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Adam Hagg
Adam Hagg
School of Biomedical Sciences, University of Queensland, Brisbane, Australia
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Eliza O’Shea
Eliza O’Shea
School of Biomedical Sciences, University of Queensland, Brisbane, Australia
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Craig A Harrison
Craig A Harrison
Department of Physiology, Monash Biomedicine Discovery Institute, Monash University, Clayton, Australia
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Kelly L Walton
Kelly L Walton
School of Biomedical Sciences, University of Queensland, Brisbane, Australia
Department of Physiology, Monash Biomedicine Discovery Institute, Monash University, Clayton, Australia
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Department of Physiology, Monash Biomedicine Discovery Institute, Monash University, Clayton, Australia
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complex with the ActRIIA/B receptors which effectively disables intracellular SMAD activation ( Fig. 2A ). Activin A and activin B have also been reported to stimulate SMAD-independent signalling pathways involving p38 MAPK, ERK1/2 and JNK ( Bao et al
Sunita M C De Sousa
Sunita M C De Sousa
Endocrine & Metabolic Unit, Royal Adelaide Hospital, Adelaide, Australia
South Australian Adult Genetics Unit, Royal Adelaide Hospital, Adelaide, Australia
Adelaide Medical School, University of Adelaide, Adelaide, Australia
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South Australian Adult Genetics Unit, Royal Adelaide Hospital, Adelaide, Australia
Adelaide Medical School, University of Adelaide, Adelaide, Australia
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Nèle F Lenders
Nèle F Lenders
Department of Endocrinology, St Vincent’s Hospital, Sydney, NSW, Australia
Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Lydia S Lamb
Lydia S Lamb
Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Warrick J Inder
Warrick J Inder
Department of Diabetes and Endocrinology, Princess Alexandra Hospital, Brisbane, Australia
Academy for Medical Education, Faculty of Medicine, the University of Queensland, Brisbane, Australia
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Academy for Medical Education, Faculty of Medicine, the University of Queensland, Brisbane, Australia
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Ann McCormack
Ann McCormack
Department of Endocrinology, St Vincent’s Hospital, Sydney, NSW, Australia
Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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. 2020 b ). EGFR is a transmembrane glycoprotein, the binding of which triggers downstream signalling via MAPK and P13K pathways. Increased EGFR expression has been associated with proliferation, angiogenesis, invasion, recurrence and transition of PAs
