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Christina Antza
Christina Antza
Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK
Department of Diabetes and Endocrinology, University Hospitals Birmingham NHS Foundation Trust, Birmingham, UK
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Department of Diabetes and Endocrinology, University Hospitals Birmingham NHS Foundation Trust, Birmingham, UK
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Georgios Kostopoulos
Georgios Kostopoulos
Department of Endocrinology, 424 General Military Hospital, Thessaloniki, Greece
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Samiul Mostafa
Samiul Mostafa
Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK
Department of Diabetes and Endocrinology, University Hospitals Birmingham NHS Foundation Trust, Birmingham, UK
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Department of Diabetes and Endocrinology, University Hospitals Birmingham NHS Foundation Trust, Birmingham, UK
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Krishnarajah Nirantharakumar
Krishnarajah Nirantharakumar
Institute of Applied Health Research, University of Birmingham, Birmingham, UK
Centre of Endocrinology Diabetes and Metabolism, Birmingham Health Partners, Birmingham, UK
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Centre of Endocrinology Diabetes and Metabolism, Birmingham Health Partners, Birmingham, UK
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Abd Tahrani
Abd Tahrani
Institute of Metabolism and Systems Research, University of Birmingham, Birmingham, UK
Department of Diabetes and Endocrinology, University Hospitals Birmingham NHS Foundation Trust, Birmingham, UK
Centre of Endocrinology Diabetes and Metabolism, Birmingham Health Partners, Birmingham, UK
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Department of Diabetes and Endocrinology, University Hospitals Birmingham NHS Foundation Trust, Birmingham, UK
Centre of Endocrinology Diabetes and Metabolism, Birmingham Health Partners, Birmingham, UK
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(shift work, working hours, occupational stress), (17) postmenopausal hormone use, (18) metabolic rate, (19) study or institution level, (20) insulin sensitivity, (21) napping, (22) total calorie intake, (23) dietary habits. HbA1c, haemoglobin A1c
Sarah J Richardson
Sarah J Richardson
Islet Biology Group, Exeter Centre for Excellence in Diabetes (EXCEED), Institute of Biomedical and Clinical Sciences (IBCS), University of Exeter, RILD Level 4, Exeter, UK
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Alberto Pugliese
Alberto Pugliese
Division of Diabetes Endocrinology and Metabolism, Departments of Medicine, Microbiology and Immunology, Diabetes Research Institute, Leonard Miller School of Medicine, University of Miami, Miami, Florida, USA
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1D pancreas Additional pancreatic pathological abnormalities have been described and appear to contribute to the disease pathogenesis. Inflammation, beta cell stress, and dysfunction Islet inflammation induces stress, protein misfolding
Robin Kristófi
Robin Kristófi
Department of Medical Sciences, Clinical Diabetes and Metabolism, Uppsala University, Uppsala, Sweden
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Jan W Eriksson
Jan W Eriksson
Department of Medical Sciences, Clinical Diabetes and Metabolism, Uppsala University, Uppsala, Sweden
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( Sun et al. 2014 ). Another AMPK-mediated effect shown in a study of mice was the inhibition of dynamin-related protein 1 (Drp1)-mediated mitochondrial fission, which prevented stress-induced activation of the NLR family pyrin domain containing 3
Nicole G Barra
Nicole G Barra
Department of Biochemistry and Biomedical Sciences, Department of Biochemistry and Biomedical Sciences, Farncombe Family Digestive Health Research Institute, Centre for Metabolism, Obesity and Diabetes Research, McMaster University, Hamilton, Ontario, Canada
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Fernando F Anhê
Fernando F Anhê
Department of Biochemistry and Biomedical Sciences, Department of Biochemistry and Biomedical Sciences, Farncombe Family Digestive Health Research Institute, Centre for Metabolism, Obesity and Diabetes Research, McMaster University, Hamilton, Ontario, Canada
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Joseph F Cavallari
Joseph F Cavallari
Department of Biochemistry and Biomedical Sciences, Department of Biochemistry and Biomedical Sciences, Farncombe Family Digestive Health Research Institute, Centre for Metabolism, Obesity and Diabetes Research, McMaster University, Hamilton, Ontario, Canada
