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Isabel R Orriss Department of Comparative Biomedical Sciences, Royal Veterinary College, London, UK

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Dilek Guneri Department of Comparative Biomedical Sciences, Royal Veterinary College, London, UK

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Mark O R Hajjawi Department of Cell & Developmental Biology, University College London, London, UK

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Kristy Shaw Department of Comparative Biomedical Sciences, Royal Veterinary College, London, UK

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Jessal J Patel Department of Comparative Biomedical Sciences, Royal Veterinary College, London, UK

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Timothy R Arnett Department of Cell & Developmental Biology, University College London, London, UK

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internal stores. Expression of the P2Y 2 receptor has been reported in many tissues including heart, blood vessels, lung, kidney and skeletal muscle ( Burnstock 2007 a ). Bone cells express multiple P2 receptor subtypes, and knowledge of the functional

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-17-0042
Volume/Issue:
Volume 233: Issue 3
Free access
Sabashini K Ramchand Department of Endocrinology, Austin Health, Heidelberg, Victoria, Australia
Department of Medicine, Austin Health, The University of Melbourne, Heidelberg, Victoria, Australia

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Yee-Ming Cheung Department of Endocrinology, Austin Health, Heidelberg, Victoria, Australia
Department of Medicine, Austin Health, The University of Melbourne, Heidelberg, Victoria, Australia

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Belinda Yeo Department of Oncology, Austin Health, Heidelberg, Victoria, Australia
Olivia Newton-John Cancer Research Institute, Heidelberg, Victoria, Australia

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Mathis Grossmann Department of Endocrinology, Austin Health, Heidelberg, Victoria, Australia
Department of Medicine, Austin Health, The University of Melbourne, Heidelberg, Victoria, Australia

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proliferative oestradiol signalling in breast cancer cells, is routine care. In addition to its effects on breast cancer cells, oestradiol also plays a vital role in a number of physiological mechanisms including the regulation of bone metabolism ( Riggs et al

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-19-0077
Volume/Issue:
Volume 241: Issue 3
Free access
Yongmei Wang Department of Medicine, Endocrine Unit, Veterans Affairs Medical Center, University of California, San Francisco, California 94117-1080, USA
Department of Radiology, University of California, San Francisco, California 94117-1080, USA

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Takeshi Sakata Department of Medicine, Endocrine Unit, Veterans Affairs Medical Center, University of California, San Francisco, California 94117-1080, USA
Department of Radiology, University of California, San Francisco, California 94117-1080, USA

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Hashem Z Elalieh Department of Medicine, Endocrine Unit, Veterans Affairs Medical Center, University of California, San Francisco, California 94117-1080, USA
Department of Radiology, University of California, San Francisco, California 94117-1080, USA

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Scott J Munson Department of Medicine, Endocrine Unit, Veterans Affairs Medical Center, University of California, San Francisco, California 94117-1080, USA
Department of Radiology, University of California, San Francisco, California 94117-1080, USA

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Andrew Burghardt Department of Medicine, Endocrine Unit, Veterans Affairs Medical Center, University of California, San Francisco, California 94117-1080, USA
Department of Radiology, University of California, San Francisco, California 94117-1080, USA

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Sharmila Majumdar Department of Medicine, Endocrine Unit, Veterans Affairs Medical Center, University of California, San Francisco, California 94117-1080, USA
Department of Radiology, University of California, San Francisco, California 94117-1080, USA

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Bernard P Halloran Department of Medicine, Endocrine Unit, Veterans Affairs Medical Center, University of California, San Francisco, California 94117-1080, USA
Department of Radiology, University of California, San Francisco, California 94117-1080, USA

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Daniel D Bikle Department of Medicine, Endocrine Unit, Veterans Affairs Medical Center, University of California, San Francisco, California 94117-1080, USA
Department of Radiology, University of California, San Francisco, California 94117-1080, USA

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Introduction Parathyroid hormone (PTH) is a major regulator of calcium and phosphate homeostasis. It has complex effects on bone. When given intermittently, PTH increases bone mass and strength ( Alexander et al. 2001 , Dempster

