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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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KG Jebsen Center for Diabetes Research, Department of Clinical Science, University of Bergen, Bergen, Norway
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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Instituto Maimónides de Investigación Biomédica (IMIBIC)/Hospital Reina Sofía, Córdoba, Spain
FiDiPro Program, University of Turku, Turku, Finland
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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, Simonian & Herbison 1997 , Voisin et al. 1997 , Osterlund et al. 1998 , Merchenthaler et al. 2004 ). Recent evidence has shown that E2 has a nucleus-specific action in the hypothalamus to modulate energy homeostasis, particularly within the ARC and
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DZHK (German Center for Cardiovascular Research), Partner Site Göttingen, Göttingen, Germany
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DZHK (German Center for Cardiovascular Research), Partner Site Göttingen, Göttingen, Germany
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DZHK (German Center for Cardiovascular Research), Partner Site Göttingen, Göttingen, Germany
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Introduction The gonadal function is controlled by a complex interaction of regulatory signals, involving hypothalamus, pituitary and the gonads themselves. Altogether, they form the reproductive axis (a.k.a. the hypothalamic
Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA
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Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA
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Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA
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Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA
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Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA
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Department of Kinesiology, East Carolina University, Greenville, North Carolina, USA
Human Performance Laboratory, College of Human Performance and Health, East Carolina University, Greenville, North Carolina, USA
Department of Physiology, East Carolina University, Greenville, North Carolina, USA
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hypothalamus have demonstrated that females have increased satiety-inducing pro-opiomelanocortin (POMC ARC ) neurons ( Nohara et al. 2011 ), reduced hunger-inducing neuropeptide Y (NPY) expression ( Urban et al. 1993 ), decreased sensitivity to central
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later in life. Thus, the perinatal perturbation of foetus/neonate nutrient supply has been proposed to be a key determinant, especially the degree of mismatch between the pre- and post-natal environments ( Gluckman et al . 2008 ). The hypothalamus
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Introduction The time at which puberty begins is the culmination of a series of events within the hypothalamus that results in the increased pulsatile release of luteinizing hormone-releasing hormone (LHRH) secretion. This change in LHRH
Graham Centre for Agricultural Innovation, Wagga Wagga, New South Wales, Australia
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Graham Centre for Agricultural Innovation, Wagga Wagga, New South Wales, Australia
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School of Animal & Veterinary Sciences, Charles Sturt University, Wagga Wagga, New South Wales, Australia
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gonadotrophin, luteinising hormone (LH), then acts as an amplifier to the GnRH signal ( Clarke & Cummins 1982 ). In sheep, at least, the neurons that produce GnRH receive relatively little synaptic input compared to other neurons within the hypothalamus ( Lehman
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studies have shown that leptin action is required for the formation of the axonal projections that extend from ARH neurons to important second-order neurons, such as the paraventricular nucleus of the hypothalamus (PVH) ( Bouret et al. 2004 a, b
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Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China
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Department of Endocrinology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China
Canada-China-New Zealand Joint Laboratory of Maternal and Fetal Medicine, Chongqing Medical University, Chongqing, China
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those from citrate buffer (CB)-treated euglycemic fathers (CB-O). Our aim was to determine the impact of a paternal hyperglycemia on metabolic homeostasis of adult offspring, with particular focus on analysis of changes in hypothalamus-mediated food
Department of Physiological Sciences, State University of Londrina, Londrina, Brazil
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Biotechnology Unit, University of Ribeirao Preto, Ribeirao Preto, Brazil
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Department of Physiological Sciences, Biomedical Sciences Institute, Federal University of Alfenas, Alfenas, Brazil
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regulation of food intake, as evidenced by increases in the expression of the anorexigenic neuropeptide corticotrophin-releasing factor ( Crf ) in the paraventricular nucleus of the hypothalamus (PVN) ( Uchoa et al. 2010 ) and reduction in the expression of
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Introduction Hypothalamus is the critical site of integration for the central and peripheral signals in regulation of energy and glucose homeostasis. Leptin and insulin are the two most important peripheral signals that play obligatory role in