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Anita M Singh
Anita M Singh
Department of Biochemistry and Biomedical Sciences, Department of Biochemistry and Biomedical Sciences, Farncombe Family Digestive Health Research Institute, Centre for Metabolism, Obesity and Diabetes Research, McMaster University, Hamilton, Ontario, Canada
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Darryl Y Chan
Darryl Y Chan
Department of Biochemistry and Biomedical Sciences, Department of Biochemistry and Biomedical Sciences, Farncombe Family Digestive Health Research Institute, Centre for Metabolism, Obesity and Diabetes Research, McMaster University, Hamilton, Ontario, Canada
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Jonathan D Schertzer
Jonathan D Schertzer
Department of Biochemistry and Biomedical Sciences, Department of Biochemistry and Biomedical Sciences, Farncombe Family Digestive Health Research Institute, Centre for Metabolism, Obesity and Diabetes Research, McMaster University, Hamilton, Ontario, Canada
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Female: 55 µg Pregnant: 60 µg Lactation: 70 µg Decreased Se associated with IBD Se supplementation in broiler chickens: ↓Enteric pathogens ; mitigates Ochratoxin-induced ↓ α-diversity, oxidative stress, inflammation Deficient and excess status
Yanli Miao
Yanli Miao
Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Haojie Qin
Haojie Qin
Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Yi Zhong
Yi Zhong
Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Kai Huang
Kai Huang
Department of Cardiology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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Caijun Rao
Caijun Rao
Department of Geriatrics, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
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addition to the change of environmental temperature, many factors such as inflammation, oxidative stress and autophagy can affect the white adipose browning ( Huerta-Delgado et al. 2020 , Yamamuro et al. 2020 ). Browning of white adipose tissue is
Andre Sarmento-Cabral
Andre Sarmento-Cabral
Department of Medicine, Section of Endocrinology, Diabetes, and Metabolism, University of Illinois at Chicago and Research and Development Division, Jesse Brown VA Medical Center, Chicago, Illinois, USA
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Mercedes del Rio-Moreno
Mercedes del Rio-Moreno
Department of Medicine, Section of Endocrinology, Diabetes, and Metabolism, University of Illinois at Chicago and Research and Development Division, Jesse Brown VA Medical Center, Chicago, Illinois, USA
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Mari C Vazquez-Borrego
Mari C Vazquez-Borrego
Department of Medicine, Section of Endocrinology, Diabetes, and Metabolism, University of Illinois at Chicago and Research and Development Division, Jesse Brown VA Medical Center, Chicago, Illinois, USA
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Mariyah Mahmood
Mariyah Mahmood
Department of Medicine, Section of Endocrinology, Diabetes, and Metabolism, University of Illinois at Chicago and Research and Development Division, Jesse Brown VA Medical Center, Chicago, Illinois, USA
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Elena Gutierrez-Casado
Elena Gutierrez-Casado
Department of Medicine, Section of Endocrinology, Diabetes, and Metabolism, University of Illinois at Chicago and Research and Development Division, Jesse Brown VA Medical Center, Chicago, Illinois, USA
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Natalie Pelke
Natalie Pelke
Department of Medicine, Section of Endocrinology, Diabetes, and Metabolism, University of Illinois at Chicago and Research and Development Division, Jesse Brown VA Medical Center, Chicago, Illinois, USA
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Grace Guzman
Grace Guzman
Department of Pathology, University of Illinois at Chicago, College of Medicine, Chicago, Illinois, USA
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Papasani V Subbaiah
Papasani V Subbaiah
Department of Medicine, Section of Endocrinology, Diabetes, and Metabolism, University of Illinois at Chicago and Research and Development Division, Jesse Brown VA Medical Center, Chicago, Illinois, USA
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Jose Cordoba-Chacon
Jose Cordoba-Chacon
Department of Medicine, Section of Endocrinology, Diabetes, and Metabolism, University of Illinois at Chicago and Research and Development Division, Jesse Brown VA Medical Center, Chicago, Illinois, USA
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Shoshana Yakar
