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( Schwartges et al . 2010 ). Apart from these effects on macro- and microcirculation, HC exerts multiple other changes in homeostasis, like an increase in vasopressin plasma level ( Philbin et al . 1970 , Forsling & Rees 1975 , Farber et al . 1982 , Rose
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Institute of Experimental Medicine, Institut für Biochemie and Zellbiologie, Centre for Behavioral Brain Sciences, Hungarian Academy of Sciences, Szigony 43, 1083 Budapest, Hungary
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level are the corticotropin-releasing hormone (CRH) and arginine vasopressin (AVP). Both neuropeptides are synthesized in and released from the parvocellular neurons of the hypothalamic paraventricular nucleus (PVN; Aguilera 1994 ). After being released
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neurohypophysis in the milk ejection reflex. Journal of Endocrinology 8 148–161. (C) The amount of oxytocin (and vasopressin) that is released from the rat posterior pituitary gland in vitro in response to a fixed number of electrical stimulus pulses varies
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Department of Physiology, Faculty of Medicine, University of Malaya, Kuala Lumpur, Malaysia
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experimental model extensively used to study these responses. WD increases osmolality and decreases plasma volume, inducing the release of hormones that act to maintain homeostasis, such as vasopressin (AVP) and oxytocin (OT), and the activation of the renin
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intergenerational calcium transfer. Further insight is needed to clarify the role of PRL in bone metabolism and determine the cellular pathways. AVP and bone loss in hyponatremia There are abundant receptors for arginine vasopressin (AVP), namely AVPR1α and
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Department of Applied Biology, Department of Anatomy and Neuroscience, Kyoto Institute of Technology, Matsugasaki, Sakyo-ku, Kyoto 606-8585, Japan
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arginine vasopressin (AVP) and oxytocin (OXT) into the blood circulation from their axonal terminals ( Miyata & Hatton 2002 ). Similarly, they release a variety of neuropeptides into the blood circulation at the median eminence (ME) to control secretion of
Department of Genomic Drug Discovery Science, Graduate School of Pharmaceutical Sciences, Kyoto University Faculty of Pharmaceutical Sciences, Kyoto University, Yoshida Shimoadachi-cho, Sakyo-ku, Kyoto 606-8501, Japan
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Department of Genomic Drug Discovery Science, Graduate School of Pharmaceutical Sciences, Kyoto University Faculty of Pharmaceutical Sciences, Kyoto University, Yoshida Shimoadachi-cho, Sakyo-ku, Kyoto 606-8501, Japan
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Department of Genomic Drug Discovery Science, Graduate School of Pharmaceutical Sciences, Kyoto University Faculty of Pharmaceutical Sciences, Kyoto University, Yoshida Shimoadachi-cho, Sakyo-ku, Kyoto 606-8501, Japan
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Department of Genomic Drug Discovery Science, Graduate School of Pharmaceutical Sciences, Kyoto University Faculty of Pharmaceutical Sciences, Kyoto University, Yoshida Shimoadachi-cho, Sakyo-ku, Kyoto 606-8501, Japan
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Department of Genomic Drug Discovery Science, Graduate School of Pharmaceutical Sciences, Kyoto University Faculty of Pharmaceutical Sciences, Kyoto University, Yoshida Shimoadachi-cho, Sakyo-ku, Kyoto 606-8501, Japan
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Department of Genomic Drug Discovery Science, Graduate School of Pharmaceutical Sciences, Kyoto University Faculty of Pharmaceutical Sciences, Kyoto University, Yoshida Shimoadachi-cho, Sakyo-ku, Kyoto 606-8501, Japan
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Introduction Arginine-vasopressin (AVP) and oxytocin (OT) are neurohypophysial hormones synthesized in the paraventricular nucleus and supraoptic nucleus of the hypothalamus. AVP acts in many organs and plays a variety of
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Introduction Arginine–vasopressin (AVP), an antidiuretic hormone, is synthesized by magnocellular neurosecretory cells (MNCs) in the paraventricular nuclei (PVN) and supraoptic nuclei (SON) of the hypothalamus and subsequently released from
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peripheral and central origin to inhibit not only water drinking, but also sodium appetite and vasopressin secretion. Here, we sought to determine if the antidipsogenic effect of obestatin could be extended to hypovolemia-induced thirst and salt appetite and
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vasopressin (AVP) plays a major role in body fluid regulation via V 2 receptors in the renal collecting ducts, and altered regulation of AVP is a major factor in developing hyponatremia ( Miller 2006 ). Thus, V 2 receptor antagonism is effective to correct