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Xiong Weng
Xiong Weng
Division of Cellular and Systems Medicine, School of Medicine, University of Dundee, Dundee, Scotland, UK
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Hao Jiang
Hao Jiang
Gene Expression and Regulation, School of Life Sciences, University of Dundee, Dundee, Scotland, UK
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David J Walker
David J Walker
Division of Cellular and Systems Medicine, School of Medicine, University of Dundee, Dundee, Scotland, UK
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Houjiang Zhou
Houjiang Zhou
MRC Protein Phosphorylation Unit, School of Life Sciences, Dundee, Scotland, UK
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De Lin
De Lin
Drug Discovery Unit, School of Life Sciences, University of Dundee, Dundee, Scotland, UK
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Jing Wang
Jing Wang
Science for Life Laboratory, Department of Biomedical and Clinical Sciences (BKV), Linköping University, Linköping, Sweden
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Li Kang
Li Kang
Division of Cellular and Systems Medicine, School of Medicine, University of Dundee, Dundee, Scotland, UK
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pathogenesis of T2D by a gene expression-based genome-wide association study ( Kodama et al . 2012 ). Ablation of CD44 by genetic deletion or anti-CD44 monoclonal antibody administration reduced adipose tissue immune cell infiltration and insulin resistance in
Ping Ye
Ping Ye
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Christopher J Kenyon
Christopher J Kenyon
MRC Blood Pressure Group, The Queen's Medical Research Institute, Division of Cardiovascular and Medical Sciences, BHF Glasgow Cardiovascular Research Centre, 126 University Place, Glasgow G12 8TA, UK
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Scott M MacKenzie
Scott M MacKenzie
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Katherine Nichol
Katherine Nichol
MRC Blood Pressure Group, The Queen's Medical Research Institute, Division of Cardiovascular and Medical Sciences, BHF Glasgow Cardiovascular Research Centre, 126 University Place, Glasgow G12 8TA, UK
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Jonathan R Seckl
Jonathan R Seckl
MRC Blood Pressure Group, The Queen's Medical Research Institute, Division of Cardiovascular and Medical Sciences, BHF Glasgow Cardiovascular Research Centre, 126 University Place, Glasgow G12 8TA, UK
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Robert Fraser
Robert Fraser
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John M C Connell
John M C Connell
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Eleanor Davies
Eleanor Davies
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2003 ). Although these extra-adrenal sites are not capable of corticosteroid production on the same scale as the adrenal cortex, due to a much lower level of relevant gene expression, their proximity to glucocorticoid receptors (GR) and
Koichiro Taguchi
Koichiro Taguchi
Department of General Internal Medicine, Gifu University Graduate School of Medicine, Gifu, Japan
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Kazuo Kajita
Kazuo Kajita
Department of General Internal Medicine, Gifu University Graduate School of Medicine, Gifu, Japan
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Yoshihiko Kitada
Yoshihiko Kitada
Department of General Internal Medicine, Gifu University Graduate School of Medicine, Gifu, Japan
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Masayuki Fuwa
Masayuki Fuwa
Department of General Internal Medicine, Gifu University Graduate School of Medicine, Gifu, Japan
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Motochika Asano
Motochika Asano
Department of General Internal Medicine, Gifu University Graduate School of Medicine, Gifu, Japan
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Takahide Ikeda
Takahide Ikeda
Department of General Internal Medicine, Gifu University Graduate School of Medicine, Gifu, Japan
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Toshiko Kajita
Toshiko Kajita
Department of General Internal Medicine, Gifu University Graduate School of Medicine, Gifu, Japan
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Tatsuo Ishizuka
Tatsuo Ishizuka
Department of General Internal Medicine and Rheumatology, Gifu Municipal Hospital, Gifu, Japan
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Itaru Kojima
Itaru Kojima
Laboratory of Cell Physiology, Institute for Molecular and Cellular Regulation, Gunma University, Gunma, Japan
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Hiroyuki Morita
Hiroyuki Morita
Department of General Internal Medicine, Gifu University Graduate School of Medicine, Gifu, Japan
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unable to isolate SPA. Subsequently, we developed a new method to isolate SPA and in this way the existence of SPA became more certain. In this study, we investigated for the first time the characteristics of SPA in terms of their gene expression and
K E Lines
K E Lines
Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism (OCDEM), Churchill Hospital, Headington, Oxford, UK
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P J Newey
P J Newey
Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism (OCDEM), Churchill Hospital, Headington, Oxford, UK
Division of Molecular & Clinical Medicine, University of Dundee, Ninewells Hospital & Medical School, Dundee, UK
