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Sunita M C De Sousa
Sunita M C De Sousa
Endocrine & Metabolic Unit, Royal Adelaide Hospital, Adelaide, Australia
South Australian Adult Genetics Unit, Royal Adelaide Hospital, Adelaide, Australia
Adelaide Medical School, University of Adelaide, Adelaide, Australia
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South Australian Adult Genetics Unit, Royal Adelaide Hospital, Adelaide, Australia
Adelaide Medical School, University of Adelaide, Adelaide, Australia
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Nèle F Lenders
Nèle F Lenders
Department of Endocrinology, St Vincent’s Hospital, Sydney, NSW, Australia
Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Lydia S Lamb
Lydia S Lamb
Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Warrick J Inder
Warrick J Inder
Department of Diabetes and Endocrinology, Princess Alexandra Hospital, Brisbane, Australia
Academy for Medical Education, Faculty of Medicine, the University of Queensland, Brisbane, Australia
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Academy for Medical Education, Faculty of Medicine, the University of Queensland, Brisbane, Australia
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Ann McCormack
Ann McCormack
Department of Endocrinology, St Vincent’s Hospital, Sydney, NSW, Australia
Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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pathway promotes tumour growth via effects on cell proliferation, angiogenesis, vascular permeability and the tumour immune microenvironment. VEGF binds tyrosine kinase receptors on the cell membrane, triggering downstream PI3k-Akt, Ras, VEGFA, Mapk and
Adam Hagg
Adam Hagg
School of Biomedical Sciences, University of Queensland, Brisbane, Australia
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Eliza O’Shea
Eliza O’Shea
School of Biomedical Sciences, University of Queensland, Brisbane, Australia
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Craig A Harrison
Craig A Harrison
Department of Physiology, Monash Biomedicine Discovery Institute, Monash University, Clayton, Australia
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Kelly L Walton
Kelly L Walton
School of Biomedical Sciences, University of Queensland, Brisbane, Australia
Department of Physiology, Monash Biomedicine Discovery Institute, Monash University, Clayton, Australia
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Department of Physiology, Monash Biomedicine Discovery Institute, Monash University, Clayton, Australia
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wasting is driven by SMAD2 and SMAD3 which act to transcriptionally regulate E3 ubiquitin ligases including Atrogin-1 ( Goodman et al. 2013 , Sartori et al. 2009 ), suppress Akt/mTOR-mediated protein synthesis ( Goodman et al. 2013 ) and decrease
