Filter

Refine by date

—

Refine by collections

  • Insulin resistance and type 2 diabetes mellitus (3)

Refine by access

  • Print
  • Save

Search Results

You are looking at 1 - 3 of 3 items for :

  • Insulin resistance and type 2 diabetes mellitus x
  • Refine by access: All content x
Clear All
Margaret K Hahn Centre for Addiction and Mental Health, Toronto, Ontario, Canada
Institute of Medical Sciences, University of Toronto, Toronto, Ontario, Canada
Department of Pharmacology, University of Toronto, Toronto, Ontario, Canada
Department of Psychiatry, University of Toronto, Toronto, Ontario, Canada
Banting & Best Diabetes Centre, Toronto, Ontario, Canada

Search for other papers by Margaret K Hahn in
Google Scholar
PubMed Close
,
Adria Giacca Institute of Medical Sciences, University of Toronto, Toronto, Ontario, Canada
Banting & Best Diabetes Centre, Toronto, Ontario, Canada
Department of Physiology, University of Toronto, Toronto, Ontario, Canada

Search for other papers by Adria Giacca in
Google Scholar
PubMed Close
, and
Sandra Pereira Centre for Addiction and Mental Health, Toronto, Ontario, Canada
Department of Physiology, University of Toronto, Toronto, Ontario, Canada

Search for other papers by Sandra Pereira in
Google Scholar
PubMed Close

unique. When glucose that is consumed orally, either by gavage or starting at the mouth, reaches the intestinal lumen, it is sensed by enteroendocrine K and L cells, which will release the incretin hormones glucose-dependent insulinotropic polypeptide

Article Type:
Review Article
Collections:
Insulin resistance and type 2 diabetes mellitus
DOI:
https://doi.org/10.1530/JOE-23-0308
Volume/Issue:
Volume 260: Issue 3
Open access
Charlotte Steenblock Department of Internal Medicine III, University Hospital Carl Gustav Carus, Technische Universität Dresden, Dresden, Germany

Search for other papers by Charlotte Steenblock in
Google Scholar
PubMed Close
,
Nicole Bechmann Institute of Clinical Chemistry and Laboratory Medicine, University Hospital Carl Gustav Carus Dresden, Technische Universität Dresden, Dresden, Germany

Search for other papers by Nicole Bechmann in
Google Scholar
PubMed Close
,
Felix Beuschlein Department of Endocrinology, Diabetology and Clinical Nutrition, University Hospital Zurich (USZ) and University of Zurich (UZH), Zürich, Switzerland

Search for other papers by Felix Beuschlein in
Google Scholar
PubMed Close
,
Christian Wolfrum Department of Health Sciences and Technology, Laboratory of Translational Nutrition Biology, Institute of Food, Nutrition and Health, ETH Zürich, Schwerzenbach, Switzerland

Search for other papers by Christian Wolfrum in
Google Scholar
PubMed Close
, and
Stefan R Bornstein Department of Internal Medicine III, University Hospital Carl Gustav Carus, Technische Universität Dresden, Dresden, Germany
Department of Endocrinology, Diabetology and Clinical Nutrition, University Hospital Zurich (USZ) and University of Zurich (UZH), Zürich, Switzerland
School of Cardiovascular and Metabolic Medicine and Sciences, Faculty of Life Sciences & Medicine, King’s College London, London, UK

Search for other papers by Stefan R Bornstein in
Google Scholar
PubMed Close

Hamad M Mohammad MG Khan NA Saber MM Semreen MH & Steenblock C 2022 Long-COVID, metabolic and endocrine disease . Hormone and Metabolic Research 54 562 – 566 . ( https://doi.org/10.1055/a-1878-9307 ) Boucher J Kleinridders A & Kahn

Article Type:
Research Article
Collections:
Insulin resistance and type 2 diabetes mellitus
DOI:
https://doi.org/10.1530/JOE-23-0027
Volume/Issue:
Volume 258: Issue 2
Restricted access
Sandra K Szlapinski London, Ontario, Canada

Search for other papers by Sandra K Szlapinski in
Google Scholar
PubMed Close
and
David J Hill Diabetes, Endocrinology and Metabolism, Lawson Health Research Institute, London, Ontario, Canada
Physiology and Pharmacology, Western University, London, Ontario, Canada

Search for other papers by David J Hill in
Google Scholar
PubMed Close

( Szlapinski & Hill 2021 ). In humans, maternal insulin resistance is driven by placental growth hormone, placental lactogen, estrogen, progesterone, and other pregnancy hormones ( Newbern & Freemark 2011 ). During this potential state of metabolic stress, the

Article Type:
Review Article
Collections:
Insulin resistance and type 2 diabetes mellitus
DOI:
https://doi.org/10.1530/JOE-23-0317
Volume/Issue:
Volume 260: Issue 3
Restricted access