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Division of Molecular & Clinical Medicine, University of Dundee, Ninewells Hospital & Medical School, Dundee, UK
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C J Yates
C J Yates
Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism (OCDEM), Churchill Hospital, Headington, Oxford, UK
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M Stevenson
M Stevenson
Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism (OCDEM), Churchill Hospital, Headington, Oxford, UK
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R Dyar
R Dyar
Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism (OCDEM), Churchill Hospital, Headington, Oxford, UK
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G V Walls
G V Walls
Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism (OCDEM), Churchill Hospital, Headington, Oxford, UK
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M R Bowl
M R Bowl
Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism (OCDEM), Churchill Hospital, Headington, Oxford, UK
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R V Thakker
R V Thakker
Academic Endocrine Unit, Radcliffe Department of Medicine, University of Oxford, Oxford Centre for Diabetes, Endocrinology and Metabolism (OCDEM), Churchill Hospital, Headington, Oxford, UK
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), which are short, non-coding, single-stranded RNAs that post-transcriptionally regulate gene expression, predominantly by imperfect base pairing to the 3′ untranslated region (UTR) of target mRNA sequences ( Filipowicz et al. 2008 , Stefani & Slack
Nikolaos Nikolaou
Nikolaos Nikolaou
Oxford Centre for Diabetes, Endocrinology and Metabolism, NIHR Oxford Biomedical Research Centre, University of Oxford, Churchill Hospital, Oxford, UK
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Anastasia Arvaniti
Anastasia Arvaniti
Oxford Centre for Diabetes, Endocrinology and Metabolism, NIHR Oxford Biomedical Research Centre, University of Oxford, Churchill Hospital, Oxford, UK
Department of Biological and Medical Sciences, Oxford Brookes University, Oxford, UK
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Department of Biological and Medical Sciences, Oxford Brookes University, Oxford, UK
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Nathan Appanna
Nathan Appanna
Oxford Centre for Diabetes, Endocrinology and Metabolism, NIHR Oxford Biomedical Research Centre, University of Oxford, Churchill Hospital, Oxford, UK
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Anna Sharp
Anna Sharp
Oxford Centre for Diabetes, Endocrinology and Metabolism, NIHR Oxford Biomedical Research Centre, University of Oxford, Churchill Hospital, Oxford, UK
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Beverly A Hughes
Beverly A Hughes
Institute of Metabolism and Systems Research, University of Birmingham, Edgbaston, Birmingham, UK
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Dena Digweed
Dena Digweed
Diurnal Ltd, Cardiff, UK
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Martin J Whitaker
Martin J Whitaker
Diurnal Ltd, Cardiff, UK
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Richard Ross
Richard Ross
Department of Oncology and Metabolism, Faculty of Medicine, Dentistry and Health, University of Sheffield, Sheffield, UK
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Wiebke Arlt
Wiebke Arlt
Institute of Metabolism and Systems Research, University of Birmingham, Edgbaston, Birmingham, UK
NIHR Birmingham Biomedical Research Centre, University Hospitals Birmingham NHS Foundation Trust and University of Birmingham, Birmingham, UK
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NIHR Birmingham Biomedical Research Centre, University Hospitals Birmingham NHS Foundation Trust and University of Birmingham, Birmingham, UK
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Trevor M Penning
Trevor M Penning
Department of Systems Pharmacology & Translational Therapeutics, University of Pennsylvania Perelman School of Medicine, Philadelphia, Pennsylvania, USA
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Karen Morris
Karen Morris
Biochemistry Department, Manchester University NHS Trust, Manchester, UK
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Sherly George
Sherly George
Biochemistry Department, Manchester University NHS Trust, Manchester, UK
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Brian G Keevil
Brian G Keevil
Biochemistry Department, Manchester University NHS Trust, Manchester, UK
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Leanne Hodson
Leanne Hodson
Oxford Centre for Diabetes, Endocrinology and Metabolism, NIHR Oxford Biomedical Research Centre, University of Oxford, Churchill Hospital, Oxford, UK
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Laura L Gathercole
Laura L Gathercole
Oxford Centre for Diabetes, Endocrinology and Metabolism, NIHR Oxford Biomedical Research Centre, University of Oxford, Churchill Hospital, Oxford, UK
Department of Biological and Medical Sciences, Oxford Brookes University, Oxford, UK
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Department of Biological and Medical Sciences, Oxford Brookes University, Oxford, UK
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Jeremy W Tomlinson
Jeremy W Tomlinson
Oxford Centre for Diabetes, Endocrinology and Metabolism, NIHR Oxford Biomedical Research Centre, University of Oxford, Churchill Hospital, Oxford, UK
