Liu Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Teruaki Sakurai Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Sodai Kubota Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan
Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan

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Saki Kubota-Okamoto Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan
Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan

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Toshinori Imaizumi Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Yoshihiro Takahashi Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Yermek Rakhat Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan
Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan

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Satoko Komori Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Tokuyuki Hirose Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Kenta Nonomura Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Takehiro Kato Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Masami Mizuno Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Tetsuya Suwa Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Yukio Horikawa Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan

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Masakatsu Sone Division of Metabolism and Endocrinology, Department of Internal Medicine, St. Marianna University School of Medicine, Kawasaki, Japan

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Daisuke Yabe Department of Diabetes and Endocrinology, Gifu University Graduate School of Medicine, Gifu, Japan
Yutaka Seino Distinguished Center for Diabetes Research, Kansai Electric Power Medical Research Institute, Kobe, Japan
Division of Molecular and Metabolic Medicine, Department of Physiology and Cell Biology, Kobe University Graduate School of Medicine, Kobe, Japan

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through synthesis and secretion of steroid hormones such as corticosterone. Patients with adrenal cortex insufficiency show plasma hyponatremia, hyperkalemia, and hypoglycemia, indicating that steroid hormones are essential for survival ( Val & Martinez

Article Type:
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DOI:
https://doi.org/10.1530/JOE-20-0442
Volume/Issue:
Volume 248: Issue 3
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A L Pierce
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H Fukada
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W W Dickhoff
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increases in circulating GH in response to insulin-induced hypoglycemia. Streptozotocin injection decreased plasma insulin and liver IGF-I mRNA or circulating IGF-I levels in coho salmon and brown trout ( Plisetskaya & Duan 1994 , Banos et al. 1999 ). Our

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.05892
Volume/Issue:
Volume 184: Issue 2
Free access
Lianne Abrahams School of Clinical and Experimental Medicine, Institute of Biomedical Research, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Edgbaston, Birmingham B15 2TT, UK

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Nina M Semjonous School of Clinical and Experimental Medicine, Institute of Biomedical Research, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Edgbaston, Birmingham B15 2TT, UK

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Phil Guest School of Clinical and Experimental Medicine, Institute of Biomedical Research, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Edgbaston, Birmingham B15 2TT, UK

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Agnieszka Zielinska School of Clinical and Experimental Medicine, Institute of Biomedical Research, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Edgbaston, Birmingham B15 2TT, UK

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Beverly Hughes School of Clinical and Experimental Medicine, Institute of Biomedical Research, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Edgbaston, Birmingham B15 2TT, UK

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Gareth G Lavery School of Clinical and Experimental Medicine, Institute of Biomedical Research, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Edgbaston, Birmingham B15 2TT, UK

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Paul M Stewart School of Clinical and Experimental Medicine, Institute of Biomedical Research, Centre for Endocrinology, Diabetes and Metabolism, University of Birmingham, Edgbaston, Birmingham B15 2TT, UK

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PM 2007 Hypoglycemia with enhanced hepatic glycogen synthesis in recombinant mice lacking hexose-6-phosphate dehydrogenase . Endocrinology 148 6100 – 6106 . doi:10.1210/en.2007-0963 . Lavery GG Walker EA Tiganescu A Ride JP

Article Type:
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DOI:
https://doi.org/10.1530/JOE-12-0178
Volume/Issue:
Volume 214: Issue 3
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E Louiset INSERM U413, INSERM U567, Department of Endocrinology, Laboratory of Cellular and Molecular Neuroendocrinology, European Institute for Peptide Research (IFRMP 23), University of Rouen, 76821 Mont-Saint-Aignan, France

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V Contesse INSERM U413, INSERM U567, Department of Endocrinology, Laboratory of Cellular and Molecular Neuroendocrinology, European Institute for Peptide Research (IFRMP 23), University of Rouen, 76821 Mont-Saint-Aignan, France

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L Groussin INSERM U413, INSERM U567, Department of Endocrinology, Laboratory of Cellular and Molecular Neuroendocrinology, European Institute for Peptide Research (IFRMP 23), University of Rouen, 76821 Mont-Saint-Aignan, France

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D Cartier INSERM U413, INSERM U567, Department of Endocrinology, Laboratory of Cellular and Molecular Neuroendocrinology, European Institute for Peptide Research (IFRMP 23), University of Rouen, 76821 Mont-Saint-Aignan, France

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C Duparc INSERM U413, INSERM U567, Department of Endocrinology, Laboratory of Cellular and Molecular Neuroendocrinology, European Institute for Peptide Research (IFRMP 23), University of Rouen, 76821 Mont-Saint-Aignan, France

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V Perraudin INSERM U413, INSERM U567, Department of Endocrinology, Laboratory of Cellular and Molecular Neuroendocrinology, European Institute for Peptide Research (IFRMP 23), University of Rouen, 76821 Mont-Saint-Aignan, France

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J Bertherat INSERM U413, INSERM U567, Department of Endocrinology, Laboratory of Cellular and Molecular Neuroendocrinology, European Institute for Peptide Research (IFRMP 23), University of Rouen, 76821 Mont-Saint-Aignan, France

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H Lefebvre INSERM U413, INSERM U567, Department of Endocrinology, Laboratory of Cellular and Molecular Neuroendocrinology, European Institute for Peptide Research (IFRMP 23), University of Rouen, 76821 Mont-Saint-Aignan, France
INSERM U413, INSERM U567, Department of Endocrinology, Laboratory of Cellular and Molecular Neuroendocrinology, European Institute for Peptide Research (IFRMP 23), University of Rouen, 76821 Mont-Saint-Aignan, France

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AIMAH responding to physiological stimuli of endogenous vasopressin. In these reports, elevation of plasma AVP provoked by upright posture, insulin-induced hypoglycemia, or hypertonic saline infusion test was followed by an increase in plasma cortisol

Article Type:
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DOI:
https://doi.org/10.1677/JOE-07-0413
Volume/Issue:
Volume 196: Issue 1
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Maria E C Amaral
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Daniel A Cunha
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Gabriel F Anhê
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Mirian Ueno
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Everardo M Carneiro
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Lício A Velloso
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Silvana Bordin
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Antonio C Boschero
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expression of placental lactogen in the beta cells of transgenic mice results in beta cell proliferation, islet mass augmentation, and hypoglycemia. Journal of Biological Chemistry 275 15399 –15406. Withers DJ , Gutierrez JS

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DOI:
https://doi.org/10.1677/joe.1.05547
Volume/Issue:
Volume 183: Issue 3
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