Shoshana Yakar
Department of Molecular Pathobiology, New York University College of Dentistry, New York, New York, USA
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Rhonda D Kineman
Rhonda D Kineman
Department of Medicine, Section of Endocrinology, Diabetes, and Metabolism, University of Illinois at Chicago and Research and Development Division, Jesse Brown VA Medical Center, Chicago, Illinois, USA
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prevent steatosis and subsequent liver injury. In addition, GH may act independent of steatosis to prevent liver injury. Specifically, GH-mediated STAT5 signaling regulates multiple genes critical in xenobiotic clearance, oxidative stress and cell cycle
Marion Régnier
Marion Régnier
UCLouvain, Université Catholique de Louvain, WELBIO – Walloon Excellence in Life Sciences and BIOtechnology, Louvain Drug Research Institute, Metabolism and Nutrition Research Group, Brussels, Belgium
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Matthias Van Hul
Matthias Van Hul
UCLouvain, Université Catholique de Louvain, WELBIO – Walloon Excellence in Life Sciences and BIOtechnology, Louvain Drug Research Institute, Metabolism and Nutrition Research Group, Brussels, Belgium
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Claude Knauf
Claude Knauf
Université Paul Sabatier, Toulouse III, INSERM U1220, Institut de Recherche en Santé Digestive (IRSD), CHU Purpan, Place du Docteur Baylac, Toulouse Cedex 3, France
European Associated Laboratory (EAL) ‘NeuroMicrobiota’, Brussels/Toulouse, Belgium
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European Associated Laboratory (EAL) ‘NeuroMicrobiota’, Brussels/Toulouse, Belgium
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Patrice D Cani
Patrice D Cani
UCLouvain, Université Catholique de Louvain, WELBIO – Walloon Excellence in Life Sciences and BIOtechnology, Louvain Drug Research Institute, Metabolism and Nutrition Research Group, Brussels, Belgium
European Associated Laboratory (EAL) ‘NeuroMicrobiota’, Brussels/Toulouse, Belgium
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European Associated Laboratory (EAL) ‘NeuroMicrobiota’, Brussels/Toulouse, Belgium
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regulating oxidative stress through the downregulation of reactive oxygen species (ROS). Another mechanism by which polyphenols could exert their activity is by targeting different members of the NF-κB pathway or by antagonizing its activation. This pathway
Wenjuan Liu
Wenjuan Liu
Department of Endocrinology and Metabolism, Huashan Hospital, Fudan University, Shanghai, China
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Harry Kevin Lau
Harry Kevin Lau
Division of Endocrinology and Metabolism, The Keenan Research Centre in the Li Ka Shing Knowledge Institute, St. Michael’s Hospital, Toronto, Ontario, Canada
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Dong Ok Son
Dong Ok Son
Division of Endocrinology and Metabolism, The Keenan Research Centre in the Li Ka Shing Knowledge Institute, St. Michael’s Hospital, Toronto, Ontario, Canada
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Tianru Jin
Tianru Jin
Department of Physiology, University of Toronto, Toronto, Ontario, Canada
Division of Advanced Diagnostics, Toronto General Research Institutes, University Health Network, Toronto, Ontario, Canada
Banting and Best Diabetes Centre, University of Toronto, Toronto, Ontario, Canada
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Division of Advanced Diagnostics, Toronto General Research Institutes, University Health Network, Toronto, Ontario, Canada
Banting and Best Diabetes Centre, University of Toronto, Toronto, Ontario, Canada
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Yehong Yang
Yehong Yang
Department of Endocrinology and Metabolism, Huashan Hospital, Fudan University, Shanghai, China
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Zhaoyun Zhang
Zhaoyun Zhang
Department of Endocrinology and Metabolism, Huashan Hospital, Fudan University, Shanghai, China
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Yiming Li
Yiming Li
Department of Endocrinology and Metabolism, Huashan Hospital, Fudan University, Shanghai, China
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Gerald J Prud’homme
Gerald J Prud’homme
Department of Laboratory Medicine and Pathobiology, Keenan Research Centre for Biomedical Science of St. Michael’s Hospital, University of Toronto, Toronto, Ontario, Canada
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Qinghua Wang
Qinghua Wang
Department of Endocrinology and Metabolism, Huashan Hospital, Fudan University, Shanghai, China