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regarding the role of AKR1D1 in GC metabolism, specificially with regard to regulation of AKR1D1 expression and activity by GCs, the capacity of AKR1D1 to metabolise synthetic steroids and its role in the regulation of established GC target genes. There is a
Karen Piper Hanley
Karen Piper Hanley
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Tom Hearn
Tom Hearn
Endocrinology and Diabetes Group, Human Genetics Division, Beta-Cell Development and Function Group, Peninsula Medical School, Manchester Academic Health Science Centre, University of Manchester, A V Hill Building, Oxford Road, Manchester M13 9PT, UK
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Andrew Berry
Andrew Berry
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Melanie J Carvell
Melanie J Carvell
Endocrinology and Diabetes Group, Human Genetics Division, Beta-Cell Development and Function Group, Peninsula Medical School, Manchester Academic Health Science Centre, University of Manchester, A V Hill Building, Oxford Road, Manchester M13 9PT, UK
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Ann-Marie Patch
Ann-Marie Patch
Endocrinology and Diabetes Group, Human Genetics Division, Beta-Cell Development and Function Group, Peninsula Medical School, Manchester Academic Health Science Centre, University of Manchester, A V Hill Building, Oxford Road, Manchester M13 9PT, UK
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Louise J Williams
Louise J Williams
Endocrinology and Diabetes Group, Human Genetics Division, Beta-Cell Development and Function Group, Peninsula Medical School, Manchester Academic Health Science Centre, University of Manchester, A V Hill Building, Oxford Road, Manchester M13 9PT, UK
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Sarah A Sugden
Sarah A Sugden
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David I Wilson
David I Wilson
Endocrinology and Diabetes Group, Human Genetics Division, Beta-Cell Development and Function Group, Peninsula Medical School, Manchester Academic Health Science Centre, University of Manchester, A V Hill Building, Oxford Road, Manchester M13 9PT, UK
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Sian Ellard
Sian Ellard
Endocrinology and Diabetes Group, Human Genetics Division, Beta-Cell Development and Function Group, Peninsula Medical School, Manchester Academic Health Science Centre, University of Manchester, A V Hill Building, Oxford Road, Manchester M13 9PT, UK
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Neil A Hanley
Neil A Hanley
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instance, Pdx1 regulates GLUT2 , glucokinase and insulin amyloid polypeptide ( IAPP ) expression ( McKinnon & Docherty 2001 ) and, in association with NeuroD1, it transactivates the insulin gene ( Babu et al . 2008 ). Many aspects of the β
Qinglei Yin
Qinglei Yin
Shanghai National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Institute of EndocrineRuijin Hospital, Shanghai Jiao-Tong University School of Medicine, China
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Liyun Shen
Liyun Shen
Shanghai National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Institute of EndocrineRuijin Hospital, Shanghai Jiao-Tong University School of Medicine, China
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Yicheng Qi
Yicheng Qi
Division of Endocrinology and Metabolism, Department of Internal Medicine, RenJi Hospital, Shanghai Jiao-Tong University School of Medicine, Pudong, Shanghai, China
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Dalong Song
Dalong Song
Reproductive Medicine Center, Guangdong Provincial People’s Hospital, Guangdong Academy of Medical Science, Guangzhou, China
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Lei Ye
Lei Ye
Shanghai National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Institute of EndocrineRuijin Hospital, Shanghai Jiao-Tong University School of Medicine, China
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Ying Peng
Ying Peng
Shanghai National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Institute of EndocrineRuijin Hospital, Shanghai Jiao-Tong University School of Medicine, China
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Yanqiu Wang
Yanqiu Wang
Shanghai National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Institute of EndocrineRuijin Hospital, Shanghai Jiao-Tong University School of Medicine, China
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Zhou Jin
Zhou Jin
Shanghai National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Institute of EndocrineRuijin Hospital, Shanghai Jiao-Tong University School of Medicine, China
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Guang Ning
Guang Ning
Shanghai National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Institute of EndocrineRuijin Hospital, Shanghai Jiao-Tong University School of Medicine, China
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Weiqing Wang
Weiqing Wang
Shanghai National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Institute of EndocrineRuijin Hospital, Shanghai Jiao-Tong University School of Medicine, China
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Dongping Lin
Dongping Lin
Department of Endocrinology and Metabolism, Shanghai Ninth People’s Hospital, Affiliated Shanghai Jiao-Tong University School of Medicine, Shanghai, China
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Shu Wang
Shu Wang
