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Ken KY Ho
Ken KY Ho
Garvan Institute of Medical Research, St. Vincent’s Hospital and the UNSW Sydney, Sydney, New South Wales, Australia
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Anthony J O’Sullivan
Anthony J O’Sullivan
St. George Hospital and the Faculty of Medicine and Health, UNSW Sydney, Sydney, New South Wales, Australia
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Morton G Burt
Morton G Burt
Southern Adelaide Diabetes and Endocrine, Flinders Medical Centre and College of Medicine and Public Health, and Flinders University, Adelaide, South Australia, Australia
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with GHD and because GH increases insulin resistance, there has been concern about the risk of developing metabolic syndrome or diabetes during GH therapy. A meta-analysis of 13 studies reported an average increase of 0.22 mmol/L in fasting glucose
Gary A Wittert
Gary A Wittert
Freemasons Centre for Male Health and Wellbeing, South Australian Health and Medical Research Institute, and University of Adelaide, Adelaide, South Australia, Australia
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Mathis Grossmann
Mathis Grossmann
Department of Medicine, The University of Melbourne and Department of Endocrinology Austin Health, Heidelberg, Australia
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Bu B Yeap
Bu B Yeap
Medical School, University of Western Australia, and Department of Endocrinology and Diabetes, Fiona Stanley Hospital, Perth, Western Australia, Australia
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David J Handelsman
David J Handelsman
ANZAC Research Institute, University of Sydney and Andrology Department, Concord Hospital, Sydney, New South Wales, Australia
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decrease in serum testosterone concentration in middle to older aged men is associated with a reduction serum SHBG with mid-normal serum LH and FSH and is characterised by an accumulation of visceral adipose tissue associated with insulin resistance
Jun Yang
Jun Yang
Centre of Endocrinology and Metabolism, Hudson Institute of Medical Research, Clayton, Victoria, Australia
Department of Medicine, Monash University, Clayton, Victoria, Australia
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Department of Medicine, Monash University, Clayton, Victoria, Australia
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Morag J Young
Morag J Young
Cardiovascular Endocrinology Laboratory, Discovery & Preclinical Domain, Baker Heart and Diabetes Institute, Melbourne, Victoria, Australia
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Timothy J Cole
Timothy J Cole
Department of Biochemistry and Molecular Biology, Biomedicine Discovery Institute, Monash University, Clayton, Victoria, Australia
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Peter J Fuller
Peter J Fuller
Centre of Endocrinology and Metabolism, Hudson Institute of Medical Research, Clayton, Victoria, Australia
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.33, 95% CI 1.01–1.74) and metabolic syndrome (OR 1.53, 1.22–1.91) in subjects with PA compared to those with essential hypertension ( Monticone et al. 2018 ). Consistent with this, treatment of PA has been shown to increase insulin secretion and
Sunita M C De Sousa
Sunita M C De Sousa
Endocrine & Metabolic Unit, Royal Adelaide Hospital, Adelaide, Australia
South Australian Adult Genetics Unit, Royal Adelaide Hospital, Adelaide, Australia
Adelaide Medical School, University of Adelaide, Adelaide, Australia
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South Australian Adult Genetics Unit, Royal Adelaide Hospital, Adelaide, Australia
Adelaide Medical School, University of Adelaide, Adelaide, Australia
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Nèle F Lenders
Nèle F Lenders
Department of Endocrinology, St Vincent’s Hospital, Sydney, NSW, Australia
Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Lydia S Lamb
Lydia S Lamb
Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Warrick J Inder
Warrick J Inder
Department of Diabetes and Endocrinology, Princess Alexandra Hospital, Brisbane, Australia
Academy for Medical Education, Faculty of Medicine, the University of Queensland, Brisbane, Australia
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Academy for Medical Education, Faculty of Medicine, the University of Queensland, Brisbane, Australia
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Ann McCormack
Ann McCormack
Department of Endocrinology, St Vincent’s Hospital, Sydney, NSW, Australia
Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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Garvan Institute of Medical Research, Sydney, NSW, Australia
St Vincent’s Clinical School, University of New South Wales, Sydney, NSW, Australia
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resistance, with reduced response to somatostatin analogues ( Jennings et al. 2009 , Joshi et al. 2018 ). CDKN1B was also implicated in familial pituitary tumours in 2006 through a process of linkage analysis, expression profiling and DNA sequencing
Chau Thien Tay
Chau Thien Tay
Monash Centre for Health Research and Implementation, School of Public Health and Preventive Medicine, Monash University, Victoria, Australia
Department of Endocrinology and Diabetes, Monash Health, Victoria, Australia
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Department of Endocrinology and Diabetes, Monash Health, Victoria, Australia
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Rhonda Garrad
Rhonda Garrad
Monash Centre for Health Research and Implementation, School of Public Health and Preventive Medicine, Monash University, Victoria, Australia
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Aya Mousa
Aya Mousa
Monash Centre for Health Research and Implementation, School of Public Health and Preventive Medicine, Monash University, Victoria, Australia
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Mahnaz Bahri
Mahnaz Bahri
Monash Centre for Health Research and Implementation, School of Public Health and Preventive Medicine, Monash University, Victoria, Australia
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Anju Joham
Anju Joham
Monash Centre for Health Research and Implementation, School of Public Health and Preventive Medicine, Monash University, Victoria, Australia
Department of Endocrinology and Diabetes, Monash Health, Victoria, Australia
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Department of Endocrinology and Diabetes, Monash Health, Victoria, Australia
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Helena Teede
Helena Teede
Monash Centre for Health Research and Implementation, School of Public Health and Preventive Medicine, Monash University, Victoria, Australia
Department of Endocrinology and Diabetes, Monash Health, Victoria, Australia
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Department of Endocrinology and Diabetes, Monash Health, Victoria, Australia
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, Dokras et al. 2018 , Kakoly et al. 2018 , Tay et al. 2019 , 2020 a ). While understanding that HA and insulin resistance are the key pathophysiological drivers of PCOS, the underlying aetiology, primarily the genetics’ and epigenetics
Eugenie Macfarlane
Eugenie Macfarlane
Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia
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Hong Zhou
Hong Zhou
Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia
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Markus J Seibel
Markus J Seibel
Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia
Department of Endocrinology and Metabolism, Concord Repatriation General Hospital, Sydney, Australia
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Department of Endocrinology and Metabolism, Concord Repatriation General Hospital, Sydney, Australia
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, hypertension, and metabolic disturbances such as insulin resistance, diabetes, and weight gain ( Buttgereit et al. 2005 , Fardet & Fève 2014 ). Intriguingly, it has been shown that deletion of the GR or exogenous glucocorticoid treatment in mice can both
