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K Katoh Department of Animal Physiology, Graduate School of Agricultural Science, Tohoku University, Amamiyamachi, Aoba-ku, Sendai 981-8555, Japan

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G Furukawa Department of Animal Physiology, Graduate School of Agricultural Science, Tohoku University, Amamiyamachi, Aoba-ku, Sendai 981-8555, Japan

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K Kitade Department of Animal Physiology, Graduate School of Agricultural Science, Tohoku University, Amamiyamachi, Aoba-ku, Sendai 981-8555, Japan

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N Katsumata Department of Animal Physiology, Graduate School of Agricultural Science, Tohoku University, Amamiyamachi, Aoba-ku, Sendai 981-8555, Japan

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Y Kobayashi Department of Animal Physiology, Graduate School of Agricultural Science, Tohoku University, Amamiyamachi, Aoba-ku, Sendai 981-8555, Japan

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Y Obara Department of Animal Physiology, Graduate School of Agricultural Science, Tohoku University, Amamiyamachi, Aoba-ku, Sendai 981-8555, Japan

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Ultradian growth hormone rhythm in the rat: effects of feeding, hyperglycemia, and insulin-induced hypoglycemia. Endocrinology 99 720 –727. Thomas GB , Mercer JE, Karalis T, Rao A, Cummins JT & Clarke IJ 1990 Effect of

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.05898
Volume/Issue:
Volume 183: Issue 3
Free access
R Wang
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N Yashpal
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F Bacchus
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J Li
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mice that over-express HGF in islets are resistant to the diabetogenic effects of the β-cell toxin streptozotocin ( Garcia-Ocana et al. 2001 ), and β-cell proliferation, islet cell mass and hypoglycemia are increased. Thus it is speculated that HGF

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.05788
Volume/Issue:
Volume 183: Issue 1
Free access
Neehar Gupta Departments of Physiology and
Medicine, Medical Sciences Building, University of Toronto, Toronto, Ontario M5S 1A8, Canada

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Edward Park Departments of Physiology and
Medicine, Medical Sciences Building, University of Toronto, Toronto, Ontario M5S 1A8, Canada

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Harmanjit Sandhu Departments of Physiology and
Medicine, Medical Sciences Building, University of Toronto, Toronto, Ontario M5S 1A8, Canada

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Tracy Goh Departments of Physiology and
Medicine, Medical Sciences Building, University of Toronto, Toronto, Ontario M5S 1A8, Canada

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Vaja Tchipashvili Departments of Physiology and
Medicine, Medical Sciences Building, University of Toronto, Toronto, Ontario M5S 1A8, Canada

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Adria Giacca Departments of Physiology and
Medicine, Medical Sciences Building, University of Toronto, Toronto, Ontario M5S 1A8, Canada

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glucagon and glucose production to hypoglycemia with intraperitoneal versus subcutaneous insulin treatment. Metabolism 49 984 –989.

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06443
Volume/Issue:
Volume 190: Issue 3
Free access
Run Yu UCLA School of Medicine, Cedars-Sinai Research Institute, Cedars-Sinai Medical Center, 8700 Beverly Blvd, Room D3066, Los Angeles, California 90048, USA

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Martha Cruz-Soto UCLA School of Medicine, Cedars-Sinai Research Institute, Cedars-Sinai Medical Center, 8700 Beverly Blvd, Room D3066, Los Angeles, California 90048, USA

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Sergio Li Calzi UCLA School of Medicine, Cedars-Sinai Research Institute, Cedars-Sinai Medical Center, 8700 Beverly Blvd, Room D3066, Los Angeles, California 90048, USA

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Hongxiang Hui UCLA School of Medicine, Cedars-Sinai Research Institute, Cedars-Sinai Medical Center, 8700 Beverly Blvd, Room D3066, Los Angeles, California 90048, USA

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Shlomo Melmed UCLA School of Medicine, Cedars-Sinai Research Institute, Cedars-Sinai Medical Center, 8700 Beverly Blvd, Room D3066, Los Angeles, California 90048, USA

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JP, Mizukami Y, Li J, Graeme-Cook F & Chung DC 2005 Overexpression of cyclin D1 in pancreatic β-cells in vivo results in islet hyperplasia without hypoglycemia. Diabetes 54 712 –719. Zou H , McGarry TJ, Bernal T