Shanghai National Clinical Research Center for Endocrine and Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of the National Health Commission of the PR China, Shanghai Institute of EndocrineRuijin Hospital, Shanghai Jiao-Tong University School of Medicine, China
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to mediate gene transcription ( Lawrence 2009 ). NF-κΒ signaling is modulated by posttranslational modifications (PTMs) ( Perkins 2006 ). PTMs are integral components of gene expression programs. To date, >200 different PTMs have been identified that
Ying Sze
Ying Sze
Centre for Discovery Brain Sciences, University of Edinburgh, Edinburgh, UK
The Roslin Institute, University of Edinburgh, Easter Bush Campus, Midlothian, UK
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The Roslin Institute, University of Edinburgh, Easter Bush Campus, Midlothian, UK
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Joana Fernandes
Joana Fernandes
The Roslin Institute, University of Edinburgh, Easter Bush Campus, Midlothian, UK
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Zofia M Kołodziejczyk
Zofia M Kołodziejczyk
Centre for Discovery Brain Sciences, University of Edinburgh, Edinburgh, UK
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Paula J Brunton
Paula J Brunton
Centre for Discovery Brain Sciences, University of Edinburgh, Edinburgh, UK
The Roslin Institute, University of Edinburgh, Easter Bush Campus, Midlothian, UK
Zhejiang University-University of Edinburgh Institute, International Campus, Haining, Zhejiang, P.R. China
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The Roslin Institute, University of Edinburgh, Easter Bush Campus, Midlothian, UK
Zhejiang University-University of Edinburgh Institute, International Campus, Haining, Zhejiang, P.R. China
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stores to effectively deal with the stressor. Given glucocorticoids have wide-ranging effects on gene expression, metabolism and neural function, excess or inappropriate glucocorticoid action can be detrimental ( Sapolsky et al. 2000 ). Therefore, HPA
F Wahab
F Wahab
Stem Cell Biology Unit, Laboratory of Reproductive Neuroendocrinology, Leibniz Institute for Primate Research, German Primate Center, Kellnerweg 4, D-37077 Göttingen, Germany
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M Shahab
M Shahab
Stem Cell Biology Unit, Laboratory of Reproductive Neuroendocrinology, Leibniz Institute for Primate Research, German Primate Center, Kellnerweg 4, D-37077 Göttingen, Germany
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R Behr
R Behr
Stem Cell Biology Unit, Laboratory of Reproductive Neuroendocrinology, Leibniz Institute for Primate Research, German Primate Center, Kellnerweg 4, D-37077 Göttingen, Germany
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noteworthy influence of lactation on the GnRH neuronal sensitivity to KP in terms of gonadotropin secretion was reported. Altogether, these results suggest that an increase in expression of the Rfrp gene during lactation may cause higher synthesis and
Valentina Pampanini
Valentina Pampanini
Department of Women’s and Children’s Health, Pediatric Endocrinology Unit, Q2:08, Karolinska Institutet and University Hospital, Stockholm, Sweden
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Daniela Germani
Daniela Germani
Department of Systems Medicine, Tor Vergata University, Rome, Italy
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Antonella Puglianiello
Antonella Puglianiello
Department of Systems Medicine, Tor Vergata University, Rome, Italy
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Jan-Bernd Stukenborg
Jan-Bernd Stukenborg
Department of Women’s and Children’s Health, Pediatric Endocrinology Unit, Q2:08, Karolinska Institutet and University Hospital, Stockholm, Sweden
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Ahmed Reda
Ahmed Reda
Department of Women’s and Children’s Health, Pediatric Endocrinology Unit, Q2:08, Karolinska Institutet and University Hospital, Stockholm, Sweden
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Iuliia Savchuk
Iuliia Savchuk
Department of Women’s and Children’s Health, Pediatric Endocrinology Unit, Q2:08, Karolinska Institutet and University Hospital, Stockholm, Sweden
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Kristín Rós Kjartansdóttir
Kristín Rós Kjartansdóttir
Department of Women’s and Children’s Health, Pediatric Endocrinology Unit, Q2:08, Karolinska Institutet and University Hospital, Stockholm, Sweden
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Stefano Cianfarani
Stefano Cianfarani
Department of Women’s and Children’s Health, Pediatric Endocrinology Unit, Q2:08, Karolinska Institutet and University Hospital, Stockholm, Sweden
Dipartimento Pediatrico Universitario Ospedaliero ‘Bambino Gesù’ Children’s Hospital – Tor Vergata University, Rome, Italy
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Dipartimento Pediatrico Universitario Ospedaliero ‘Bambino Gesù’ Children’s Hospital – Tor Vergata University, Rome, Italy
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Olle Söder
Olle Söder
Department of Women’s and Children’s Health, Pediatric Endocrinology Unit, Q2:08, Karolinska Institutet and University Hospital, Stockholm, Sweden
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environment can influence the expression of fetal genes and has an impact on growth and development of multiple organs and long-term health. This principle, pioneered by David Barker and coworkers in the early 90s, has later been termed ‘Developmental Origins
