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S Schmidt, A Hommel, V Gawlik, R Augustin, N Junicke, S Florian, M Richter, D J Walther, D Montag, H-G Joost and A Schürmann

concentration. We cultivated blastocysts in the presence of 4.5 g glucose/l, which might alter the rate of glycolysis and thereby GLUT1 expression and/or localization. Several parameters, for example, oxygen concentration ( Harvey et al . 2004 ), modify GLUT1

Open access

Kunihisa Hamano, Yuko Nakagawa, Yoshiaki Ohtsu, Longfei Li, Johan Medina, Yuji Tanaka, Katsuyoshi Masuda, Mitsuhisa Komatsu and Itaru Kojima

activation of the glucose-sensing receptor led to an increase in [ATP] c by facilitating glucose metabolism ( Nakagawa et al . 2014 ). Accordingly, artificial sweeteners increase [ATP] c even though they do not serve as substrates for glycolysis. We next

Open access

Saeed Alshahrani, Mohammed Mashari Almutairi, Shams Kursan, Eduardo Dias-Junior, Mohamed Mahmoud Almiahuob, Lydia Aguilar-Bryan and Mauricio Di Fulvio

: facilitated diffusion of glucose into β-cells, phosphorylation of the sugar and consumption via glycolysis, increased ATP/ADP ratio, closure of ATP-sensitive K + (K ATP )-channels, plasma membrane depolarization, increased Ca 2+ influx due to opening of

Open access

Valentina Pampanini, Daniela Germani, Antonella Puglianiello, Jan-Bernd Stukenborg, Ahmed Reda, Iuliia Savchuk, Kristín Rós Kjartansdóttir, Stefano Cianfarani and Olle Söder

substrates. To do this, SCs internalize glucose from the interstitial space via GLUT-1 and GLUT-3 glucose transporters ( Galardo et al . 2008 ) and use it to produce lactate via glycolysis pathway. Lactate produced from SCs is the main energy source for