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Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands
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Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands
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Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands
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Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands
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Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands
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Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands
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Einthoven Laboratory for Experimental Vascular Medicine, Leiden University Medical Center, Leiden, The Netherlands
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transcriptional coregulator proteins. As the factors that determine GR transcriptional output are highly context dependent, genomic GC action is strongly tissue specific. Likewise, the efficacy of GR antagonists can depend on context-specific pharmacokinetics and
Centre for Neuroendocrinology and Department of Anatomy, Maurice Wilkins Centre for Molecular Biodiscovery, University of Otago, PO Box 913, Dunedin 9054, New Zealand
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impaired in mice lacking the transcription factor STAT5b ( Grattan et al . 2001 ), likely through impairment in the long-term regulation of expression of the rate limiting enzyme in dopamine synthesis, tyrosine hydroxylase ( Arbogast & Voogt 1991 a , Ma
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analyzing levels of mRNA within a tissue, studies are open to the criticism that variations in transcription may not feed through to differences in enzyme level, or that the real limiting factor in the pathway may lie elsewhere. An elegant study by Gomez
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transcriptional activity of the wildtype MR in murine renal cells ( Lema et al. 2017 ). More studies are therefore required to determine if such differences are cell- or species-specific, or a factor of circumstance ( Tables 1 , 2 , 3 and 4 ). Figure 2
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Faculty of Pharmacy, King Khalid University, Abha, Saudi Arabia
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Imperial College London, London, UK
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Department of Biochemistry, University of São Paulo, São Paulo, Brazil
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might determine disease onset and clinical progression in MODY patients. Mutations in the PDX1 gene account for the well-characterized MODY4 subtype ( Stoffers et al. 1997 , Ahlgren et al. 1998 ). PDX1 is a transcription factor critically involved
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-releasing hormone (GnRH) and gonadal steroids. Despite these common regulators, divergences in the circulating patterns of LH and FSH are often encountered, suggesting that these regulators act differentially on the production of LH and FSH and/or that other factors
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-binding protein (SREBP) is a transcription factor that binds to the sterol regulatory element and regulates the expression of lipogenic enzymes, such as stearoyl-CoA desaturase-1, FAS, and ACC1 ( Brown & Goldstein 1997 , Postic & Girard 2008 ). Prolonged
Department of Pediatrics, University of Arkansas for Medical Sciences, Little Rock, Arkansas, USA
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Department of Pediatrics, University of Arkansas for Medical Sciences, Little Rock, Arkansas, USA
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Department of Pediatrics, University of Arkansas for Medical Sciences, Little Rock, Arkansas, USA
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Department of Pediatrics, University of Arkansas for Medical Sciences, Little Rock, Arkansas, USA
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with activation of hundreds of transcription factors including nuclear receptors such as PPARγ. We found that PPARγ expression was increased in embryonic calvarial osteo-progenitor cells from HFD-induced obese dams ( Fig. 2A ). To determine whether HFD
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Safe S 1999 Transcriptional activation of insulin-like growth factor-binding protein-4 by 17beta-estradiol in MCF-7 cells: role of estrogen receptor-Sp1 complexes . Endocrinology 140 2501 – 2508 . ( doi:10.1210/en.140.6.2501 ) Raykha C
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extracellular matrix and on the cell surface ( Goldoni & Iozzo 2008 , Seidler & Dreier 2008 ). Decorin has been recently reported to sequester multiple growth factors, such as transforming growth factor β1 (TGFβ1), and directly antagonizes several members of