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receptor transactivation, c-fos expression, adenylyl cyclase and cAMP mediated signalling and ERK-1/2 in a variety of cell types ( Prossnitz et al . 2008 ). Intriguingly, only a small fraction of total cellular GPR30 appears to be expressed on the cell
Department of Animal Sciences, Department of Molecular and Cellular Physiology, University of Arizona, Tucson, Arizona 85721, USA
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positively coupled to adenylyl cyclase through G s , and has been detected in both human and mouse mammary epithelial cells on the basolateral side of the membrane ( Stull et al . 2007 ). We also detected HTR7 mRNA in BMT and BMEC ( Fig. 2 ). Additionally
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Department of Physiology, Institute of Neuroscience and Physiology, Metabolic Research Unit, University of Gothenburg, Göteborg, Sweden
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CNC - Center for Neuroscience and Cell Biology, CIBB - Centre for Innovative Biomedicine and Biotechnology, University of Coimbra, Coimbra, Portugal
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intracellular metabolites (i.e. ATP and cAMP). CFTR-mediated Cl- efflux regulates the membrane potential through an intrinsic α-cell effect, also resulting in the inhibition of glucagon secretion. AC, adenylyl cyclase; FFA, free fatty acids; CPT1a, carnitine
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Resveratrol activates adenylyl-cyclase in human breast cancer cells: a novel, estrogen receptor-independent cytostatic mechanism . Carcinogenesis 24 869 – 873 . ( doi:10.1093/carcin/bgg015 ) Fan X Zhang C Liu D Yan J Liang H 2013 The
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secretion in chromaffin cells. Cis and trans signaling determinants of pituitary adenylyl cyclase-activating polypeptide (PACAP) . Journal of Clinical Investigation 101 863 – 876 . ( doi:10.1172/JCI1129 ) Tomaszek A Kiczak L Bania J
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Centre of Membrane Proteins and Receptors (COMPARE), Universities of Birmingham and Nottingham, Birmingham, UK
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mobilisation was not dependent on signalling by Gα s , adenylyl cyclase-protein kinase A, Gα i /o-Gβγ, or protein kinase C, but did require PLC; and GHSR1a constitutive activity was not required as GHSR1a-F279L and -A204E did not prevent DRD1 non
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. 2013 ). The stimulated glucagon receptor couples to G proteins which activate adenylyl cyclase (AC) activity to produce cAMP. In rats, increasing age was associated with a decrease in glucagon-stimulated cAMP production, alongside a reduction in
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contributing to the stimulation of lipolysis. The cAMP pathway plays a crucial role in GIP-induced lipolysis, as demonstrated by the use of an adenylyl cyclase inhibitor MDL 12330A (10 −4 M), which reduced cAMP and glycerol levels in the presence of GIP (0
Centre for Neuroendocrinology and Department of Anatomy, Maurice Wilkins Centre for Molecular Biodiscovery, University of Otago, PO Box 913, Dunedin 9054, New Zealand
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.1002/ajhb.20907 ) Valerio A Alberici A Tinti C Spano P Memo M 1994 Antisense strategy unravels a dopamine receptor distinct from the D2 subtype, uncoupled with adenylyl cyclase, inhibiting prolactin release from rat pituitary cells . Journal