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06351
Volume/Issue:
Volume 189: Issue 2
Free access
Katherine A Staines The Roslin Institute and Royal (Dick) School of Veterinary Studies, The University of Edinburgh, Easter Bush, Edinburgh, Midlothian EH25 9RG, UK

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Vicky E MacRae The Roslin Institute and Royal (Dick) School of Veterinary Studies, The University of Edinburgh, Easter Bush, Edinburgh, Midlothian EH25 9RG, UK

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Colin Farquharson The Roslin Institute and Royal (Dick) School of Veterinary Studies, The University of Edinburgh, Easter Bush, Edinburgh, Midlothian EH25 9RG, UK

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movement and flexible to prevent fracture. The organic component of bone, termed the osteoid, comprises an extracellular matrix (ECM) primarily composed of collagen type I together with several non-collagenous proteins (NCPs). One such family of NCPs is the

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-12-0143
Volume/Issue:
Volume 214: Issue 3
Free access
Shun-Neng Hsu The Roslin Institute and Royal (Dick) School of Veterinary Studies, University of Edinburgh, Easter Bush, Midlothian, UK
Division of Nephrology, Department of Medicine, Tri-Service General Hospital, National Defense Medical Center, Taipei, Taiwan

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Louise A Stephen The Roslin Institute and Royal (Dick) School of Veterinary Studies, University of Edinburgh, Easter Bush, Midlothian, UK

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Scott Dillon The Roslin Institute and Royal (Dick) School of Veterinary Studies, University of Edinburgh, Easter Bush, Midlothian, UK

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Elspeth Milne The Roslin Institute and Royal (Dick) School of Veterinary Studies, University of Edinburgh, Easter Bush, Midlothian, UK

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Behzad Javaheri Comparative Biomedical Sciences, The Royal Veterinary College, London, UK

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Andrew A Pitsillides Comparative Biomedical Sciences, The Royal Veterinary College, London, UK

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Amanda Novak The Roslin Institute and Royal (Dick) School of Veterinary Studies, University of Edinburgh, Easter Bush, Midlothian, UK

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Jose Luis Millán Sanford Burnham Prebys Medical Discovery Institute, La Jolla, California, USA

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Vicky E MacRae The Roslin Institute and Royal (Dick) School of Veterinary Studies, University of Edinburgh, Easter Bush, Midlothian, UK

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Katherine A Staines Centre for Stress and Age-Related Disease, University of Brighton, Brighton, UK

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Colin Farquharson The Roslin Institute and Royal (Dick) School of Veterinary Studies, University of Edinburgh, Easter Bush, Midlothian, UK

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Introduction Chronic kidney disease (CKD) is a disorder characterized by progressive loss of kidney function over time. Patients with advanced CKD frequently develop disturbances in mineral and bone metabolism and fail to maintain normal

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-22-0097
Volume/Issue:
Volume 254: Issue 3
Open access
Russell T Turner Skeletal Biology Laboratory, School of Biological and Population Health Sciences, Oregon State University, Corvallis, Oregon, USA
Center for Healthy Aging Research, Oregon State University, Corvallis, Oregon, USA

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Kenneth A Philbrick Skeletal Biology Laboratory, School of Biological and Population Health Sciences, Oregon State University, Corvallis, Oregon, USA

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Carmen P Wong Skeletal Biology Laboratory, School of Biological and Population Health Sciences, Oregon State University, Corvallis, Oregon, USA

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Adam J Branscum Biostatistics Program, School of Biological and Population Health Sciences, Oregon State University, Corvallis, Oregon, USA

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Urszula T Iwaniec Skeletal Biology Laboratory, School of Biological and Population Health Sciences, Oregon State University, Corvallis, Oregon, USA
Center for Healthy Aging Research, Oregon State University, Corvallis, Oregon, USA

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Introduction Generally, there is a positive association between body weight gain and bone mass ( Iwaniec & Turner 2016 ). However, morbidly obese leptin-deficient ob/ob mice have low bone mass and exhibit skeletal abnormalities, including