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investigators have proposed that β-cell dedifferentiation is a crucial pathophysiological process underlying the loss of β-cell mass ( Gershengorn et al. 2004 , Weinberg et al. 2007 , Talchai et al. 2012 ). Under metabolic or environmental stress, β
Lei Du
Lei Du
State Key Lab of Reproductive Medicine, Nanjing Medical University, Nanjing, Jiangsu, China
The Third Xiangya Hospital of Central South University, Changsha, Hunan, China
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The Third Xiangya Hospital of Central South University, Changsha, Hunan, China
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Yang Wang
Yang Wang
State Key Lab of Reproductive Medicine, Nanjing Medical University, Nanjing, Jiangsu, China
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Cong-Rong Li
Cong-Rong Li
State Key Lab of Reproductive Medicine, Nanjing Medical University, Nanjing, Jiangsu, China
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Liang-Jian Chen
Liang-Jian Chen
State Key Lab of Reproductive Medicine, Nanjing Medical University, Nanjing, Jiangsu, China
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Jin-Yang Cai
Jin-Yang Cai
State Key Lab of Reproductive Medicine, Nanjing Medical University, Nanjing, Jiangsu, China
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Zheng-Rong Xia
Zheng-Rong Xia
Analysis and Test Center, Nanjing Medical University, Nanjing, Jiangsu, China
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Wen-Tao Zeng
Wen-Tao Zeng
Animal Core Facility, Nanjing Medical University, Nanjing, Jiangsu, China
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Zi-Bin Wang
Zi-Bin Wang
Analysis and Test Center, Nanjing Medical University, Nanjing, Jiangsu, China
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Xi-Chen Chen
Xi-Chen Chen
Analysis and Test Center, Nanjing Medical University, Nanjing, Jiangsu, China
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Fan Hu
Fan Hu
State Key Lab of Reproductive Medicine, Nanjing Medical University, Nanjing, Jiangsu, China
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Dong Zhang
Dong Zhang
State Key Lab of Reproductive Medicine, Nanjing Medical University, Nanjing, Jiangsu, China
Animal Core Facility, Nanjing Medical University, Nanjing, Jiangsu, China
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Animal Core Facility, Nanjing Medical University, Nanjing, Jiangsu, China
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Xiao-Wei Xing
Xiao-Wei Xing
The Third Xiangya Hospital of Central South University, Changsha, Hunan, China
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Zhi-Xia Yang
Zhi-Xia Yang
State Key Lab of Reproductive Medicine, Nanjing Medical University, Nanjing, Jiangsu, China
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oxidative stress via PI3K/AKT/NF-κB signaling in the granulosa cells of PCOS patients . Journal of Clinical Endocrinology and Metabolism 100 201 – 211 . ( https://doi.org/10.1210/jc.2014-2419 )
Prashant Regmi
Prashant Regmi
Adelaide Medical School, University of Adelaide, Adelaide, South Australia, Australia
Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Rajesh Chaudhary
Rajesh Chaudhary
Adelaide Medical School, University of Adelaide, Adelaide, South Australia, Australia
Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Amanda J Page
Amanda J Page
Adelaide Medical School, University of Adelaide, Adelaide, South Australia, Australia
Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Amy T Hutchison
Amy T Hutchison
Adelaide Medical School, University of Adelaide, Adelaide, South Australia, Australia
Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Andrew D Vincent
Andrew D Vincent
Adelaide Medical School, University of Adelaide, Adelaide, South Australia, Australia
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Bo Liu
Bo Liu
Adelaide Medical School, University of Adelaide, Adelaide, South Australia, Australia
Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Leonie Heilbronn
Leonie Heilbronn
Adelaide Medical School, University of Adelaide, Adelaide, South Australia, Australia
Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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Lifelong Health Theme, South Australian Health and Medical Research Institute (SAHMRI), Adelaide, South Australia, Australia
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, blood pressure, and oxidative stress even without weight loss in men with prediabetes . Cell Metabolism 27 1212 .e3 – 1221.e3 . ( https://doi.org/10.1016/j.cmet.2018.04.010 ) Tschop MH Speakman JR Arch JR Auwerx J Bruning JC Chan L