Article Type:
Research Article
DOI:
https://doi.org/10.1677/joe.1.06885
Volume/Issue:
Volume 191: Issue 1
Free access
Sarah L Armour Section for Cell Biology and Physiology, Department of Biology, University of Copenhagen, Denmark

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Jade E Stanley Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, Tennessee, USA

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James Cantley Division of Cellular and Systems Medicine, School of Medicine, University of Dundee, UK

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E Danielle Dean Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, Tennessee, USA
Division of Diabetes, Endocrinology, & Metabolism, Vanderbilt University Medical Center School of Medicine, Nashville, Tennessee, USA

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Jakob G Knudsen Section for Cell Biology and Physiology, Department of Biology, University of Copenhagen, Denmark

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normal glucose concentrations . Metabolism: Clinical and Experimental 53 1443 – 1448 . ( https://doi.org/10.1016/j.metabol.2004.06.011 ) Bolli GB Gottesman IS Cryer PE & Gerich JE 1984 Glucose counterregulation during prolonged hypoglycemia

Article Type:
Review Article
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100 years of glucagon: Anniversary collection
DOI:
https://doi.org/10.1530/JOE-23-0081
Volume/Issue:
Volume 259: Issue 1
Free access
Margaret K Hahn Centre for Addiction and Mental Health, Toronto, Ontario, Canada
Institute of Medical Sciences, University of Toronto, Toronto, Ontario, Canada
Department of Pharmacology, University of Toronto, Toronto, Ontario, Canada
Department of Psychiatry, University of Toronto, Toronto, Ontario, Canada
Banting & Best Diabetes Centre, Toronto, Ontario, Canada

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Adria Giacca Institute of Medical Sciences, University of Toronto, Toronto, Ontario, Canada
Banting & Best Diabetes Centre, Toronto, Ontario, Canada
Department of Physiology, University of Toronto, Toronto, Ontario, Canada

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Sandra Pereira Centre for Addiction and Mental Health, Toronto, Ontario, Canada
Department of Physiology, University of Toronto, Toronto, Ontario, Canada

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reported that shorter fasting (2 h) is ideal when performing ITTs because hepatic glycogen content is similar to the nonfasted state ( Carper et al. 2020 ) and there is less risk of fatal hypoglycemia. Genetic background Genetic background of mice

Article Type:
Review Article
Collections:
Insulin resistance and type 2 diabetes mellitus
DOI:
https://doi.org/10.1530/JOE-23-0308
Volume/Issue:
Volume 260: Issue 3
Open access
Andrew W Norris Department of Pediatrics, University of Iowa, Iowa City, Iowa, USA
Fraternal Order of Eagles Diabetes Research Center, University of Iowa, Iowa City, Iowa, USA

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Katie Larson Ode Department of Pediatrics, University of Iowa, Iowa City, Iowa, USA
Fraternal Order of Eagles Diabetes Research Center, University of Iowa, Iowa City, Iowa, USA

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Lina Merjaneh Division of Endocrinology & Diabetes, Seattle Children’s Hospital, Seattle, Washington, USA

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Srinath Sanda Department of Pediatrics, University of California San Francisco, San Francisco, California, USA
Diabetes Center, University of California San Francisco, San Francisco, California, USA

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Yaling Yi Department of Anatomy and Cell Biology, University of Iowa, Iowa City, Iowa, USA

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Xingshen Sun Department of Anatomy and Cell Biology, University of Iowa, Iowa City, Iowa, USA

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John F Engelhardt Fraternal Order of Eagles Diabetes Research Center, University of Iowa, Iowa City, Iowa, USA
Department of Anatomy and Cell Biology, University of Iowa, Iowa City, Iowa, USA

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Rebecca L Hull Veterans Affairs Puget Sound Health Care System, Seattle, Washington, USA
Division of Metabolism, Endocrinology and Nutrition, Department of Medicine, University of Washington, Seattle, Washington, USA

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hyperglycemia and hepatic insulin resistance in type 2 diabetes, while an inappropriately low counter-regulatory glucagon response is an important contributor to hypoglycemia in diabetes. An insufficient glucagon response to arginine has been reported in CF

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-18-0468
Volume/Issue:
Volume 241: Issue 1
Free access
Xianxian Huang Pharmaceutical College, Guangxi Medical University, Nanning, Guangxi, China

