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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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Department of Diabetes, Endocrinology and Nutrition, Hospital de Girona ‘Dr Josep Trueta’, Institut D’investigació Biomèdica de Girona (IdIBGi) and University of Girona, Girona, Spain
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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Department of Clinical Science, KG Jebsen Center for Diabetes Research, University of Bergen, Bergen, Norway
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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Department of Diabetes, Endocrinology and Nutrition, Hospital de Girona ‘Dr Josep Trueta’, Institut D’investigació Biomèdica de Girona (IdIBGi) and University of Girona, Girona, Spain
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CIBER Fisiopatología de la Obesidad y Nutrición (CIBERobn), Santiago de Compostela, Spain
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Introduction Thyroid hormones (THs; 3,3′,5,5′ tetraiodothyroxyne or T 4 and 3,3′,5-triiodothyronine or T 3 ) exert important biological actions, not only modulating the development and growth but also regulating metabolism and energy balance
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thyroid hormones. Studies in rats on the existence of a daily oscillating pattern of thyroid hormones in the circulation have, however, not been quite unequivocal, although most favour a diurnal peak of thyroxin (T4) and triiodothyronine (T3) during the
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School of Clinical and Experimental Medicine, Department of Pathology, Fetal Medicine Centre, College of Medical and Dental Sciences, University of Birmingham, Edgbaston, Birmingham B15 2TT, UK
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al . 1998 ) and decreased expression of cerebral thyroid hormone receptor (TR) expression ( Kilby et al . 2000 ) in growth-restricted human fetuses are postulated to contribute to this neurodevelopmental morbidity. Examination of growth
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Introduction Thyroid hormone (TH) has a profound influence on normal development, differentiation and metabolism. Genomic actions of THs are mainly mediated and regulated by thyroid hormone receptors (THRs) ( Cheng et al. 2010 , Cioffi et
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paradox. How might a pituitary target site be involved in remodelling of hypothalamic neuroendocrine circuits – the hallmark of the seasonal response? The answer to this lies in the remarkable discovery of thyroid hormones (THs) as key seasonal switches
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Laboratório de Fisiologia Endócrina Doris Rosenthal, Laboratório de Biologia do Exercício, Instituto de Biofísica Carlos Chagas Filho and Instituto de Pesquisa Translacional em Saúde e Ambiente na Região Amazônica (INPeTAM), CCS-Bloco G- Cidade Universitria, Ilha do Fundo, Rio de Janeiro 21949-900, Brazil
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-genomic effects of thyroid hormones on the resting metabolic rate (RMR) and oxygen consumption ( Lanni et al . 1996 , Moreno et al . 2002 ). Compound 3,5-T2 by itself increases the rate of oxygen consumption in perfused rat liver ( Horst et al . 1989 ), blood
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Introduction Central congenital hypothyroidism (CCH) is a rare disorder in which inadequate thyroid hormone biosynthesis occurs due to defective stimulation of a normal thyroid gland by thyroid stimulating hormone (TSH). The underlying molecular
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The Ritchie Centre, Hudson Institute of Medical Research, Clayton, Australia
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Institute for Women’s Health, University College London, London, UK
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Department of Biological and Medical Sciences, Oxford Brookes University, Oxford, UK
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-assay coefficients of variation for the cortisol assay were 3 and 5%, respectively, and the limit of detection was 5.2 ng/mL. Because cortisol increases fetal T 3 concentrations towards term and thyroid hormones are known to affect O 2 consumption by fetal tissues
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The Ritchie Centre, Hudson Institute of Medical Research, Clayton, Australia
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Department of Biological and Medical Sciences, Oxford Brookes University, Oxford, UK
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affect fetal concentrations of the thyroid hormones and leptin ( Forhead et al. 2007 , Fowden & Forhead 2015 ). In turn, these hormones have actions on metabolism, tissue differentiation and the development of other endocrine systems, including the
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Tg/mouse) was injected into the soleus muscle once every 2 weeks. All immunizations were performed in the presence of complete Freund's adjuvant. 125 I uptake and measurement of thyroid hormones 125 I–Na (3.7 GBq/ml) was obtained from GE Healthcare