Search Results
Search for other papers by Jennifer M Ikle in
Google Scholar
PubMed
Stanford Diabetes Research Center, Stanford University, Stanford, California, USA
Search for other papers by Anna L Gloyn in
Google Scholar
PubMed
mutations lead to over-secretion of insulin and hypoglycemia, and given the lack of compensation by the WT allele, the phenotype is more varied ( Glaser et al. 1998 , Gloyn 2003 , Cuesta-Muñoz et al. 2004 ). Patients with GCK-MODY have stable mild
Search for other papers by Emma Ahlqvist in
Google Scholar
PubMed
Institute for Molecular Medicine Finland (FIMM), Helsinki University, Helsinki, Finland
Search for other papers by Rashmi B Prasad in
Google Scholar
PubMed
Institute for Molecular Medicine Finland (FIMM), Helsinki University, Helsinki, Finland
Search for other papers by Leif Groop in
Google Scholar
PubMed
. However, this needs to be tested in a clinical setting taking the risk of hypoglycemia associated with sulfonylureas in elderly patients into account. Using continuous variables could provide even better prediction of specific complications and response to
Search for other papers by Jennifer Chen in
Google Scholar
PubMed
Department of Endocrinology and Diabetes, Westmead Hospital, Sydney, New South Wales, Australia
Garvan Institute of Medical Research, Sydney, New South Wales, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, New South Wales, Australia
Search for other papers by Jenny E Gunton in
Google Scholar
PubMed
hypoglycemia was eliminated in 90% of recipients over 5 years of follow-up. Variables affecting graft outcomes included recipient age, induction and maintenance immunosuppression, and total islet equivalents (IEQs) infused (CITR). Long-term graft function is