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-23-0182
Volume/Issue:
Volume 259: Issue 3
Restricted access
Kenneth A Philbrick Skeletal Biology Laboratory, School of Biological and Population Health Sciences, Oregon State University, Corvallis, Oregon, USA

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Carmen P Wong Skeletal Biology Laboratory, School of Biological and Population Health Sciences, Oregon State University, Corvallis, Oregon, USA

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Adam J Branscum Biostatistics Program, School of Biological and Population Health Sciences, Oregon State University, Corvallis, Oregon, USA

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Russell T Turner Skeletal Biology Laboratory, School of Biological and Population Health Sciences, Oregon State University, Corvallis, Oregon, USA
Center for Healthy Aging Research, Oregon State University, Corvallis, Oregon, USA

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Urszula T Iwaniec Skeletal Biology Laboratory, School of Biological and Population Health Sciences, Oregon State University, Corvallis, Oregon, USA
Center for Healthy Aging Research, Oregon State University, Corvallis, Oregon, USA

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it functions to regulate appetite and energy expenditure ( Banks et al . 1996 ). Leptin signaling is required for normal bone growth in rodents. The absence of leptin signaling in leptin-deficient ob/ob mice or leptin receptor-deficient db

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0484
Volume/Issue:
Volume 232: Issue 3
Free access
Aran Son Department of Oral Biology, Yonsei University College of Dentistry, Seoul, Korea

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Namju Kang Department of Oral Biology, Yonsei University College of Dentistry, Seoul, Korea
BK21 PLUS Project, Yonsei University College of Dentistry, Seoul, Korea

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Sue Young Oh Department of Oral Biology, Yonsei University College of Dentistry, Seoul, Korea

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Ki Woo Kim Department of Oral Biology, Yonsei University College of Dentistry, Seoul, Korea

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Shmuel Muallem Epithelial Signaling and Transport Section, Molecular Physiology and Therapeutics Branch, National Institute of Dental and Craniofacial Research, National Institutes of Health, Bethesda, Maryland, USA

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Yu-Mi Yang Department of Oral Biology, Yonsei University College of Dentistry, Seoul, Korea

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Dong Min Shin Department of Oral Biology, Yonsei University College of Dentistry, Seoul, Korea
BK21 PLUS Project, Yonsei University College of Dentistry, Seoul, Korea

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Introduction Bone is a dynamic organ. It is constantly renewing through a bone remodeling process, mediated by osteoblastic bone formation and osteoclastic bone resorption. In numerous bone diseases, an imbalance in this process can be found

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-19-0123
Volume/Issue:
Volume 242: Issue 3
Free access
Ke Ke Department of Biological Sciences, University of Ulsan, Ulsan, Korea

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Ok-Joo Sul Department of Biological Sciences, University of Ulsan, Ulsan, Korea

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Soo-Wol Chung Department of Biological Sciences, University of Ulsan, Ulsan, Korea

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Jae-Hee Suh Department of Pathology, Ulsan University Hospital, Ulsan, Korea

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Hye-Seon Choi Department of Biological Sciences, University of Ulsan, Ulsan, Korea

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Introduction Bone is a dynamic tissue, aging bone being constantly replaced by new tissue. Bone remodeling occurs via the balanced action of osteoblasts and OCs. Loss of ovarian function increases the rate of bone remodeling and results in a

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-16-0591
Volume/Issue:
Volume 235: Issue 2
Free access
William F Powell Jr
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Kevin J Barry
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Irena Tulum
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Tatsuya Kobayashi
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Stephen E Harris Endocrine Unit, Department of Periodontics, Massachusetts General Hospital and Harvard Medical School, Thier 1101, 50 Blossom Street, Boston, Massachusetts 02114, USA

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F Richard Bringhurst
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Paola Divieti Pajevic
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Introduction Osteocytes are the most abundant cells in bone, outnumbering osteoblasts by 10-fold and osteoclasts by 1000-fold ( Aguirre et al . 2006 ), and yet their function is still incompletely understood. Osteocytes act as

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-10-0308
Volume/Issue:
Volume 209: Issue 1
Free access