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Jing Liu Pharmaceutical College, Guangxi Medical University, Nanning, Guangxi, China

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Guangquan Peng Pharmaceutical College, Guangxi Medical University, Nanning, Guangxi, China

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Mingyue Lu Pharmaceutical College, Guangxi Medical University, Nanning, Guangxi, China

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Zhongbo Zhou School of Pharmacy, Youjiang Medical University for Nationalities, Baise, Guangxi, China

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Neng Jiang Pharmaceutical College, Guangxi Medical University, Nanning, Guangxi, China

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Zhiming Yan Pharmaceutical College, Guangxi Medical University, Nanning, Guangxi, China

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glucagon release during hypoglycemia, ultimately regulating glucose levels without impacting insulin secretion ( Christensen et al. 2011 ). However, compared to GLP-1, GIP has exhibited a noticeable decrease in acute insulinotropic activity in individuals

Article Type:
Review Article
DOI:
https://doi.org/10.1530/JOE-23-0404
Volume/Issue:
Volume 262: Issue 3
Restricted access
Niels L Mulder Department of Pediatrics, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands

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Rick Havinga Department of Pediatrics, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands

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Joost Kluiver Department of Pathology and Medical Biology, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands

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Albert K Groen Department of Pediatrics, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands
Department of Laboratory Medicine, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands

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Janine K Kruit Department of Pediatrics, University of Groningen, University Medical Center Groningen, Groningen, The Netherlands

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homeostasis remained unaltered in chow-fed mice overexpressing miR-132 indicating that the risk on hypoglycemia is low. Mice treated with dsAAV-RIP-miR132, however, did show improved insulin secretion during high-fat diet feeding. Recent findings identified

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-18-0287
Volume/Issue:
Volume 240: Issue 2
Free access
Tatiana Dorfman Laboratory of Intestinal Adaptation and Recovery, Departments of Pediatric Surgery B, Pathology, Section of Pediatric Surgery, The Ruth and Bruce Rappaport Faculty of Medicine, Technion‐Israel Institute of Technology, Haifa, Israel

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Yulia Pollak Laboratory of Intestinal Adaptation and Recovery, Departments of Pediatric Surgery B, Pathology, Section of Pediatric Surgery, The Ruth and Bruce Rappaport Faculty of Medicine, Technion‐Israel Institute of Technology, Haifa, Israel

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Rima Sohotnik Laboratory of Intestinal Adaptation and Recovery, Departments of Pediatric Surgery B, Pathology, Section of Pediatric Surgery, The Ruth and Bruce Rappaport Faculty of Medicine, Technion‐Israel Institute of Technology, Haifa, Israel

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Arnold G Coran Laboratory of Intestinal Adaptation and Recovery, Departments of Pediatric Surgery B, Pathology, Section of Pediatric Surgery, The Ruth and Bruce Rappaport Faculty of Medicine, Technion‐Israel Institute of Technology, Haifa, Israel

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Jacob Bejar Laboratory of Intestinal Adaptation and Recovery, Departments of Pediatric Surgery B, Pathology, Section of Pediatric Surgery, The Ruth and Bruce Rappaport Faculty of Medicine, Technion‐Israel Institute of Technology, Haifa, Israel

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Igor Sukhotnik Laboratory of Intestinal Adaptation and Recovery, Departments of Pediatric Surgery B, Pathology, Section of Pediatric Surgery, The Ruth and Bruce Rappaport Faculty of Medicine, Technion‐Israel Institute of Technology, Haifa, Israel
Laboratory of Intestinal Adaptation and Recovery, Departments of Pediatric Surgery B, Pathology, Section of Pediatric Surgery, The Ruth and Bruce Rappaport Faculty of Medicine, Technion‐Israel Institute of Technology, Haifa, Israel

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.5). Blood glucose levels vary between 400–550 in diabetic rats and 300–350 in diabetic rats treated with insulin. The animals had no Clinical signs of hypoglycemia. Cell proliferation and apoptosis Diabetic rats (group B) demonstrated a significant increase

Article Type:
Research Article
DOI:
https://doi.org/10.1530/JOE-14-0725
Volume/Issue:
Volume 226: Issue 3
Free access